Novel isoforms of the sodium channels Nav1.8 and Nav1.5 are produced by a conserved mechanism in mouse and rat.
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Alternative RNA splicing: contribution to pain and potential therapeutic strategyCloning and expression of the two new variants of Nav1.5/SCN5A in rat brainThe expression of ELK transcription factors in adult DRG: Novel isoforms, antisense transcripts and upregulation by nerve damageMultiple Nav1.5 isoforms are functionally expressed in the brain and present distinct expression patterns compared with cardiac Nav1.5.Characterisation of the nociceptive phenotype of suppressible galanin overexpressing transgenic mice.Exon 11 skipping of SCN10A coding for voltage-gated sodium channels in dorsal root gangliaThe generation of knock-in mice expressing fluorescently tagged galanin receptors 1 and 2.Identification of the sensory neuron specific regulatory region for the mouse gene encoding the voltage-gated sodium channel NaV1.8.A novel Na+ channel splice form contributes to the regulation of an androgen-dependent social signalMice deficient for galanin receptor 2 have decreased neurite outgrowth from adult sensory neurons and impaired pain-like behaviourThe sodium channel Nav1.5a is the predominant isoform expressed in adult mouse dorsal root ganglia and exhibits distinct inactivation properties from the full-length Nav1.5 channel.Intra-neural administration of fractalkine attenuates neuropathic pain-related behaviourAssociation of SCN10A Polymorphisms with the Recurrence of Atrial Fibrillation after Catheter Ablation in a Chinese Han Population.Biology of cardiac sodium channel Nav1.5 expression.Recent developments regarding voltage-gated sodium channel blockers for the treatment of inherited and acquired neuropathic pain syndromes.Biophysical properties of human Na v1.7 splice variants and their regulation by protein kinase A.Novel mRNA isoforms of the sodium channels Na(v)1.2, Na(v)1.3 and Na(v)1.7 encode predicted two-domain, truncated proteins.Targeted disruption of the orphan receptor Gpr151 does not alter pain-related behaviour despite a strong induction in dorsal root ganglion expression in a model of neuropathic pain.Modulation of Nav1.5 channel function by an alternatively spliced sequence in the DII/DIII linker region.Pyramidal cells of rodent presubiculum express a tetrodotoxin-insensitive Na+ current.Molecular expression of multiple Nav1.5 splice variants in the frontal lobe of the human brain.Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Nav1.8 and Nav1.9 null mice.
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P2860
Novel isoforms of the sodium channels Nav1.8 and Nav1.5 are produced by a conserved mechanism in mouse and rat.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 26 March 2004
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@en
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@nl
type
label
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@en
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@nl
prefLabel
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@en
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@nl
P2093
P2860
P356
P1476
Novel isoforms of the sodium c ...... ed mechanism in mouse and rat.
@en
P2093
David Wynick
Fiona E Holmes
Niall C H Kerr
P2860
P304
24826-24833
P356
10.1074/JBC.M401281200
P407
P577
2004-03-26T00:00:00Z