Different roles for TGF-beta and VEGF in the pathogenesis of the cardinal features of diabetic nephropathy.
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Present and future in the treatment of diabetic kidney diseaseGene expression differences in skin fibroblasts in identical twins discordant for type 1 diabetes.Dedifferentiation of immortalized human podocytes in response to transforming growth factor-β: a model for diabetic podocytopathy.Targeting SGK1 in diabetesLaminin α1 regulates age-related mesangial cell proliferation and mesangial matrix accumulation through the TGF-β pathwayThe effect of chronic candesartan therapy on the metabolic profile and renal tissue cytokine levels in the obese Zucker rat.Hyperglycaemia emerging during general anaesthesia induces rat acute kidney injury via impaired microcirculation, augmented apoptosis and inhibited cell proliferation.The protective role of Smad7 in diabetic kidney disease: mechanism and therapeutic potentialPoly(ADP-ribose) polymerase (PARP) inhibition counteracts multiple manifestations of kidney disease in long-term streptozotocin-diabetic rat modelExpression profiles of podocytes exposed to high glucose reveal new insights into early diabetic glomerulopathySilencing of hypoxia-inducible factor-1α gene attenuated angiotensin II-induced renal injury in Sprague-Dawley rats.Therapeutic potential of Nrf2 activators in streptozotocin-induced diabetic nephropathy.The Decrement of Hemoglobin Concentration with Angiotensin II Receptor Blocker Treatment Is Correlated with the Reduction of Albuminuria in Non-Diabetic Hypertensive Patients: Post-Hoc Analysis of ESPECIAL Trial.Pathophysiological role and therapeutic implications of inflammation in diabetic nephropathyPrediabetic nephropathy as an early consequence of the high-calorie/high-fat diet: relation to oxidative stress.VEGF165b overexpression restores normal glomerular water permeability in VEGF164-overexpressing adult mice.A transcriptional blueprint for human and murine diabetic kidney diseaseImplications of treatment that target protective mechanisms against diabetic nephropathyVascular Endothelial Growth Factor and Podocyte Protection in Chronic Hypoxia: Effects of Endothelin-A Receptor Antagonism.Poly(Adenosine 5'-diphosphate-ribose) polymerase inhibition counteracts multiple manifestations of experimental type 1 diabetic nephropathy.Heterocyclic indazole derivatives as SGK1 inhibitors, WO2008138448.Renal effects of chronic pharmacological manipulation of CB2 receptors in rats with diet-induced obesity.Role of angiotensin II in the development of nephropathy and podocytopathy of diabetes.New perspectives on diabetic vascular complications: the loss of endogenous protective factors induced by hyperglycemia.Role of TGF-β in chronic kidney disease: an integration of tubular, glomerular and vascular effects.Oxidative-stress-induced epigenetic changes in chronic diabetic complications.Therapeutic approaches to diabetic nephropathy--beyond the RAS.Association between the T869C polymorphism of transforming growth factor-beta 1 and diabetic nephropathy: a meta-analysis.Prostaglandin E2 increases proximal tubule fluid reabsorption, and modulates cultured proximal tubule cell responses via EP1 and EP4 receptors.Urinary platelet-derived growth factor-BB as an early marker of nephropathy in patients with type 2 diabetes: an Egyptian study.Nodes with high centrality in protein interaction networks are responsible for driving signaling pathways in diabetic nephropathy.FK506 ameliorates podocyte injury in type 2 diabetic nephropathy by down-regulating TRPC6 and NFAT expression.Type VIII collagen modulates TGF-β1-induced proliferation of mesangial cellsAdenosine mediates transforming growth factor-beta 1 release in kidney glomeruli of diabetic rats.Renal damage following Alloxan-induced diabetes is associated with generation of reactive oxygen species, alterations of p53, TGF-β1, and extracellular matrix metalloproteinases in rats.Albumin inhibits the insulin-mediated ACE2 increase in cultured podocytes.Effects of garlic extract on TNF-α expression and oxidative stress status in the kidneys of rats with STZ + nicotinamide-induced diabetes.
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P2860
Different roles for TGF-beta and VEGF in the pathogenesis of the cardinal features of diabetic nephropathy.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 07 October 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
@en
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
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type
label
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
@en
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
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prefLabel
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
@en
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
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P1476
Different roles for TGF-beta a ...... tures of diabetic nephropathy.
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P2093
Fuad N Ziyadeh
P304
P356
10.1016/J.DIABRES.2008.09.016
P478
82 Suppl 1
P577
2008-10-07T00:00:00Z