NADPH oxidase drives cytokine and neurotoxin release from microglia and macrophages in response to HIV-Tat.
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The cross-talk of HIV-1 Tat and methamphetamine in HIV-associated neurocognitive disordersOxidative stress and cerebral endothelial cells: regulation of the blood-brain-barrier and antioxidant based interventionsRoles and functions of HIV-1 Tat protein in the CNS: an overviewA growth factor attenuates HIV-1 Tat and morphine induced damage to human neurons: implication in HIV/AIDS-drug abuse casesHIV-1 Tat co-operates with IFN-gamma and TNF-alpha to increase CXCL10 in human astrocytes.Brain dysfunction in the era of combination antiretroviral therapy: implications for the treatment of the aging population of HIV-infected individualsCooperative induction of CXCL10 involves NADPH oxidase: Implications for HIV dementia.Crosstalk between HDAC6 and Nox2-based NADPH oxidase mediates HIV-1 Tat-induced pro-inflammatory responses in astrocytes.NOX activity is increased in mild cognitive impairmentIbudilast, a pharmacologic phosphodiesterase inhibitor, prevents human immunodeficiency virus-1 Tat-mediated activation of microglial cellsMolecular mechanisms involving sigma receptor-mediated induction of MCP-1: implication for increased monocyte transmigration.Herpes simplex virus induces neural oxidative damage via microglial cell Toll-like receptor-2.Nox-4-dependent nuclear H2O2 drives DNA oxidation resulting in 8-OHdG as urinary biomarker and hemangioendothelioma formationPrimary murine microglia are resistant to nitric oxide inhibition of indoleamine 2,3-dioxygenase.Modulation of experimental herpes encephalitis-associated neurotoxicity through sulforaphane treatment.Cocaine potentiates cathepsin B secretion and neuronal apoptosis from HIV-infected macrophages.HIV neuropathogenesis: a tight rope walk of innate immunity.Methionine sulfoxide reductase A negatively controls microglia-mediated neuroinflammation via inhibiting ROS/MAPKs/NF-κB signaling pathways through a catalytic antioxidant functionReactive oxygen species drive herpes simplex virus (HSV)-1-induced proinflammatory cytokine production by murine microglia.HIV-1, reactive oxygen species, and vascular complicationsNOX2 deficiency attenuates markers of adiposopathy and brain injury induced by high-fat diet.Oxidative stress and the HIV-infected brain proteome.CNS inflammation and macrophage/microglial biology associated with HIV-1 infection.Microglial activation and chronic neurodegeneration.Targeting microglia-mediated neurotoxicity: the potential of NOX2 inhibitors.Myeloid-specific deletion of NOX2 prevents the metabolic and neurologic consequences of high fat diet.Microglia antioxidant systems and redox signalling.Fate of microglia during HIV-1 infection: From activation to senescence?HIV-1 Tat C modulates NOX2 and NOX4 expressions through miR-17 in a human microglial cell line.ROS upregulation during the early phase of retroviral infection plays an important role in viral establishment in the host cell.Oligomeric Aβ-induced microglial activation is possibly mediated by NADPH oxidase.Childhood maltreatment and HIV-associated neurocognitive disorders share similar pathophysiology: a potential sensitisation mechanism?Molecular mechanisms underlying the involvement of the sigma-1 receptor in methamphetamine-mediated microglial polarization.Altered expression pattern of Nrf2/HO-1 axis during accelerated-senescence in HIV-1 transgenic rat.HIV-Tat elicits microglial glutamate release: role of NAPDH oxidase and the cystine-glutamate antiporter.Flipping the switches: CD40 and CD45 modulation of microglial activation states in HIV associated dementia (HAD).15-deoxy-Δ¹²,¹⁴ -prostaglandin J₂ inhibits human immunodeficiency virus-1 tat-induced monocyte chemoattractant protein-1/CCL2 production by blocking the extracellular signal-regulated kinase-1/2 signaling pathway independently of peroxisome proliferNeonatal morphine administration leads to changes in hippocampal BDNF levels and antioxidant enzyme activity in the adult life of rats.
P2860
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P2860
NADPH oxidase drives cytokine and neurotoxin release from microglia and macrophages in response to HIV-Tat.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on February 2009
@en
vedecký článok
@sk
vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
NADPH oxidase drives cytokine ...... phages in response to HIV-Tat.
@en
NADPH oxidase drives cytokine ...... phages in response to HIV-Tat.
@nl
type
label
NADPH oxidase drives cytokine ...... phages in response to HIV-Tat.
@en
NADPH oxidase drives cytokine ...... phages in response to HIV-Tat.
@nl
prefLabel
NADPH oxidase drives cytokine ...... phages in response to HIV-Tat.
@en
NADPH oxidase drives cytokine ...... phages in response to HIV-Tat.
@nl
P2093
P2860
P921
P356
P1476
NADPH oxidase drives cytokine ...... ophages in response to HIV-Tat
@en
P2093
Annadora J Bruce-Keller
Jadwiga Turchan-Cholewo
R M Charlotte Gorospe
Sunita Gupta
Vanessa M Dimayuga
P2860
P304
P356
10.1089/ARS.2008.2097
P50
P577
2009-02-01T00:00:00Z