Granulocyte macrophage colony-stimulating factor inhibits breast cancer growth and metastasis by invoking an anti-angiogenic program in tumor-educated macrophages.
about
Regulation of epithelial-mesenchymal transition by tumor-associated macrophages in cancerMechanisms driving macrophage diversity and specialization in distinct tumor microenvironments and parallelisms with other tissuesMathematical modeling of Interleukin-35 promoting tumor growth and angiogenesisInterstitial Inorganic Phosphate as a Tumor Microenvironment Marker for Tumor ProgressionMacrophage diversity enhances tumor progression and metastasisMolecular changes in bone marrow, tumor and serum after conductive ablation of murine 4T1 breast carcinoma.Preferential, enhanced breast cancer cell migration on biomimetic electrospun nanofiber 'cell highways'Role of the VEGF ligand to receptor ratio in the progression of mismatch repair-proficient colorectal cancer.Targeting distinct tumor-infiltrating myeloid cells by inhibiting CSF-1 receptor: combating tumor evasion of antiangiogenic therapy.The immunosuppressive surface ligand CD200 augments the metastatic capacity of squamous cell carcinoma.Theory, instrumentation, and applications of electron paramagnetic resonance oximetry.LyGDI, a novel SHIP-interacting protein, is a negative regulator of FcγR-mediated phagocytosis.Involvement of tumor macrophage HIFs in chemotherapy effectiveness: mathematical modeling of oxygen, pH, and glutathione.Opposing roles for HIF-1α and HIF-2α in the regulation of angiogenesis by mononuclear phagocytes.Angiocidin inhibits breast cancer proliferation through activation of epidermal growth factor receptor and nuclear factor kappa (NF-ĸB).Advances in probes and methods for clinical EPR oximetry.Vascular normalization induced by sinomenine hydrochloride results in suppressed mammary tumor growth and metastasisMammary gland involution as an immunotherapeutic target for postpartum breast cancer.Transcription factor ets-2 plays an important role in the pathogenesis of pulmonary fibrosis.uPA and uPAR shRNA inhibit angiogenesis via enhanced secretion of SVEGFR1 independent of GM-CSF but dependent on TIMP-1 in endothelial and glioblastoma cells.In vivo monitoring of pH, redox status, and glutathione using L-band EPR for assessment of therapeutic effectiveness in solid tumors.Serum uPAR as Biomarker in Breast Cancer Recurrence: A Mathematical Model.A nonpeptidic cathepsin S activity-based probe for noninvasive optical imaging of tumor-associated macrophagesEnhanced metastasis in RNF13 knockout mice is mediated by a reduction in GM-CSF levels.Stabilization of HIF-2α induces sVEGFR-1 production from tumor-associated macrophages and decreases tumor growth in a murine melanoma model.Angiotensin II type 1 receptor (AT-1R) expression correlates with VEGF-A and VEGF-D expression in invasive ductal breast cancerAnalysis of gene expression of secreted factors associated with breast cancer metastases in breast cancer subtypes.Baseline blood immunological profiling differentiates between Her2-breast cancer molecular subtypes: implications for immunomediated mechanisms of treatment response.Temporal phenotypic features distinguish polarized macrophages in vitroTumor-associated macrophages: function, phenotype, and link to prognosis in human lung cancer.Thyroid Transcription Factor 1 Reprograms Angiogenic Activities of Secretome.Neutrophils drive accelerated tumor progression in the collagen-dense mammary tumor microenvironment.Lymphatic endothelial cells support tumor growth in breast cancer.Fcγ receptor-induced soluble vascular endothelial growth factor receptor-1 (VEGFR-1) production inhibits angiogenesis and enhances efficacy of anti-tumor antibodies.Intermittent hypoxia exacerbates pancreatic β-cell dysfunction in a mouse model of diabetes mellitusThe Angiogenic Secretome in VEGF overexpressing Breast Cancer Xenografts.Bioconjugated Manganese Dioxide Nanoparticles Enhance Chemotherapy Response by Priming Tumor-Associated Macrophages toward M1-like Phenotype and Attenuating Tumor Hypoxia.Hypoxia inducible factors-mediated inhibition of cancer by GM-CSF: a mathematical model.Monocytes and macrophages regulate immunity through dynamic networks of survival and cell death.Tumor-associated macrophages in breast cancer as potential biomarkers for new treatments and diagnostics.
