IGF-IR tyrosine kinase interacts with NPM-ALK oncogene to induce survival of T-cell ALK+ anaplastic large-cell lymphoma cells.
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ALK Signaling and Target Therapy in Anaplastic Large Cell LymphomaPicropodophyllin causes mitotic arrest and catastrophe by depolymerizing microtubules via insulin-like growth factor-1 receptor-independent mechanismThe ALK inhibitor ASP3026 eradicates NPM-ALK⁺ T-cell anaplastic large-cell lymphoma in vitro and in a systemic xenograft lymphoma model.Expression and effects of inhibition of type I insulin-like growth factor receptor tyrosine kinase in mantle cell lymphoma.Inhibition of type I insulin-like growth factor receptor tyrosine kinase by picropodophyllin induces apoptosis and cell cycle arrest in T lymphoblastic leukemia/lymphoma.The transcription factors Ik-1 and MZF1 downregulate IGF-IR expression in NPM-ALK⁺ T-cell lymphomaMicroRNA 96 is a post-transcriptional suppressor of anaplastic lymphoma kinase expression.Targeting Notch1 and proteasome as an effective strategy to suppress T-cell lymphoproliferative neoplasmsSUMOylation Confers Posttranslational Stability on NPM-ALK Oncogenic Protein.Inhibition of IGF-IR tyrosine kinase induces apoptosis and cell cycle arrest in imatinib-resistant chronic myeloid leukaemia cellsEndogenous dendritic cells from the tumor microenvironment support T-ALL growth via IGF1R activation.Molecular and functional characterizations of the association and interactions between nucleophosmin-anaplastic lymphoma kinase and type I insulin-like growth factor receptor.NPM-ALK up-regulates iNOS expression through a STAT3/microRNA-26a-dependent mechanism.Type I insulin-like growth factor receptor signaling in hematological malignancies.Treatment for ALK-mutated non-small-cell lung cancer: a new miracle in the research race.ALK-positive cancer: still a growing entity.Therapeutic targeting of anaplastic lymphoma kinase in lung cancer: a paradigm for precision cancer medicine.TrkA is a binding partner of NPM-ALK that promotes the survival of ALK+ T-cell lymphoma.Crizotinib-induced antitumour activity in human alveolar rhabdomyosarcoma cells is not solely dependent on ALK and MET inhibition.Picropodophyllin inhibits proliferation and survival of diffuse large B-cell lymphoma cells.Studies of phosphoproteomic changes induced by nucleophosmin-anaplastic lymphoma kinase (ALK) highlight deregulation of tumor necrosis factor (TNF)/Fas/TNF-related apoptosis-induced ligand signaling pathway in ALK-positive anaplastic large cell lympCytotoxic effects of loperamide hydrochloride on canine cancer cells.Involvement of oncogenic tyrosine kinase NPM-ALK in trifluoperazine-induced cell cycle arrest and apoptosis in ALK(+) anaplastic large cell lymphoma.
P2860
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P2860
IGF-IR tyrosine kinase interacts with NPM-ALK oncogene to induce survival of T-cell ALK+ anaplastic large-cell lymphoma cells.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 May 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
@en
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
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type
label
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
@en
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
@nl
prefLabel
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
@en
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
@nl
P2093
P2860
P1433
P1476
IGF-IR tyrosine kinase interac ...... tic large-cell lymphoma cells.
@en
P2093
Abid S Iqbal
Hesham M Amin
Larry W Kwak
Raymond Lai
Richard J Ford
P2860
P304
P356
10.1182/BLOOD-2007-11-125658
P407
P50
P577
2009-05-07T00:00:00Z