IRAK1-dependent signaling mediates mortality in polymicrobial sepsis. - Wikidata - awgldk - TriplyDB

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

http://www.wikidata.org/entity/Q37296875

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T cells down-regulate macrophage TNF production by IRAK1-mediated IL-10 expression and control innate hyperinflammation Syk Plays a Critical Role in the Expression and Activation of IRAK1 in LPS-Treated Macrophages.Cellular mosaicism for X-linked polymorphisms and IRAK1 expression presents a distinct phenotype and improves survival following sepsis Inherited human IRAK-1 deficiency selectively impairs TLR signaling in fibroblasts Intravenous infusion of mesenteric lymph from severe intraperitoneal infection rats causes lung injury in healthy rats.The IRAK-ERK-p67phox-Nox-2 axis mediates TLR4, 2-induced ROS production for IL-1β transcription and processing in monocytes Expression of X-linked Toll-like receptor 4 signaling genes in female vs. male neonates.Inhibition of IRAK1 Ubiquitination Determines Glucocorticoid Sensitivity for TLR9-Induced Inflammation in Macrophages.Inherent X-Linked Genetic Variability and Cellular Mosaicism Unique to Females Contribute to Sex-Related Differences in the Innate Immune Response.TRAF molecules in inflammation and inflammatory diseases.IL-1 signaling mediates intrauterine inflammation and chorio-decidua neutrophil recruitment and activation.Inhibition of interleukin-1 receptor-associated kinase 1 (IRAK1) as a therapeutic strategy

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IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

http://www.wikidata.org/entity/Q37296875

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on December 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

@en

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

@nl

type

label

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

@en

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

@nl

prefLabel

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

@en

IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

@nl

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IRAK1-dependent signaling mediates mortality in polymicrobial sepsis.

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James A Thomas

http://www.w3.org/2001/XMLSchema#string

Rachna Chandra

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Stephanie Federici

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Tripti Bishwas

http://www.w3.org/2001/XMLSchema#string

Zoltán H Németh

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Zoltán Spolarics

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P2860

Endotoxin tolerance impairs IL-1 receptor-associated kinase (IRAK) 4 and TGF-beta-activated kinase 1 activation, K63-linked polyubiquitination and assembly of IRAK1, TNF receptor-associated factor 6, and IkappaB kinase gamma and increases A20 expres

Toll-like receptor signalling

Extracellular administration of BCL2 protein reduces apoptosis and improves survival in a murine model of sepsis

Impaired cytokine signaling in mice lacking the IL-1 receptor-associated kinase

Augmented IL-10 production and redox-dependent signaling pathways in glucose-6-phosphate dehydrogenase-deficient mouse peritoneal macrophages

The role of interleukin 1 receptor-associated kinase-4 (IRAK-4) kinase activity in IRAK-4-mediated signaling

Tolerance induced via TLR2 and TLR4 in human dendritic cells: role of IRAK-1.

Role of interleukin-6 in mortality from and physiologic response to sepsis

Immunopathologic alterations in murine models of sepsis of increasing severity

Female X-chromosome mosaicism for NOX2 deficiency presents unique inflammatory phenotype and improves outcome in polymicrobial sepsis.

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1503-1512

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Edwin A. Deitch

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