Evidence for defective energy homeostasis in amyotrophic lateral sclerosis: benefit of a high-energy diet in a transgenic mouse model.
about
The autophagic tumor stroma model of cancer: Role of oxidative stress and ketone production in fueling tumor cell metabolismSperm and oocyte communication mechanisms controlling C. elegans fertilityVgf is a novel biomarker associated with muscle weakness in amyotrophic lateral sclerosis (ALS), with a potential role in disease pathogenesisThe Interaction Between Statins and Exercise: Mechanisms and Strategies to Counter the Musculoskeletal Side Effects of This Combination TherapyComplementary and Alternative Therapies in Amyotrophic Lateral SclerosisThe Neuromuscular Junction: Aging at the Crossroad between Nerves and MuscleThe complex molecular biology of amyotrophic lateral sclerosis (ALS)Characterization of a novel SOD-1(G93A) transgenic mouse line with very decelerated disease developmentBlood Cell Palmitoleate-Palmitate Ratio Is an Independent Prognostic Factor for Amyotrophic Lateral SclerosisA randomized, double blind, placebo-controlled trial of pioglitazone in combination with riluzole in amyotrophic lateral sclerosisPlatelet serotonin level predicts survival in amyotrophic lateral sclerosisThe effect of diet on the protective action of D156844 observed in spinal muscular atrophy mice.Mitochondrial dysfunction and decrease in body weight of a transgenic knock-in mouse model for TDP-43.Comparison of Sirtuin 3 Levels in ALS and Huntington's Disease-Differential Effects in Human Tissue Samples vs. Transgenic Mouse Models.Effect of diet on the survival and phenotype of a mouse model for spinal muscular atrophySkeletal muscle-restricted expression of human SOD1 causes motor neuron degeneration in transgenic miceAdipokines, C-reactive protein and Amyotrophic Lateral Sclerosis - results from a population- based ALS registry in Germany.Amyotrophic lateral sclerosis is associated with hypolipidemia at the presymptomatic stage in miceThe metabolic phenotype of SCD1-deficient mice is independent of melanin-concentrating hormoneInhibition of β-Glucocerebrosidase Activity Preserves Motor Unit Integrity in a Mouse Model of Amyotrophic Lateral Sclerosis.Abnormal changes in NKT cells, the IGF-1 axis, and liver pathology in an animal model of ALS.Effects of natural antioxidants in neurodegenerative disease.Genetically altering organismal metabolism by leptin-deficiency benefits a mouse model of amyotrophic lateral sclerosisTwo-point magnitude MRI for rapid mapping of brown adipose tissue and its application to the R6/2 mouse model of Huntington diseaseDeletion of TDP-43 down-regulates Tbc1d1, a gene linked to obesity, and alters body fat metabolism.Hypercaloric enteral nutrition in patients with amyotrophic lateral sclerosis: a randomised, double-blind, placebo-controlled phase 2 trialAblation of Sim1 neurons causes obesity through hyperphagia and reduced energy expenditure.Dihydrotestosterone ameliorates degeneration in muscle, axons and motoneurons and improves motor function in amyotrophic lateral sclerosis model mice.Mechanisms, models and biomarkers in amyotrophic lateral sclerosisAdenosine monophosphate-activated protein kinase activation enhances embryonic neural stem cell apoptosis in a mouse model of amyotrophic lateral sclerosis.Cholesterol, oxysterol, triglyceride, and coenzyme Q homeostasis in ALS. Evidence against the hypothesis that elevated 27-hydroxycholesterol is a pathogenic factor.Systemic down-regulation of delta-9 desaturase promotes muscle oxidative metabolism and accelerates muscle function recovery following nerve injury.Adipose Tissue Distribution Predicts Survival in Amyotrophic Lateral SclerosisWeight loss, dysphagia and supplement intake in patients with amyotrophic lateral sclerosis (ALS): impact on quality of life and therapeutic options.Mitochondrial abnormalities and low grade inflammation are present in the skeletal muscle of a minority of patients with amyotrophic lateral sclerosis; an observational myopathology study.Gene expression signatures in motor neurone disease fibroblasts reveal dysregulation of metabolism, hypoxia-response and RNA processing functionsMutant TDP-43 deregulates AMPK activation by PP2A in ALS models.The neurite outgrowth inhibitor Nogo-A promotes denervation in an amyotrophic lateral sclerosis modelPPAR gamma activation is neuroprotective in a Drosophila model of ALS based on TDP-43.TDP-43-based animal models of neurodegeneration: new insights into ALS pathology and pathophysiology.
