Epigallocatechin-3-gallate prevents oxidative stress-induced cellular senescence in human mesenchymal stem cells via Nrf2
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Antioxidants Mediate Both Iron Homeostasis and Oxidative Stress.Caveolin‑1 regulates oxidative stress‑induced senescence in nucleus pulposus cells primarily via the p53/p21 signaling pathway in vitro.Protective effects of a green tea polyphenol, epigallocatechin-3-gallate, against sevoflurane-induced neuronal apoptosis involve regulation of CREB/BDNF/TrkB and PI3K/Akt/mTOR signalling pathways in neonatal mice.Inducers of Senescence, Toxic Compounds, and Senolytics: The Multiple Faces of Nrf2-Activating Phytochemicals in Cancer Adjuvant Therapy.Nectar Supplementation Reduced Biomarkers of Oxidative Stress, Muscle Damage, and Improved Psychological Response in Highly Trained Young Handball Players
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Epigallocatechin-3-gallate prevents oxidative stress-induced cellular senescence in human mesenchymal stem cells via Nrf2
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on August 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Epigallocatechin-3-gallate pre ...... esenchymal stem cells via Nrf2
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Epigallocatechin-3-gallate pre ...... senchymal stem cells via Nrf2.
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Epigallocatechin-3-gallate pre ...... esenchymal stem cells via Nrf2
@en
Epigallocatechin-3-gallate pre ...... senchymal stem cells via Nrf2.
@nl
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Epigallocatechin-3-gallate pre ...... esenchymal stem cells via Nrf2
@en
Epigallocatechin-3-gallate pre ...... senchymal stem cells via Nrf2.
@nl
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Epigallocatechin-3-gallate pre ...... esenchymal stem cells via Nrf2
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Hyo-Jin Jeon
Jihye Park
Mi-Sook Chang
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10.3892/IJMM.2016.2694
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2016-08-01T00:00:00Z