Arginase activity mediates retinal inflammation in endotoxin-induced uveitis. - Wikidata - awgldk - TriplyDB

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

http://www.wikidata.org/entity/Q37278531

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Lacritin and the tear proteome as natural replacement therapy for dry eye Vascular dysfunction in retinopathy-an emerging role for arginase Mutations in zebrafish lrp2 result in adult-onset ocular pathogenesis that models myopia and other risk factors for glaucoma Arginase 2 deletion reduces neuro-glial injury and improves retinal function in a model of retinopathy of prematurity.Human papillomavirus e7 oncoprotein transgenic skin develops an enhanced inflammatory response to 2,4-dinitrochlorobenzene by an arginase-1-dependent mechanism.Arginase 2 deficiency prevents oxidative stress and limits hyperoxia-induced retinal vascular degeneration.The role of arginase I in diabetes-induced retinal vascular dysfunction in mouse and rat models of diabetes.Arginase in retinopathy.Angiotensin II-induced vascular endothelial dysfunction through RhoA/Rho kinase/p38 mitogen-activated protein kinase/arginase pathway.Preconditioning of microglia by α-synuclein strongly affects the response induced by toll-like receptor (TLR) stimulation.Requirement of NOX2 expression in both retina and bone marrow for diabetes-induced retinal vascular injury.Posttranslational modification of Sirt6 activity by peroxynitrite.Arginase inhibition enhances angiogenesis in endothelial cells exposed to hypoxia.Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms.Oxidative species increase arginase activity in endothelial cells through the RhoA/Rho kinase pathway.Transcription factor MEF2C suppresses endothelial cell inflammation via regulation of NF-κB and KLF2 Epithelium Expressing the E7 Oncoprotein of HPV16 Attracts Immune-Modulatory Dendritic Cells to the Skin and Suppresses Their Antigen-Processing Capacity Treatment with the arginase inhibitor Nw-hydroxy-nor-L-arginine restores endothelial function in rat adjuvant-induced arthritis.Cyclic AMP is a key regulator of M1 to M2a phenotypic conversion of microglia in the presence of Th2 cytokines Targeting arginase-II protects mice from high-fat-diet-induced hepatic steatosis through suppression of macrophage inflammation.Prevention of diabetes-induced arginase activation and vascular dysfunction by Rho kinase (ROCK) knockout.Arginase 1 mediates increased blood pressure and contributes to vascular endothelial dysfunction in deoxycorticosterone acetate-salt hypertension MMP-3 Deficiency Alleviates Endotoxin-Induced Acute Inflammation in the Posterior Eye Segment.Novel mechanisms of endothelial dysfunction in diabetes.Anti-inflammatory therapy for diabetic retinopathy.Inflammation and diabetic retinal microvascular complications.Arginase as a potential target in the treatment of cardiovascular disease: reversal of arginine steal?Arginase: an old enzyme with new tricks.Growth Factors in the Pathogenesis of Retinal Neurodegeneration in Diabetes Mellitus Arginase as a mediator of diabetic retinopathy.Supplemental carvacrol can reduce the severity of inflammation by influencing the production of mediators of inflammation.Influence of the AT(2) receptor on the L-arginine-nitric oxide pathway and effects of (-)-epicatechin on HUVECs from women with preeclampsia.Obesity-induced vascular dysfunction and arterial stiffening requires endothelial cell arginase 1.Enzymes of urea synthesis are expressed at the ocular surface, and decreased urea in the tear fluid is associated with dry-eye syndrome.

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Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

http://www.wikidata.org/entity/Q37278531

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 July 2009

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

@cs

name

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

@en

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

@nl

type

label

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

@en

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

@nl

prefLabel

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

@en

Arginase activity mediates retinal inflammation in endotoxin-induced uveitis.

@nl

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AJPATH.2009.081115

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The American Journal of Pathology

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Arginase activity mediates retinal inflammation in endotoxin-induced uveitis

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Babak Baban

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Chintan Patel

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M Ali Behzadian

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Maritza J Romero

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Robert W Caldwell

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Ruth B Caldwell

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Sohrab Tofigh

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Suvika K Virmani

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Wenbo Zhang

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P2860

Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production

Coinduction of nitric-oxide synthase and arginase I in cultured rat peritoneal macrophages and rat tissues in vivo by lipopolysaccharide

Coinduction of nitric oxide synthase and arginine metabolic enzymes in endotoxin-induced uveitis rats

Peroxynitrite: biochemistry, pathophysiology and development of therapeutics

Convergence of nitric oxide and lipid signaling: anti-inflammatory nitro-fatty acids.

Suppression and regression of choroidal neovascularization by polyamine analogues.

Inhibition of Hsp90 attenuates inflammation in endotoxin-induced uveitis.

NAD(P)H oxidase-dependent regulation of CCL2 production during retinal inflammation.

Elevated arginase I expression in rat aortic smooth muscle cells increases cell proliferation.

Diffusion and reaction of nitric oxide in suspension cell cultures.

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891-902

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10.2353/AJPATH.2009.081115

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19590038

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