P2860
Q24611412-29B4D21E-C47D-4273-812D-87BC61A267D9Q24626882-BAA85F8A-BA95-4949-B540-9A7A10DDD12BQ24644968-491AB2D6-FFE6-4780-A537-445BBB2565D7Q26771168-F9D46AB4-4D92-44FD-82EE-54B0556664B5Q26777929-1B45A0D0-23C0-4B62-B6CF-FD69C72FE54CQ27030836-79146B8D-A0DD-4DE8-885D-0601F0EFF455Q28263899-B9C5B9A7-8416-4812-8CF9-707B10C75F8AQ28476121-350E3EB1-DB6E-4973-B9C9-2FC2654D72D0Q28546154-AC5AD2B0-9DF5-42A3-8908-059C1022C096Q28728672-A88B225F-6DBC-4E5C-930A-2E1B13E2FBF3Q28748875-9223D043-1C05-45C6-ABC2-6B141930B21DQ33645966-E7F68BC8-6368-4757-AD93-6F556D6004DBQ33675871-88A3CCAB-D549-4BF0-9E33-174DBA73B18AQ33729341-C3E6B596-DD95-450B-B92D-360DEC2809AAQ33730194-7F0C96D0-A2C4-4494-95B8-CAE461A144EEQ33832905-3DCCCCFA-81EF-4B4C-9D12-B213EB42B011Q33854270-1CD08203-2C7C-4185-A535-12C388B6E152Q33862624-831355F5-F24C-4EDC-8B44-5CADFAC5F684Q33897038-EB303757-9D25-41A5-A3D5-0B0DD856213CQ33899405-D7366A89-3568-4F0F-AE95-C2175D8CC5B3Q33988381-750B7CC2-ECED-411B-A627-D7620E4D5B44Q34030539-A7ED434B-51A1-461B-A51A-79532AC4C4B1Q34074537-C4C00962-13B7-490F-B572-F0CA463DC9FBQ34075646-59366F77-25D0-44DF-8846-44BE89314FCAQ34136356-C4CD93AB-D538-446B-8CD0-F2D6AA18DF54Q34251334-028DA57D-AD10-49CD-B003-CD0C1D2015F6Q34257527-F95CB17E-51CC-4CA9-90DE-C97B2582E562Q34273954-33374AA9-2634-4C66-AA18-6E59CC72B273Q34344992-E96B5103-7ED3-45B7-A353-76B7CB40AAFBQ34546198-9A5138A2-E1A6-4696-B62B-DB126355839AQ34557247-F5561133-02C8-4594-ADE4-CFF4CF619DC0Q34778610-95F32A73-6330-4CD2-9E65-A522B07205B3Q34797530-3B8255CE-B669-430A-A1B2-B4CF6242015BQ34815254-E8B4764B-684E-41B8-B30A-0E7990682A23Q34978980-D4519644-AE68-424F-A7A3-DE235BCDFD7DQ35086167-874C6CC2-D014-4190-8287-0F1506F323EFQ35111080-48B08A5D-26E1-454D-8B27-9423C9346B3CQ35191180-802D8C29-6FD1-41AB-82C4-DFAF8A662DB3Q35239387-0F4BEBC5-564F-4131-9AA8-05069884AB11Q35546452-55A023A6-74E1-4AA2-8CDF-61C4AE3EF39C
P2860
Evidence for defective energy homeostasis in amyotrophic lateral sclerosis: benefit of a high-energy diet in a transgenic mouse model.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 19 July 2004
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Evidence for defective energy ...... t in a transgenic mouse model.
@en
Evidence for defective energy ...... t in a transgenic mouse model.
@nl
type
label
Evidence for defective energy ...... t in a transgenic mouse model.
@en
Evidence for defective energy ...... t in a transgenic mouse model.
@nl
prefLabel
Evidence for defective energy ...... t in a transgenic mouse model.
@en
Evidence for defective energy ...... t in a transgenic mouse model.
@nl
P2093
P2860
P356
P1476
Evidence for defective energy ...... t in a transgenic mouse model.
@en
P2093
Frédérique René
Hugues Oudart
Jean-Philippe Loeffler
Jose-Luis Gonzalez de Aguilar
Luc Dupuis
P2860
P304
11159-11164
P356
10.1073/PNAS.0402026101
P407
P577
2004-07-19T00:00:00Z