Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy - Wikidata - awgldk - TriplyDB

Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

http://www.wikidata.org/entity/Q37258307

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The role of integrins in TGFβ activation in the tumour stroma Signatures of breast cancer metastasis at a glance Phase 1 dose-finding study of rebastinib (DCC-2036) in patients with relapsed chronic myeloid leukemia and acute myeloid leukemia.Endothelial cell-derived angiopoietin-2 is a therapeutic target in treatment-naive and bevacizumab-resistant glioblastoma.Soluble Tie2 overrides the heightened invasion induced by anti-angiogenesis therapies in gliomas.Macrophage-Induced Lymphangiogenesis and Metastasis following Paclitaxel Chemotherapy Is Regulated by VEGFR3 SDF-1 Blockade Enhances Anti-VEGF Therapy of Glioblastoma and Can Be Monitored by MRI.Metronomic chemotherapy prevents therapy-induced stromal activation and induction of tumor-initiating cells.Microfluidics: A new tool for modeling cancer-immune interactions.Stress-inducible gene Atf3 in the noncancer host cells contributes to chemotherapy-exacerbated breast cancer metastasis.The contribution of tumor-associated macrophages in glioma neo-angiogenesis and implications for anti-angiogenic strategies.The Promise of Targeting Macrophages in Cancer Therapy.The Multifaceted Role of Perivascular Macrophages in Tumors.Tumour-associated macrophages as treatment targets in oncology.Tumor Microenvironment and Differential Responses to Therapy.Progress in tumor-associated macrophage (TAM)-targeted therapeutics.Tumor Dormancy and Relapse: From a Natural Byproduct of Evolution to a Disease State.Tumor-Associated Macrophages as Target for Antitumor Therapy.Microenvironmental regulation of tumour angiogenesis.Neoadjuvant chemotherapy induces breast cancer metastasis through a TMEM-mediated mechanism.Circulating tumor cell interactions with macrophages: implications for biology and treatment.The impact of hypoxia on tumor-associated macrophages.Noninvasive Imaging of CD206-Positive M2 Macrophages as an Early Biomarker for Post-Chemotherapy Tumor Relapse and Lymph Node Metastasis.Precision Targeting of Tumor Macrophages with a CD206 Binding Peptide.Cystatin C deficiency suppresses tumor growth in a breast cancer model through decreased proliferation of tumor cells.Tumor-Associated Macrophages Derived from Circulating Inflammatory Monocytes Degrade Collagen through Cellular Uptake.Dynamic stroma reorganization drives blood vessel dysmorphia during glioma growth.Assessment of the Cancer Risk of the Fat-Grafted Breast in a Murine Model.MiR-770 suppresses the chemo-resistance and metastasis of triple negative breast cancer via direct targeting of STMN1.Tumor Associated Macrophages as Therapeutic Targets for Breast Cancer.The role of macrophage phenotype in regulating the response to radiation therapy.Tumor initiating cells induce Cxcr4-mediated infiltration of pro-tumoral macrophages into the brain.Repurposing tin mesoporphyrin as an immune checkpoint inhibitor shows therapeutic efficacy in preclinical models of cancer.Chemotherapy-induced metastasis: mechanisms and translational opportunities.Cancer Cell Death-Inducing Radiotherapy: Impact on Local Tumour Control, Tumour Cell Proliferation and Induction of Systemic Anti-tumour Immunity.The Selective Tie2 Inhibitor Rebastinib Blocks Recruitment and Function of Tie2Hi Macrophages in Breast Cancer and Pancreatic Neuroendocrine Tumors.Beyond the M-CSF receptor - novel therapeutic targets in tumor-associated macrophages.A Unidirectional Transition from Migratory to Perivascular Macrophage Is Required for Tumor Cell Intravasation.Contribution to Tumor Angiogenesis From Innate Immune Cells Within the Tumor Microenvironment: Implications for Immunotherapy.The role of IL-1B in breast cancer bone metastasis.

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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

http://www.wikidata.org/entity/Q37258307

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 12 August 2015

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy.

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type

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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy.

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prefLabel

Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy.

@nl

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0008-5472.CAN-14-3587

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Cancer Research

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Perivascular M2 Macrophages Stimulate Tumor Relapse after Chemotherapy

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Ana Maria Gonzalez-Angulo

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Bin-Zhi Qian

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Charlotte Rowan

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Christina Addison

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Claire E Lewis

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Ioanna Keklikoglou

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Jeffrey W Pollard

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Johanna A Joyce

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Mark Clemons

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Michele De Palma

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P2860

Anti-PlGF inhibits growth of VEGF(R)-inhibitor-resistant tumors without affecting healthy vessels

TIE2-expressing macrophages limit the therapeutic efficacy of the vascular-disrupting agent combretastatin A4 phosphate in mice

Deletion of vascular endothelial growth factor in myeloid cells accelerates tumorigenesis

CXCR7 (RDC1) promotes breast and lung tumor growth in vivo and is expressed on tumor-associated vasculature

Inhibition of vasculogenesis, but not angiogenesis, prevents the recurrence of glioblastoma after irradiation in mice.

Antiangiogenic scheduling of chemotherapy improves efficacy against experimental drug-resistant cancer

CCL2 recruits inflammatory monocytes to facilitate breast-tumour metastasis

Drug resistance by evasion of antiangiogenic targeting of VEGF signaling in late-stage pancreatic islet tumors

Macrophage diversity enhances tumor progression and metastasis

Direct visualization of macrophage-assisted tumor cell intravasation in mammary tumors

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3479-3491

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scholarly article

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10.1158/0008-5472.CAN-14-3587

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17

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75

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Munitta Muthana

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2015-08-12T00:00:00Z

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26269531

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