Reduction of circulating soluble Flt-1 alleviates preeclampsia-like symptoms in a mouse model.
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Therapeutic potential of statins and the induction of heme oxygenase-1 in preeclampsiaThe renal circulation in normal pregnancy and preeclampsia: is there a place for relaxin?Is inflammation the cause of pre-eclampsia?New approaches for managing preeclampsia: clues from clinical and basic researchADAMTS13 Endopeptidase Protects against Vascular Endothelial Growth Factor Inhibitor-Induced Thrombotic Microangiopathy.Carbon monoxide prevents hypertension and proteinuria in an adenovirus sFlt-1 preeclampsia-like mouse model.Sildenafil Inhibits Advanced Glycation End Products-induced sFlt-1 Release Through Upregulation of Heme Oxygenase-1.Recent insights into the pathophysiology of preeclampsia.Mitochondrial Channel Opener Diazoxide Attenuates Hypoxia-Induced sFlt-1 Release in Human Choriocarcinoma Cells.In vivo experiments reveal the good, the bad and the ugly faces of sFlt-1 in pregnancy.Design of growth factor sequestering biomaterialsPlacental ischemia induces changes in gene expression in chorionic tissue.Characterisation of syncytiotrophoblast vesicles in normal pregnancy and pre-eclampsia: expression of Flt-1 and endoglin.Reduced uterine perfusion pressure induces hypertension in the pregnant mouse.Angiogenic factors and preeclampsia.Evaluation of association of maternal IL-10 polymorphisms with risk of preeclampsia by A meta-analysis.SFlt-1 elevates blood pressure by augmenting endothelin-1-mediated vasoconstriction in mice.Endometrial VEGF induces placental sFLT1 and leads to pregnancy complicationsPreeclampsia, a disease of the maternal endothelium: the role of antiangiogenic factors and implications for later cardiovascular disease.Exposure to experimental preeclampsia in mice enhances the vascular response to future injuryFull-length human placental sFlt-1-e15a isoform induces distinct maternal phenotypes of preeclampsia in mice.Nitric oxide generation affects pro- and anti-angiogenic growth factor expression in primary human trophoblast.Classical Complement Pathway Activation in the Kidneys of Women With Preeclampsia.Transcriptionally active syncytial aggregates in the maternal circulation may contribute to circulating soluble fms-like tyrosine kinase 1 in preeclampsia.Maternal plasma concentrations of angiogenic/anti-angiogenic factors are of prognostic value in patients presenting to the obstetrical triage area with the suspicion of preeclampsia.Endothelin: key mediator of hypertension in preeclampsia.Linking placental ischemia and hypertension in preeclampsia: role of endothelin 1.The elevation in circulating anti-angiogenic factors is independent of markers of neutrophil activation in preeclampsiaEndothelin as a final common pathway in the pathophysiology of preeclampsia: therapeutic implicationsThe fetal origins of the metabolic syndrome: can we intervene?Angiogenic and immune signatures in plasma of young relatives at familial high-risk for psychosis and first-episode patients: A preliminary study.Removal of Soluble Fms-Like Tyrosine Kinase-1 by Dextran Sulfate Apheresis in Preeclampsia.Endoglin: a critical mediator of cardiovascular healthSoluble fms-like tyrosine kinase 1 promotes angiotensin II sensitivity in preeclampsia.Mechanisms and potential therapies for preeclampsiaPlacental Ischemia and Resultant Phenotype in Animal Models of Preeclampsia.Aspirin for prevention of preeclampsia in lupus pregnancy.Pre-eclampsia, severe pre-eclampsia and hemolysis, elevated liver enzymes and low platelets syndrome: what is new?Involvement of the heme oxygenase system in the development of preeclampsia and as a possible therapeutic target.Autocrine activity of soluble Flt-1 controls endothelial cell function and angiogenesis.
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P2860
Reduction of circulating soluble Flt-1 alleviates preeclampsia-like symptoms in a mouse model.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 16 June 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Reduction of circulating solub ...... ike symptoms in a mouse model.
@en
Reduction of circulating solub ...... ike symptoms in a mouse model.
@nl
type
label
Reduction of circulating solub ...... ike symptoms in a mouse model.
@en
Reduction of circulating solub ...... ike symptoms in a mouse model.
@nl
prefLabel
Reduction of circulating solub ...... ike symptoms in a mouse model.
@en
Reduction of circulating solub ...... ike symptoms in a mouse model.
@nl
P2093
P2860
P1476
Reduction of circulating solub ...... ike symptoms in a mouse model.
@en
P2093
Achim D Gruber
Andrey Ch da Costa Gonzalves
Asif Ahmed
Astrid Bergmann
Gerhard Christofori
Herbert A Weich
Hermann-Josef Gröne
Melissa Cudmore
Petra Wittschen
Volkmar Gross
P2860
P304
P356
10.1111/J.1582-4934.2009.00820.X
P50
P577
2009-06-16T00:00:00Z