Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction. - Wikidata - awgldk - TriplyDB

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

http://www.wikidata.org/entity/Q37268365

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Redox regulation of ischemic limb neovascularization - What we have learned from animal studies High-throughput screening identifies microRNAs that target Nox2 and improve function after acute myocardial infarction.The Impact of Environmental Factors in Influencing Epigenetics Related to Oxidative States in the Cardiovascular System.Protection by Inhaled Hydrogen Therapy in a Rat Model of Acute Lung Injury can be Tracked in vivo Using Molecular Imaging.Maternal diabetes up-regulates NOX2 and enhances myocardial ischaemia/reperfusion injury in adult offspring.Cardiomyocytes Sense Matrix Rigidity through a Combination of Muscle and Non-muscle Myosin Contractions.Dunye Guanxinning Improves Acute Myocardial Ischemia-Reperfusion Injury by Inhibiting Neutrophil Infiltration and Caspase-1 Activity.

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Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

http://www.wikidata.org/entity/Q37268365

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 07 July 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

@en

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

@nl

type

label

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

@en

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

@nl

prefLabel

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

@en

Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction.

@nl

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J.YJMCC.2016.07.003

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Journal of Molecular and Cellular Cardiology

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Cell-specific effects of Nox2 on the acute and chronic response to myocardial infarction

@en

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Ajay M Shah

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Alexander Sirker

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Alison C Brewer

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Andrea Protti

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Celio X C Santos

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Daniel Martin

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Greta J Sawyer

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Min Zhang

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Xiaohong Zhang

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P2860

Biochemistry, physiology, and pathophysiology of NADPH oxidases in the cardiovascular system

NADPH oxidases in heart failure: poachers or gamekeepers?

Direct interaction of the novel Nox proteins with p22phox is required for the formation of a functionally active NADPH oxidase

NOX enzymes and the biology of reactive oxygen

High-frequency speckle tracking echocardiography in the assessment of left ventricular function and remodeling after murine myocardial infarction

Expression of p22-phox and gp91-phox, essential components of NADPH oxidase, increases after myocardial infarction.

Effects of cardiac overexpression of the angiotensin II type 2 receptor on remodeling and dysfunction in mice post-myocardial infarction

NADPH oxidase-4 mediates protection against chronic load-induced stress in mouse hearts by enhancing angiogenesis.

Inhibition of NADPH oxidase reduces myocardial oxidative stress and apoptosis and improves cardiac function in heart failure after myocardial infarction.

Increased Nox2 expression in human cardiomyocytes after acute myocardial infarction.

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11-17

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scholarly article

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10.1016/J.YJMCC.2016.07.003

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