Congenital nephrogenic diabetes insipidus: what can we learn from mouse models? - Wikidata - awgldk - TriplyDB

Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

http://www.wikidata.org/entity/Q37268479

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The Trafficking of the Water Channel Aquaporin-2 in Renal Principal Cells-a Potential Target for Pharmacological Intervention in Cardiovascular Diseases A novel therapeutic effect of statins on nephrogenic diabetes insipidus Aquaporins: important but elusive drug targets.Partial nephrogenic diabetes insipidus caused by a novel AQP2 variation impairing trafficking of the aquaporin-2 water channel.Regulation of transport in the connecting tubule and cortical collecting duct Nephrogenic diabetes insipidus: essential insights into the molecular background and potential therapies for treatment.A selective EP4 PGE2 receptor agonist alleviates disease in a new mouse model of X-linked nephrogenic diabetes insipidus.Aquaporin-2 abundance in the renal collecting duct: new insights from cultured cell models.Congenital nephrogenic diabetes insipidus: the current state of affairs.Reduced aldehyde dehydrogenase activity and arginine vasopressin receptor 2 expression in the kidneys of male TALLYHO/JngJ mice of prediabetic age.

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Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

http://www.wikidata.org/entity/Q37268479

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 12 September 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

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Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

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type

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Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

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Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

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prefLabel

Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

@en

Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

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EXPPHYSIOL.2008.043000

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Experimental Physiology

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Congenital nephrogenic diabetes insipidus: what can we learn from mouse models?

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Michelle Boone

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Peter M T Deen

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P2860

Vasopressin increases water permeability of kidney collecting duct by inducing translocation of aquaporin-CD water channels to plasma membrane

An aquaporin-2 water channel mutant which causes autosomal dominant nephrogenic diabetes insipidus is retained in the Golgi complex

Diabetes insipidus in mice with a mutation in aquaporin-2.

The subcellular localization of an aquaporin-2 tetramer depends on the stoichiometry of phosphorylated and nonphosphorylated monomers.

Cell biological aspects of the vasopressin type-2 receptor and aquaporin 2 water channel in nephrogenic diabetes insipidus

Heteroligomerization of an Aquaporin-2 mutant with wild-type Aquaporin-2 and their misrouting to late endosomes/lysosomes explains dominant nephrogenic diabetes insipidus.

Regulation and dysregulation of aquaporins in water balance disorders.

Hereditary polyuric disorders: new concepts and differential diagnosis.

Nephrogenic diabetes insipidus in mice caused by deleting COOH-terminal tail of aquaporin-2.

Three families with autosomal dominant nephrogenic diabetes insipidus caused by aquaporin-2 mutations in the C-terminus.

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186-190

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scholarly article

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10.1113/EXPPHYSIOL.2008.043000

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2

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94

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2008-09-12T00:00:00Z

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23255319

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18790812

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