P2860
Q26773786-C155798B-B178-48E3-AA74-69F5ECE29531Q27003907-9841D2A1-D66D-4752-9F38-7E276C6F99E5Q28544606-9C5E932B-D495-471F-964B-1CB67C0F1C4EQ28818464-234E5959-AEF7-4700-90BC-FDB6EBA01BD8Q29615847-CC446A45-52E6-4FE4-BE8A-A10B61702C04Q30444038-7FA85267-8C8C-47CC-8549-86562F7DA393Q30599338-3C2D1DE1-37F4-4668-AACA-26F2B5373423Q33539996-5986111B-D28B-4AA7-B03E-7A3FF1AB65C3Q33680309-255F09F5-FB4B-44A7-B487-858E5786BB92Q33767115-B921C0DC-1606-4844-BFAB-AFE0E678FDEEQ33847052-EB313DF9-0EDB-4AC7-8B4D-FF524B634305Q33939284-52B7C2A1-A960-445D-A67B-86ED4074A42AQ34306854-BEC20A62-1E41-4B7D-A9D7-D122182855D6Q34568910-E686B876-ADA2-4397-93D1-B98068F814F2Q34970505-CB0B8457-CB0A-4ED3-ACF2-DAC5C3AF10D3Q35037691-E567FCFC-6721-4763-8491-F77543E28765Q35157184-77A5536D-AD3B-4428-BFB5-052A20AA61F5Q35186471-60FFCA9C-9036-4EE3-B8F1-EB21391615BFQ35684856-4BB91E43-FC24-4CDB-BF5E-17F1BA5FF114Q35713784-C4C123D7-0400-4A85-B88A-DF9B1F6A050EQ35834783-8BE2E547-F29D-4005-AF52-5F635F92F86FQ35990029-0C250938-1D61-456F-9EFD-27C95B6C6B0FQ35993592-00A8E83F-B0C7-41D6-9741-B5C038DA2EEEQ36137690-BE3E4587-8AED-4D12-B64F-72D99B3B955FQ36218573-F6856CF9-422C-4268-BC43-26E179E8D819Q36250185-C99F46F6-0412-4B19-BCA0-DEE79F120182Q36288549-A9181CF1-4DD1-44F4-8AEA-C5B26F0F1637Q36307206-B4309269-E42D-465E-8A17-6FD232723DE2Q36377797-19DB4E4C-90DA-4584-8024-C50D93076958Q36383090-DF6B7B0A-1E9A-4E39-9A9E-1D0A39C9B5EBQ36615870-425780E8-7989-41A0-8B58-974557BF1A05Q36895360-9562FBFE-46CC-4F08-89E4-0B765CBAF189Q37057680-D6FC847B-DF2D-413E-95A4-8940ACC0717BQ37168454-B96D5D84-163D-44E4-BA0D-0357F3D2B8E3Q37301887-2B4348A8-F316-4D37-8344-1F4E6D9B1B2CQ37519061-E9350C4B-5987-4376-BD6B-95D665788E10Q37592070-CF63921B-B2DF-4E19-8D41-95D2F08A626EQ37606594-EAA51BF8-4CD7-45A0-8A1B-FF9FD52127F4Q37726689-AA40F719-0CBD-4A8F-AB3F-FA3173DC2547Q37822464-6EC0A4BF-327E-4BB4-9E20-38DE426C7566
P2860
Granulocyte macrophage colony-stimulating factor inhibits breast cancer growth and metastasis by invoking an anti-angiogenic program in tumor-educated macrophages.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 17 February 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@en
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@nl
type
label
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@en
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@nl
prefLabel
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@en
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@nl
P2093
P2860
P1433
P1476
Granulocyte macrophage colony- ...... in tumor-educated macrophages.
@en
P2093
Clay B Marsh
Gerard J Nuovo
Jennifer M Curry
Mahmood Khan
Periannan Kuppusamy
Ryan D Roberts
Tim D Eubank
P2860
P304
P356
10.1158/0008-5472.CAN-08-1405
P407
P577
2009-02-17T00:00:00Z