Interactions between inflammatory signals and the progesterone receptor in regulating gene expression in pregnant human uterine myocytes. - Wikidata - awgldk - TriplyDB

Interactions between inflammatory signals and the progesterone receptor in regulating gene expression in pregnant human uterine myocytes.

Interactions between inflammatory signals and the progesterone receptor in regulating gene expression in pregnant human uterine myocytes.

http://www.wikidata.org/entity/Q37296256

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Novel concepts on pregnancy clocks and alarms: redundancy and synergy in human parturition Progesterone-induced activation of membrane-bound progesterone receptors in murine macrophage cells Progesterone-mediated effects on gene expression and oocyte-cumulus complex transport in the mouse fallopian tube.The idiopathic preterm delivery methylation profile in umbilical cord blood DNA.Modeling hormonal and inflammatory contributions to preterm and term labor using uterine temporal transcriptomics.Progesterone and the Repression of Myometrial Inflammation: The Roles of MKP-1 and the AP-1 System The entry of fetal and amniotic fluid components into the uterine vessel circulation leads to sterile inflammatory processes during parturition.Progesterone action in the myometrium and decidua in preterm birth Molecular Regulation of Parturition: A Myometrial Perspective.The study of progesterone action in human myometrial explants.Cord blood hematopoietic cells from preterm infants display altered DNA methylation patterns.Expression of Matrix Metalloproteinases in the Mouse Uterus and Human Myometrium During Pregnancy, Labor, and Preterm Labor.The transcriptional repressor GATAD2B mediates progesterone receptor suppression of myometrial contractile gene expression.

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Interactions between inflammatory signals and the progesterone receptor in regulating gene expression in pregnant human uterine myocytes.

http://www.wikidata.org/entity/Q37296256

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on October 2012

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vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Interactions between inflammat ...... egnant human uterine myocytes.

@en

Interactions between inflammat ...... egnant human uterine myocytes.

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type

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Interactions between inflammat ...... egnant human uterine myocytes.

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Interactions between inflammat ...... egnant human uterine myocytes.

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prefLabel

Interactions between inflammat ...... egnant human uterine myocytes.

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Interactions between inflammat ...... egnant human uterine myocytes.

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J.1582-4934.2012.01567.X

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Journal of Cellular and Molecular Medicine

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Interactions between inflammat ...... egnant human uterine myocytes.

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Geraint Barton

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Mark R Johnson

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Matti Poutanen

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Phillip R Bennett

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Suren R Sooranna

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Vasso Terzidou

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Yun Lee

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P2860

Progestational agents for treating threatened or established preterm labour

Surfactant protein secreted by the maturing mouse fetal lung acts as a hormone that signals the initiation of parturition

Inflammatory aetiology of human myometrial activation tested using directed graphs

Reduced TRPC channel expression in psoriatic keratinocytes is associated with impaired differentiation and enhanced proliferation

Nuclear factor-kappa B localization and function within intrauterine tissues from term and preterm labor and cultured fetal membranes.

Negative interaction between the RelA(p65) subunit of NF-kappaB and the progesterone receptor.

5 Beta-dihydroprogesterone and steroid 5 beta-reductase decrease in association with human parturition at term.

Characterization of the myometrial transcriptome and biological pathways of spontaneous human labor at term.

A decline in the levels of progesterone receptor coactivators in the pregnant uterus at term may antagonize progesterone receptor function and contribute to the initiation of parturition.

Integration of endocrine and mechanical signals in the regulation of myometrial functions during pregnancy and labour.

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2487-2503

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10.1111/J.1582-4934.2012.01567.X

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