Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2. - Wikidata - awgldk - TriplyDB

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

http://www.wikidata.org/entity/Q37257403

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Deletion of Nkx2-5 in trabecular myocardium reveals the developmental origins of pathological heterogeneity associated with ventricular non-compaction cardiomyopathy

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Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

http://www.wikidata.org/entity/Q37257403

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on December 2014

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vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@en

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@nl

type

label

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@en

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@nl

prefLabel

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@en

Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@nl

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ESC heart failure

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Six1 is down-regulated in end-stage human dilated cardiomyopathy independently of Ezh2.

@en

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Anika Tschirner

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Jochen Springer

http://www.w3.org/2001/XMLSchema#string

Rudolf Meyer

http://www.w3.org/2001/XMLSchema#string

Sandra Palus

http://www.w3.org/2001/XMLSchema#string

P2860

Six1 and Eya1 expression can reprogram adult muscle from the slow-twitch phenotype into the fast-twitch phenotype

Rapamycin-FKBP specifically blocks growth-dependent activation of and signaling by the 70 kd S6 protein kinases

A Tbx1-Six1/Eya1-Fgf8 genetic pathway controls mammalian cardiovascular and craniofacial morphogenesis

Functional consequences of sarcomeric protein abnormalities in failing myocardium.

Ezh1 and Ezh2 maintain repressive chromatin through different mechanisms

Increased phosphorylation-dependent nuclear export of class II histone deacetylases in failing human heart.

What causes a broken heart--molecular insights into heart failure.

Distribution of cardiac myosin isozymes in cardiomyopathy: immunohistochemical and gene analysis.

Epigenetic repression of cardiac progenitor gene expression by Ezh2 is required for postnatal cardiac homeostasis.

Skeletal muscle proteins oxidation in chronic right heart failure in rats: can different beta-blockers prevent it to the same degree?

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154-159

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scholarly article

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10.1002/EHF2.12017

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2

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1

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Stefan D. Anker

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2014-12-01T00:00:00Z

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271275679

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27668088

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5024036

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