MMP-9 controls Schwann cell proliferation and phenotypic remodeling via IGF-1 and ErbB receptor-mediated activation of MEK/ERK pathway.
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A delicate balance: role of MMP-9 in brain development and pathophysiology of neurodevelopmental disordersBiochemistry and molecular biology of gelatinase B or matrix metalloproteinase-9 (MMP-9): the next decadeUridine 5'-triphosphate promotes in vitro Schwannoma cell migration through matrix metalloproteinase-2 activationMatrix metalloproteinase inhibition enhances the rate of nerve regeneration in vivo by promoting dedifferentiation and mitosis of supporting schwann cellsThe MMP-9/TIMP-1 axis controls the status of differentiation and function of myelin-forming Schwann cells in nerve regeneration.Schwann cell-derived Apolipoprotein D controls the dynamics of post-injury myelin recognition and degradationExtracellular matrix protein 1 regulates cell proliferation and trastuzumab resistance through activation of epidermal growth factor signaling.The effects of claudin 14 during early Wallerian degeneration after sciatic nerve injury.Matrix metalloproteinase-14 both sheds cell surface neuronal glial antigen 2 (NG2) proteoglycan on macrophages and governs the response to peripheral nerve injurySmall-molecule anticancer compounds selectively target the hemopexin domain of matrix metalloproteinase-9Matrix metalloproteinase-9 controls proliferation of NG2+ progenitor cells immediately after spinal cord injury.The calcium-binding proteins S100A8 and S100A9 initiate the early inflammatory program in injured peripheral nerves.Acute morphine activates satellite glial cells and up-regulates IL-1β in dorsal root ganglia in mice via matrix metalloprotease-9The alternatively spliced fibronectin CS1 isoform regulates IL-17A levels and mechanical allodynia after peripheral nerve injury.Immunodominant fragments of myelin basic protein initiate T cell-dependent pain.miR-182 inhibits Schwann cell proliferation and migration by targeting FGF9 and NTM, respectively at an early stage following sciatic nerve injury.Diabetic retinopathy and signaling mechanism for activation of matrix metalloproteinase-9Expression of MMP-2 and MMP-9 in the rat trigeminal ganglion during the development of temporomandibular joint inflammation.Biochemical insights into the role of matrix metalloproteinases in regeneration: challenges and recent developments.Collagen triple helix repeat containing 1 (CTHRC1) acts via ERK-dependent induction of MMP9 to promote invasion of colorectal cancer cells.Current state of the development of mesenchymal stem cells into clinically applicable Schwann cell transplants.Repair of the Peripheral Nerve-Remyelination that Works.Growth factor choice is critical for successful functionalization of nanoparticles.Schwann Cell and Axon: An Interlaced Unit-From Action Potential to Phenotype Expression.Reciprocal relationship between membrane type 1 matrix metalloproteinase and the algesic peptides of myelin basic protein contributes to chronic neuropathic pain.Schwann cells genetically modified to express S100A4 increases GAP43 expression in spiral ganglion neurons in vitro.Involvement of MAPK ERK activation in upregulation of water channel protein aquaporin 1 in a mouse model of Bell's palsy.Improved axonal regeneration of transected spinal cord mediated by multichannel collagen conduits functionalized with neurotrophin-3 gene.Overexpression of endogenous TIMP-2 increases the proliferation of BeWo choriocarcinoma cells through the MAPK-signaling pathway.Involvement of placental growth factor in Wallerian degeneration.A sensitive and selective ELISA methodology quantifies a demyelination marker in experimental and clinical samples.Perineural expression of high-mobility group box-1 contributes to long-lasting mechanical hypersensitivity via matrix metalloproteinase-9 upregulation in mice with painful peripheral neuropathy.Acute- and late-phase matrix metalloproteinase (MMP)-9 activity is comparable in female and male rats after peripheral nerve injury.Assisted peripheral nerve recovery by KMUP-1, an activator of large-conductance Ca(2+)-activated potassium channel, in a rat model of sciatic nerve crush injury.Immunolocalization and activity of the MMP-9 and MMP-2 in odontogenic region of the rat incisor tooth after post shortening procedure.Dok4 is involved in Schwann cell myelination and axonal interaction in vitro.
P2860
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P2860
MMP-9 controls Schwann cell proliferation and phenotypic remodeling via IGF-1 and ErbB receptor-mediated activation of MEK/ERK pathway.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@en
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@nl
type
label
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@en
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@nl
prefLabel
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@en
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@nl
P2860
P356
P1433
P1476
MMP-9 controls Schwann cell pr ...... activation of MEK/ERK pathway.
@en
P2093
Sharmila Chattopadhyay
Veronica I Shubayev
P2860
P304
P356
10.1002/GLIA.20851
P577
2009-09-01T00:00:00Z