Sporadic Alzheimer's disease begins as episodes of brain ischemia and ischemically dysregulated Alzheimer's disease genes. - Wikidata - awgldk - TriplyDB

Sporadic Alzheimer's disease begins as episodes of brain ischemia and ischemically dysregulated Alzheimer's disease genes.

Sporadic Alzheimer's disease begins as episodes of brain ischemia and ischemically dysregulated Alzheimer's disease genes.

http://www.wikidata.org/entity/Q37301029

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Amidated and Ibuprofen-Conjugated Kyotorphins Promote Neuronal Rescue and Memory Recovery in Cerebral Hypoperfusion Dementia Model Amyloid-β and proinflammatory cytokines utilize a prion protein-dependent pathway to activate NADPH oxidase and induce cofilin-actin rods in hippocampal neurons Analysis of RNA from Alzheimer's Disease Post-mortem Brain Tissues.RAGE inhibition in microglia prevents ischemia-dependent synaptic dysfunction in an amyloid-enriched environment APP as a Protective Factor in Acute Neuronal Insults.Dysregulation of Amyloid-β Protein Precursor, β-Secretase, Presenilin 1 and 2 Genes in the Rat Selectively Vulnerable CA1 Subfield of Hippocampus Following Transient Global Brain Ischemia.Dysregulation of Autophagy, Mitophagy, and Apoptotic Genes in the Medial Temporal Lobe Cortex in an Ischemic Model of Alzheimer's Disease Synaptic Dysfunction in Alzheimer's Disease and Glaucoma: From Common Degenerative Mechanisms Toward Neuroprotection.Neuronal autophagy: self-eating or self-cannibalism in Alzheimer's disease.ABCA7 in Alzheimer's Disease.Amyloid-clearing proteins and their epigenetic regulation as a therapeutic target in Alzheimer's disease.From chronic cerebral hypoperfusion to Alzheimer-like brain pathology and neurodegeneration.Dysfunctional Sensory Modalities, Locus Coeruleus, and Basal Forebrain: Early Determinants that Promote Neuropathogenesis of Cognitive and Memory Decline and Alzheimer's Disease.Amyloid-beta disrupts calcium and redox homeostasis in brain endothelial cells.Hypoxia/ischemia activate processing of Amyloid Precursor Protein: impact of vascular dysfunction in the pathogenesis of Alzheimer's disease.Hypoxia Affects Neprilysin Expression Through Caspase Activation and an APP Intracellular Domain-dependent Mechanism.Optimized Model of Cerebral Ischemia In situ for the Long-Lasting Assessment of Hippocampal Cell Death.Pretreatment by evodiamine is neuroprotective in cerebral ischemia: up-regulated pAkt, pGSK3β, down-regulated NF-κB expression, and ameliorated BBB permeability.Impaired hypoxic tolerance in APP23 mice: a dysregulation of neuroprotective globin levels.Alpha-synuclein oligomerization in manganese-induced nerve cell injury in brain slices: a role of NO-mediated S-nitrosylation of protein disulfide isomerase.Protective effect of Tanshinone IIA against infarct size and increased HMGB1, NFκB, GFAP and apoptosis consequent to transient middle cerebral artery occlusion.Silymarin ameliorates memory deficits and neuropathological changes in mouse model of high-fat-diet-induced experimental dementia.Microglial Function during Glucose Deprivation: Inflammatory and Neuropsychiatric Implications.Oxygen and Glucose Deprivation Alter Synaptic Distribution of Tau Protein: The Role of Phosphorylation.Lipid signaling to membrane proteins: From second messengers to membrane domains and adapter-free endocytosis.Discrepancy in Expression of β-Secretase and Amyloid-β Protein Precursor in Alzheimer-Related Genes in the Rat Medial Temporal Lobe Cortex Following Transient Global Brain Ischemia.Tau Protein Dysfunction after Brain Ischemia The Hippocampal Neuro-Glio-Vascular Network: Metabolic Vulnerability and Potential Neurogenic Regeneration in Disease

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Sporadic Alzheimer's disease begins as episodes of brain ischemia and ischemically dysregulated Alzheimer's disease genes.

http://www.wikidata.org/entity/Q37301029

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article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on 22 March 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Sporadic Alzheimer's disease b ...... ted Alzheimer's disease genes.

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Sporadic Alzheimer's disease b ...... ted Alzheimer's disease genes.

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Sporadic Alzheimer's disease b ...... ted Alzheimer's disease genes.

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Sporadic Alzheimer's disease b ...... ted Alzheimer's disease genes.

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Sporadic Alzheimer's disease b ...... ted Alzheimer's disease genes.

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Sporadic Alzheimer's disease b ...... ted Alzheimer's disease genes.

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Molecular Neurobiology

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Sporadic Alzheimer's disease b ...... ated Alzheimer's disease genes

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Janusz Kocki

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Judyta Brzozowska

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Marzena Ułamek-Kozioł

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Mirosław Jabłoński

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Ryszard Maciejewski

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Ryszard Pluta

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Stanisław J Czuczwar

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Sławomir Januszewski

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Wanda Furmaga-Jabłońska

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P2860

Alzheimer's Disease

Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein

Soluble amyloid beta peptide concentration as a predictor of synaptic change in Alzheimer's disease

Hippocampal neuron loss exceeds amyloid plaque load in a transgenic mouse model of Alzheimer's disease

Alpha-synuclein and neurodegenerative diseases

Apolipoprotein E: a pathological chaperone protein in patients with cerebral and systemic amyloid

Presenilin-dependent gamma-secretase processing of beta-amyloid precursor protein at a site corresponding to the S3 cleavage of Notch

Presenilin-1 and intracellular calcium stores regulate neuronal glutamate uptake

Delayed, but prolonged increases in astrocytic clusterin (ApoJ) mRNA expression following acute cortical spreading depression in the rat: evidence for a role of clusterin in ischemic tolerance

Spatiotemporal changes of apolipoprotein E immunoreactivity and apolipoprotein E mRNA expression after transient middle cerebral artery occlusion in rat brain

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s12035-013-8439-1

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500-515

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scholarly article

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10.1007/S12035-013-8439-1

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3

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48

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Anna Bogucka-Kocka

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2013-03-22T00:00:00Z

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236071454

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23519520

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Alzheimer's disease

regulation of gene expression

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3825141

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