Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
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APOBEC3G-Mediated G-to-A Hypermutation of the HIV-1 Genome: The Missing Link in Antiviral Molecular MechanismsSingle-Stranded Nucleic Acids Bind to the Tetramer Interface of SAMHD1 and Prevent Formation of the Catalytic Homotetramer.Imaging HIV-1 Genomic DNA from Entry through Productive InfectionDeep sequencing of HIV-1 reverse transcripts reveals the multifaceted antiviral functions of APOBEC3G.HIV-1 Vpr Reprograms CLR4 E3 Ubiquitin Ligase to Antagonize Exonuclease 1-Mediated Restriction of HIV-1 Infection
P2860
Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 20 September 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
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Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
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type
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Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
@en
Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
@nl
prefLabel
Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
@en
Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells.
@nl
P2093
P2860
P356
P1433
P1476
Diverse fates of uracilated HIV-1 DNA during infection of myeloid lineage cells
@en
P2093
Adam A Capoferri
Erik C Hansen
Janet M Siliciano
Katherine M Bruner
Michael Bradley Drummond
Monica Ransom
Nina N Hosmane
Robert Siliciano
Ross A Pollack
P2860
P356
10.7554/ELIFE.18447
P407
P577
2016-09-20T00:00:00Z