Increased chemoresistance via Snail-Raf kinase inhibitor protein signaling in colorectal cancer in response to a nicotine derivative
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A novel long non-coding RNA lnc-GNAT1-1 is low expressed in colorectal cancer and acts as a tumor suppressor through regulating RKIP-NF-κB-Snail circuit.The molecular mechanisms of chemoresistance in cancers.Acquisition cancer stemness, mesenchymal transdifferentiation, and chemoresistance properties by chronic exposure of oral epithelial cells to arecoline.SMO Inhibition Modulates Cellular Plasticity and Invasiveness in Colorectal Cancer.Characterisation of mesenchymal colon tumour-derived cells in tumourspheres as a model for colorectal cancer progressionRKIP: A Key Regulator in Tumor Metastasis Initiation and Resistance to Apoptosis: Therapeutic Targeting and Impact
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Increased chemoresistance via Snail-Raf kinase inhibitor protein signaling in colorectal cancer in response to a nicotine derivative
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 14 March 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Increased chemoresistance via ...... ponse to a nicotine derivative
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Increased chemoresistance via ...... onse to a nicotine derivative.
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type
label
Increased chemoresistance via ...... ponse to a nicotine derivative
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Increased chemoresistance via ...... onse to a nicotine derivative.
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Increased chemoresistance via ...... ponse to a nicotine derivative
@en
Increased chemoresistance via ...... onse to a nicotine derivative.
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P2093
P2860
P356
P1433
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Increased chemoresistance via ...... ponse to a nicotine derivative
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P2093
Chia-Lin Liu
Shu-Wen Jao
Su-Feng Chen
Tsai-Yu Lee
Tsung-Yun Liu
Yaoh-Shiang Lin
Yun-Ching Chang
P2860
P304
23512-23520
P356
10.18632/ONCOTARGET.8049
P407
P577
2016-03-14T00:00:00Z