Blocked early-stage latency in the peripheral blood cells of certain individuals infected with human immunodeficiency virus type 1 - Wikidata - awgldk - TriplyDB

Blocked early-stage latency in the peripheral blood cells of certain individuals infected with human immunodeficiency virus type 1

Blocked early-stage latency in the peripheral blood cells of certain individuals infected with human immunodeficiency virus type 1

http://www.wikidata.org/entity/Q37292229

about

ERK MAP kinase links cytokine signals to activation of latent HIV-1 infection by stimulating a cooperative interaction of AP-1 and NF-kappaB Isoquinolinesulphonamide derivatives inhibit transcriptional elongation of human immunodeficiency virus type 1 RNA in a promyelocytic model of latency An "escape clock" for estimating the turnover of SIV DNA in resting CD4⁺ T cells Presence of an inducible HIV-1 latent reservoir during highly active antiretroviral therapy Nef-induced CD4 and major histocompatibility complex class I (MHC-I) down-regulation are governed by distinct determinants: N-terminal alpha helix and proline repeat of Nef selectively regulate MHC-I trafficking.Control of human immunodeficiency virus type 1 RNA metabolism: role of splice sites and intron sequences in unspliced viral RNA subcellular distribution.Cytomegalovirus replication in semen is associated with higher levels of proviral HIV DNA and CD4+ T cell activation during antiretroviral treatment.Human immunodeficiency virus 1 reservoir in CD4+ T cells is restricted to certain V beta subsets Double-stranded RNA adenosine deaminases enhance expression of human immunodeficiency virus type 1 proteins Defective virus drives human immunodeficiency virus infection, persistence, and pathogenesis.Cell-associated viral burden provides evidence of ongoing viral replication in aviremic HIV-2-infected patients Analysis of human immunodeficiency virus type 1 gene expression in latently infected resting CD4+ T lymphocytes in vivo Cellular latency in human immunodeficiency virus-infected individuals with high CD4 levels can be detected by the presence of promoter-proximal transcripts.Human immunodeficiency virus type 1 cellular RNA load and splicing patterns predict disease progression in a longitudinally studied cohort.Changes in the viral mRNA expression pattern correlate with a rapid rate of CD4+ T-cell number decline in human immunodeficiency virus type 1-infected individuals Establishment of a stable, inducible form of human immunodeficiency virus type 1 DNA in quiescent CD4 lymphocytes in vitro Detection of CD4+ T cells harboring human immunodeficiency virus type 1 DNA by flow cytometry using simultaneous immunophenotyping and PCR-driven in situ hybridization: evidence of epitope masking of the CD4 cell surface molecule in vivo Persistence of attenuated rev genes in a human immunodeficiency virus type 1-infected asymptomatic individual.Dynamics and modulation of human immunodeficiency virus type 1 transcripts in vitro and in vivo Sequence Analysis of In Vivo-Expressed HIV-1 Spliced RNAs Reveals the Usage of New and Unusual Splice Sites by Viruses of Different Subtypes.RNAi as an antiviral therapy.Human immunodeficiency virus type 1 RNA expression by four chronically infected cell lines indicates multiple mechanisms of latency Experimental approaches to the study of HIV-1 latency.Cell-associated HIV RNA: a dynamic biomarker of viral persistence.Short Intracellular HIV-1 Transcripts as Biomarkers of Residual Immune Activation in Patients on Antiretroviral Therapy Analysis of human immunodeficiency virus type 1 transcriptional elongation in resting CD4+ T cells in vivo Preferential replication of HIV-1 in the CD45RO memory cell subset of primary CD4 lymphocytes in vitro.Dynamics of HIV-1 mRNA expression in patients with long-term nonprogressive HIV-1 infection.Human immunodeficiency virus type 1 mRNA expression in peripheral blood cells predicts disease progression independently of the numbers of CD4+ lymphocytes Positive and negative modulation of human immunodeficiency virus type 1 Rev function by cis and trans regulators of viral RNA splicing The importance of nef in the induction of human immunodeficiency virus type 1 replication from primary quiescent CD4 lymphocytes.Regulated expression of HIV-1 Rev function in mammalian cell lines.Antiviral effect and ex vivo CD4+ T cell proliferation in HIV-positive patients as a result of CD28 costimulation.Latent HIV-1 TAR Regulates 7SK-responsive P-TEFb Target Genes and Targets Cellular Immune Responses in the Absence of Tat.What do we measure when we measure cell-associated HIV RNA.Quantitative image analysis of HIV-1 infection in lymphoid tissue.

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P2860

Blocked early-stage latency in the peripheral blood cells of certain individuals infected with human immunodeficiency virus type 1

http://www.wikidata.org/entity/Q37292229

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1992 nî lūn-bûn

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P2860

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

HIV-1 entry into quiescent primary lymphocytes: molecular analysis reveals a labile, latent viral structure

The human immunodeficiency virus: infectivity and mechanisms of pathogenesis.

Rapidly and slowly replicating human immunodeficiency virus type 1 isolates can be distinguished according to target-cell tropism in T-cell and monocyte cell lines.

Detection of lymphocytes expressing human T-lymphotropic virus type III in lymph nodes and peripheral blood from infected individuals by in situ hybridization

Lipopolysaccharide is a potent monocyte/macrophage-specific stimulator of human immunodeficiency virus type 1 expression.

NIH conference. Immunopathogenic mechanisms in human immunodeficiency virus (HIV) infection.

Induction of human immunodeficiency virus type 1 expression in chronically infected cells is associated primarily with a shift in RNA splicing patterns.

Analysis of rev gene function on human immunodeficiency virus type 1 replication in lymphoid cells by using a quantitative polymerase chain reaction method.

Seropositivity for HIV and the development of AIDS or AIDS related condition: three year follow up of the San Francisco General Hospital cohort.

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