Genomic aberrations occurring in subsets of serrated colorectal lesions but not conventional adenomas. - Wikidata - awgldk - TriplyDB

Genomic aberrations occurring in subsets of serrated colorectal lesions but not conventional adenomas.

Genomic aberrations occurring in subsets of serrated colorectal lesions but not conventional adenomas.

http://www.wikidata.org/entity/Q37274510

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Molecular phenotypes of colorectal cancer and potential clinical applications Similar but different: distinct roles for KRAS and BRAF oncogenes in colorectal cancer development and therapy resistance Etiologic field effect: reappraisal of the field effect concept in cancer predisposition and progression Discovery of colorectal cancer PIK3CA mutation as potential predictive biomarker: power and promise of molecular pathological epidemiology Serrated polyps and their alternative pathway to the colorectal cancer: a systematic review Differences in DNA methylation signatures reveal multiple pathways of progression from adenoma to colorectal cancer β-Catenin promotes colitis and colon cancer through imprinting of proinflammatory properties in T cells.IGF2 differentially methylated region hypomethylation in relation to pathological and molecular features of serrated lesions.Progress and opportunities in molecular pathological epidemiology of colorectal premalignant lesions.Claudin-1 Expression Is Elevated in Colorectal Cancer Precursor Lesions Harboring the BRAF V600E Mutation.Challenges and opportunities in international molecular cancer prevention research: An ASPO Molecular Epidemiology and the Environment and International Cancer Prevention Interest Groups Report.Proceedings of the second international molecular pathological epidemiology (MPE) meeting The significance of ectopic crypt formation in the differential diagnosis of colorectal polyps.Molecular markers identify subtypes of stage III colon cancer associated with patient outcomes Methylation and expression of the tumour suppressor, PRDM5, in colorectal cancer and polyp subgroups.Correlation between smoking history and molecular pathways in sporadic colorectal cancer: a meta-analysis.Altered promoter nucleosome positioning is an early event in gene silencing.Smoking status informs about the risk of advanced serrated polyps in a screening population Integration of molecular pathology, epidemiology and social science for global precision medicine.Genetic and epigenetic aberrations occurring in colorectal tumors associated with serrated pathway The molecular pathogenesis of colorectal cancer and its potential application to colorectal cancer screening Risk factors for serrated polyps of the colorectum.Review Article: The Role of Molecular Pathological Epidemiology in the Study of Neoplastic and Non-neoplastic Diseases in the Era of Precision Medicine.Gene Signature in Sessile Serrated Polyps Identifies Colon Cancer Subtype.Aspirin use and risk of colorectal cancer according to BRAF mutation status TP53 mutation at early stage of colorectal cancer progression from two types of laterally spreading tumors Long-term colorectal-cancer incidence and mortality after lower endoscopy.Predictive and prognostic analysis of PIK3CA mutation in stage III colon cancer intergroup trial.Assessment of epigenetic alterations in early colorectal lesions containing BRAF mutations.Accumulation of aberrant DNA methylation during colorectal cancer development.Interval colorectal cancers: what and why.DNA methylation accumulation and its predetermination of future cancer phenotypes.Colorectal serrated pathway cancers and precursors.Serrated neoplasia-role in colorectal carcinogenesis and clinical implications.The CIMP Phenotype in BRAF Mutant Serrated Polyps from a Prospective Colonoscopy Patient Cohort.Inflammatory response in serrated precursor lesions of the colon classified according to WHO entities, clinical parameters and phenotype-genotype correlation.Molecular genetic changes in benign colorectal tumors synchronous with microsatellite unstable carcinomas do not support a field defect BRAF inhibitor treatment of melanoma causing colonic polyps: An alternative hypothesis.MicroRNA-31 expression in relation to BRAF mutation, CpG island methylation and colorectal continuum in serrated lesions.Clinicopathological and molecular features of sessile serrated adenomas with dysplasia or carcinoma.

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P2860

Genomic aberrations occurring in subsets of serrated colorectal lesions but not conventional adenomas.

http://www.wikidata.org/entity/Q37274510

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 28 March 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Genomic aberrations occurring ...... but not conventional adenomas.

@en

Genomic aberrations occurring ...... but not conventional adenomas.

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type

label

Genomic aberrations occurring ...... but not conventional adenomas.

@en

Genomic aberrations occurring ...... but not conventional adenomas.

@nl

prefLabel

Genomic aberrations occurring ...... but not conventional adenomas.

@en

Genomic aberrations occurring ...... but not conventional adenomas.

@nl

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0008-5472.CAN-12-3462

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Cancer Research

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Genomic aberrations occurring ...... but not conventional adenomas.

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Amanda I Phipps

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Andrea N Burnett-Hartman

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Karen W Makar

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Lee-Ching Zhu

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Melissa P Upton

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Michael N Passarelli

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Michelle A Wurscher

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William M Grady

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P2860

Molecular pathological epidemiology of colorectal neoplasia: an emerging transdisciplinary and interdisciplinary field

Detection of BRAF V600E mutation in colorectal cancer: comparison of automatic sequencing and real-time chemistry methodology

Serrated lesions of the colorectum: review and recommendations from an expert panel

Lessons from hereditary colorectal cancer

Diet and lifestyle factor associations with CpG island methylator phenotype and BRAF mutations in colon cancer

Assessment of colorectal cancer molecular features along bowel subsites challenges the conception of distinct dichotomy of proximal versus distal colorectum

CpG island methylator phenotype underlies sporadic microsatellite instability and is tightly associated with BRAF mutation in colorectal cancer

Hyperplastic and serrated polyps of the colorectum.

Hyperplastic (serrated) polyps of the colorectum: relationship of CpG island methylator phenotype and K-ras mutation to location and histologic subtype.

Comparison of microsatellite instability, CpG island methylation phenotype, BRAF and KRAS status in serrated polyps and traditional adenomas indicates separate pathways to distinct colorectal carcinoma end points.

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2863-2872

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scholarly article

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10.1158/0008-5472.CAN-12-3462

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9

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73

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Polly A Newcomb

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2013-03-28T00:00:00Z

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23539450

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3815695

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