Tumor suppressor PDCD4 modulates miR-184-mediated direct suppression of C-MYC and BCL2 blocking cell growth and survival in nasopharyngeal carcinoma.
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Therapeutic implications of Epstein-Barr virus infection for the treatment of nasopharyngeal carcinomaAlpha-enolase as a potential cancer prognostic marker promotes cell growth, migration, and invasion in gliomaExpression of circulating miRNAs associated with lymphocyte differentiation and activation in CLL-another piece in the puzzle.Artesunate suppresses the viability and mobility of prostate cancer cells through UCA1, the sponge of miR-184Knocking down CDK4 mediates the elevation of let-7c suppressing cell growth in nasopharyngeal carcinoma.MicroRNA profiling of ovarian granulosa cell tumours reveals novel diagnostic and prognostic markers.miR-150 modulates cisplatin chemosensitivity and invasiveness of muscle-invasive bladder cancer cells via targeting PDCD4 in vitroThe role of microRNAs in nasopharyngeal carcinoma.microRNA-184 functions as tumor suppressor in renal cell carcinoma.SPOCK1 as a potential cancer prognostic marker promotes the proliferation and metastasis of gallbladder cancer cells by activating the PI3K/AKT pathway.Histone demethylase RBP2 decreases miR-21 in blast crisis of chronic myeloid leukemia.Alpha-enolase promotes cell glycolysis, growth, migration, and invasion in non-small cell lung cancer through FAK-mediated PI3K/AKT pathwayMicroRNA-184 inhibits cell proliferation and invasion, and specifically targets TNFAIP2 in GliomaSelective inhibition of miR-21 by phage display screened peptide.MiR-506 suppresses tumor proliferation and invasion by targeting FOXQ1 in nasopharyngeal carcinomaGold nano-particles (AuNPs) carrying anti-EBV-miR-BART7-3p inhibit growth of EBV-positive nasopharyngeal carcinoma.Integrated analysis of microRNA regulatory network in nasopharyngeal carcinoma with deep sequencing.CDK4 and miR-15a comprise an abnormal automodulatory feedback loop stimulating the pathogenesis and inducing chemotherapy resistance in nasopharyngeal carcinoma.Oxidative stress response and Nrf2 signaling in aging.Broad targeting of resistance to apoptosis in cancer.Tumor invasion and metastasis regulated by microRNA-184 and microRNA-574-5p in small-cell lung cancer.miR-3188 regulates nasopharyngeal carcinoma proliferation and chemosensitivity through a FOXO1-modulated positive feedback loop with mTOR-p-PI3K/AKT-c-JUN.Genetic Variations in Key MicroRNAs are Associated With the Survival of Nonsmall Cell Lung Cancer.Reduction of microRNA-184 by E6 oncoprotein confers cisplatin resistance in lung cancer via increasing Bcl-2.Decreased P27 protein expression is correlated with the progression and poor prognosis of nasopharyngeal carcinoma.MicroRNA-1275 suppresses cell growth, and retards G1/S transition in human nasopharyngeal carcinoma by down-regulation of HOXB5.MicroRNA-based screens for synthetic lethal interactions with c-MycA directly negative interaction of miR-203 and ZEB2 modulates tumor stemness and chemotherapy resistance in nasopharyngeal carcinoma.miR‑150 inhibits proliferation and tumorigenicity via retarding G1/S phase transition in nasopharyngeal carcinoma.Identification of a TLR2-regulated gene signature associated with tumor cell growth in gastric cancer.Direct interaction between miR-203 and ZEB2 suppresses epithelial-mesenchymal transition signaling and reduces lung adenocarcinoma chemoresistance.miR-374a-CCND1-pPI3K/AKT-c-JUN feedback loop modulated by PDCD4 suppresses cell growth, metastasis, and sensitizes nasopharyngeal carcinoma to cisplatin.Downregulation of MicroRNA-21 in Colonic CD3+ T Cells in UC Remission.miR-16 induction after CDK4 knockdown is mediated by c-Myc suppression and inhibits cell growth as well as sensitizes nasopharyngeal carcinoma cells to chemotherapy.Hypoxia-induced PLOD2 promotes proliferation, migration and invasion via PI3K/Akt signaling in glioma.Noncoding RNAs Regulating p53 and c-Myc Signaling.Knockdown of c-Myc inhibits cell proliferation by negatively regulating the Cdk/Rb/E2F pathway in nasopharyngeal carcinoma cells.Tumor suppressor p53 induces miR-1915 processing to inhibit Bcl-2 in the apoptotic response to DNA damage.The clinical association of programmed cell death protein 4 (PDCD4) with solid tumors and its prognostic significance: a meta-analysis.Compounds From Celastraceae Targeting Cancer Pathways and Their Potential Application in Head and Neck Squamous Cell Carcinoma: A Review.
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Tumor suppressor PDCD4 modulates miR-184-mediated direct suppression of C-MYC and BCL2 blocking cell growth and survival in nasopharyngeal carcinoma.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 24 October 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@en
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@nl
type
label
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@en
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@nl
prefLabel
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@en
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@nl
P2093
P2860
P356
P1476
Tumor suppressor PDCD4 modulat ...... l in nasopharyngeal carcinoma.
@en
P2093
Chao Cheng
Huiling Yang
Menyang Zhao
Qiangyun Wu
Qiaofen Fu
Qingping Jiang
Shengni Hua
Weiyi Fang
Xiaobin Long
P2860
P356
10.1038/CDDIS.2013.376
P50
P577
2013-10-24T00:00:00Z