Pivotal role for TGF-beta in infectious heart disease: The case of Trypanosoma cruzi infection and consequent Chagasic myocardiopathy.
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Scrutinizing the Biomarkers for the Neglected Chagas Disease: How Remarkable!The Dialogue of the Host-Parasite Relationship: Leishmania spp. and Trypanosoma cruzi InfectionOral administration of GW788388, an inhibitor of transforming growth factor beta signaling, prevents heart fibrosis in Chagas diseasePathogenesis of chagas' disease: parasite persistence and autoimmunityRegulatory T cells phenotype in different clinical forms of Chagas' diseaseBiomarkers in Trypanosoma cruzi-infected and uninfected individuals with varying severity of cardiomyopathy in Santa Cruz, Bolivia.Effects of omega-3 polyunsaturated fatty acid supplementation in patients with chronic chagasic cardiomyopathy: study protocol for a randomized controlled trial.Circulating serum markers and QRS scar score in Chagas cardiomyopathy.Differential Expression of Matrix Metalloproteinases 2, 9 and Cytokines by Neutrophils and Monocytes in the Clinical Forms of Chagas Disease.Trypanosoma cruzi High Mobility Group B (TcHMGB) can act as an inflammatory mediator on mammalian cellsGap junctions and chagas diseasePharmacological inhibition of transforming growth factor beta signaling decreases infection and prevents heart damage in acute Chagas' diseaseEvasion of immune responses by Trypanosoma cruzi, the etiological agent of Chagas disease.The TGF-β pathway as an emerging target for Chagas disease therapy.Contribution of myeloid cell subsets to liver fibrosis in parasite infection.Inhibition of cyclooxygenase-1 and cyclooxygenase-2 impairs Trypanosoma cruzi entry into cardiac cells and promotes differential modulation of the inflammatory response.Characterization of Trypanosoma cruzi infectivity, proliferation, and cytokine patterns in gut and pancreatic epithelial cells maintained in vitro.Synergic and antagonistic relationship between MMP-2 and MMP-9 with fibrosis and inflammation in Chagas' cardiomyopathy.Priming astrocytes with TNF enhances their susceptibility to Trypanosoma cruzi infection and creates a self-sustaining inflammatory milieu.Therapeutic effects of sphingosine kinase inhibitor N,N-dimethylsphingosine (DMS) in experimental chronic Chagas disease cardiomyopathy.TGF-β receptor type II costameric localization in cardiomyocytes and host cell TGF-β response is disrupted by Trypanosoma cruzi infection.Gap junction reduction in cardiomyocytes following transforming growth factor-beta treatment and Trypanosoma cruzi infection.Chronic indeterminate phase of Chagas' disease: mitochondrial involvement in infection with two strains.Blocking pathogen entry into the cell: the future of infectious disease treatment and control?Circulating Plasma MicroRNA-208a as Potential Biomarker of Chronic Indeterminate Phase of Chagas Disease.
P2860
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P2860
Pivotal role for TGF-beta in infectious heart disease: The case of Trypanosoma cruzi infection and consequent Chagasic myocardiopathy.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 19 October 2008
@en
vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Pivotal role for TGF-beta in i ...... quent Chagasic myocardiopathy.
@en
Pivotal role for TGF-beta in i ...... quent Chagasic myocardiopathy.
@nl
type
label
Pivotal role for TGF-beta in i ...... quent Chagasic myocardiopathy.
@en
Pivotal role for TGF-beta in i ...... quent Chagasic myocardiopathy.
@nl
prefLabel
Pivotal role for TGF-beta in i ...... quent Chagasic myocardiopathy.
@en
Pivotal role for TGF-beta in i ...... quent Chagasic myocardiopathy.
@nl
P2093
P1476
Pivotal role for TGF-beta in i ...... equent Chagasic myocardiopathy
@en
P2093
Maria de Nazaré C Soeiro
Mariana C Waghabi
Michelle Keramidas
Tania C Araújo-Jorge
P304
P356
10.1016/J.CYTOGFR.2008.08.002
P577
2008-10-19T00:00:00Z