Deletion of the activating NKG2C receptor and a functional polymorphism in its ligand HLA-E in psoriasis susceptibility - Wikidata - awgldk - TriplyDB

Deletion of the activating NKG2C receptor and a functional polymorphism in its ligand HLA-E in psoriasis susceptibility

Deletion of the activating NKG2C receptor and a functional polymorphism in its ligand HLA-E in psoriasis susceptibility

http://www.wikidata.org/entity/Q37268945

about

Coherent somatic mutation in autoimmune disease.Rapid NK cell differentiation in a population with near-universal human cytomegalovirus infection is attenuated by NKG2C deletions.Tumor necrosis factor superfamily in innate immunity and inflammation.Differential frequency of NKG2C/KLRC2 deletion in distinct African populations and susceptibility to Trachoma: a new method for imputation of KLRC2 genotypes from SNP genotyping data NK cell autoreactivity and autoimmune diseases.Endoplasmic reticulum aminopeptidase 1 function and its pathogenic role in regulating innate and adaptive immunity in cancer and major histocompatibility complex class I-associated autoimmune diseases.HLA-E: a novel player for histocompatibility.Adaptive natural killer cell response to cytomegalovirus and disability progression in multiple sclerosis.NKG2C, HLA-E and their association with psoriasis Functional and Phenotypic Changes of Natural Killer Cells in Whole Blood during Mycobacterium tuberculosis Infection and Disease.HLA-E allelic genotype correlates with HLA-E plasma levels and predicts early progression in chronic lymphocytic leukemia.

P2860

Q33837769-179CC802-B545-4564-8E34-ACE1037C6463 Q34390895-53CA2879-25F4-4900-97BD-484C6E36E07F Q35245316-2704CD16-CE28-4C0E-B6B0-B2EACD821637 Q36054801-FBF6D5EC-884F-49C9-AF89-93B13BB3880A Q37552129-0F779D39-7251-4640-B88E-9026C033ADA0 Q38234401-1224EB15-9EB4-4959-819F-A7C729042097 Q38268679-1346B9A2-354F-40DA-82BC-D56A2120BBD3 Q41004127-9D31B075-4219-45A7-AEE1-59543A112D99 Q42426824-54E7F092-4FF8-43C4-9BDD-43370F10084B Q50531642-D8274F69-0DD3-4A02-869C-645E6FAF704F Q51320808-BF2B4D37-D5F1-436E-AD5E-18386CBF5E2D

P2860

Deletion of the activating NKG2C receptor and a functional polymorphism in its ligand HLA-E in psoriasis susceptibility

http://www.wikidata.org/entity/Q37268945

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on October 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Deletion of the activating NKG ...... -E in psoriasis susceptibility

@en

Deletion of the activating NKG ...... E in psoriasis susceptibility.

@nl

type

label

Deletion of the activating NKG ...... -E in psoriasis susceptibility

@en

Deletion of the activating NKG ...... E in psoriasis susceptibility.

@nl

prefLabel

Deletion of the activating NKG ...... -E in psoriasis susceptibility

@en

Deletion of the activating NKG ...... E in psoriasis susceptibility.

@nl

P1433

Q37268945-C226E816-45A4-4F5C-8B3D-2CB964F32041

P1476

Q37268945-E9970C65-60FF-4387-93AC-03E53E033C56

P2093

Q37268945-2E13E48F-0AA2-43E7-9EC6-7B8C2C687EBB

Q37268945-66A7D001-3A00-4791-A3B9-8FF006DD5290

Q37268945-BEB1B9CB-7EF5-441D-945E-1F71DF486122

Q37268945-D7C15820-4E0D-4F10-BBA7-0892BD45A003

P2860

Q37268945-01991B72-8D83-42FA-8D78-AEBD397D40F5

Q37268945-0BEAD206-0E68-4AAE-BDB1-E90C36E1E923

Q37268945-1052FFD0-BF90-4EF8-9A03-44D3528128F6

Q37268945-10AC4B9D-2B94-428F-A00F-88848104414A

Q37268945-1BA7E7E7-AE3C-4E8E-BEE2-654E93C5E55B

Q37268945-1EBEFA5B-8B14-4F06-A792-0971368FBF54

Q37268945-24203F00-86D4-41D3-81C1-DF5D3D26FEB0

Q37268945-3AEF7B05-2470-4C80-892B-9F4942991FAF

Q37268945-4E3D89BA-1231-4374-BDDF-35ACA7BF9FBB

Q37268945-5D9C6E30-2DD5-436B-9E13-EFECAB497340

P304

Q37268945-F023C6B8-90CB-42FB-80CD-24B4E34E9F9D

P31

Q37268945-0709BB11-C36B-4DD6-9124-4176C0347F2E

P356

Q37268945-422C1BE3-9061-4CC5-A821-A8D3DD0E2AC9

P433

Q37268945-4647A7DB-8FD6-4A05-968C-AA621CE264EC

P478

Q37268945-3A470F32-19DC-4180-8547-2784822C9D38

P50

Q37268945-252E5A09-DB7B-4706-BDFD-156762EA3780

Q37268945-91E3602B-F262-4A9D-81E4-B634C422C7DD

P577

Q37268945-AA49E0BC-97AD-4B9D-BB8D-0878105AF043

P5875

Q37268945-6120BF3F-990F-4701-AC66-F95B5B0A051D

P698

Q37268945-4D7F088B-9EB7-4C2D-AB4C-602E5332D023

P932

Q37268945-DEFAE282-ED5D-4BE3-B80B-9F2696C6D5F8

P356

P1433

Experimental Dermatology

P1476

Deletion of the activating NKG ...... -E in psoriasis susceptibility

@en

P2093

Haoyan Chen

http://www.w3.org/2001/XMLSchema#string

Oscar Paz-Altschul

http://www.w3.org/2001/XMLSchema#string

Rashmi Gupta

http://www.w3.org/2001/XMLSchema#string

Xue Zeng

http://www.w3.org/2001/XMLSchema#string

P2860

Genome-wide association study in alopecia areata implicates both innate and adaptive immunity

Psoriasis patients are enriched for genetic variants that protect against HIV-1 disease

A genome-wide association study identifies new psoriasis susceptibility loci and an interaction between HLA-C and ERAP1.

Molecular genetic analyses of human NKG2C (KLRC2) gene deletion.

HLA-E expression by gynecological cancers restrains tumor-infiltrating CD8⁺ T lymphocytes.

Natural killer cell activation enhances immune pathology and promotes chronic infection by limiting CD8+ T-cell immunity

NKG2C deletion is a risk factor of HIV infection.

Regulation of experimental autoimmune encephalomyelitis by natural killer (NK) cells

Natural killer cells act as rheostats modulating antiviral T cells.

Skewed distribution of natural killer cells in psoriasis skin lesions.

P304

679-681

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1111/EXD.12233

http://www.w3.org/2001/XMLSchema#string

P433

10

http://www.w3.org/2001/XMLSchema#string

P478

22

http://www.w3.org/2001/XMLSchema#string

P50

Anne M. Bowcock

P577

2013-10-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

257248271

http://www.w3.org/2001/XMLSchema#string

P698

24079744

http://www.w3.org/2001/XMLSchema#string

P932

3813441

http://www.w3.org/2001/XMLSchema#string