Thymosin β4 and its degradation product, Ac-SDKP, are novel reparative factors in renal fibrosis. - Wikidata - awgldk - TriplyDB

Thymosin β4 and its degradation product, Ac-SDKP, are novel reparative factors in renal fibrosis.

Thymosin β4 and its degradation product, Ac-SDKP, are novel reparative factors in renal fibrosis.

http://www.wikidata.org/entity/Q37315924

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Key fibrogenic mediators: old players. Renin–angiotensin system Potential role of thymosin Beta 4 in liver fibrosis N-acetyl-seryl-aspartyl-lysyl-proline: a valuable endogenous anti-fibrotic peptide for combating kidney fibrosis in diabetes Hepatic stellate cells express thymosin Beta 4 in chronically damaged liver Thymosin beta-4 regulates activation of hepatic stellate cells via hedgehog signaling.Effects of exogenous thymosin β4 on carbon tetrachloride-induced liver injury and fibrosis Thymosin Beta 4 protects mice from monocrotaline-induced pulmonary hypertension and right ventricular hypertrophy.The anti-inflammatory peptide Ac-SDKP is released from thymosin-β4 by renal meprin-α and prolyl oligopeptidase.A Tobacco-Derived Thymosin β4 Concatemer Promotes Cell Proliferation and Wound Healing in Mice Loss of endogenous thymosin β4 accelerates glomerular disease.Renal disease pathophysiology and treatment: contributions from the rat.Combination treatment with N-acetyl-seryl-aspartyl-lysyl-proline and tissue plasminogen activator provides potent neuroprotection in rats after stroke The biological significance of angiotensin-converting enzyme inhibition to combat kidney fibrosis.Causes and pathogenesis of focal segmental glomerulosclerosis.Advances in the basic and clinical applications of thymosin β4.FGFR1 is critical for the anti-endothelial mesenchymal transition effect of N-acetyl-seryl-aspartyl-lysyl-proline via induction of the MAP4K4 pathway.Suppression of tumor growth in mice by rationally designed pseudopeptide inhibitors of fibroblast activation protein and prolyl oligopeptidase.Preventive and therapeutic effects of thymosin β4 N-terminal fragment Ac-SDKP in the bleomycin model of pulmonary fibrosis.Effect of Antifibrotic MicroRNAs Crosstalk on the Action of N-acetyl-seryl-aspartyl-lysyl-proline in Diabetes-related Kidney Fibrosis Thymosin-β4 inhibits proliferation and induces apoptosis of hepatic stellate cells through PI3K/AKT pathway.Thymosin β4 alleviates renal fibrosis and tubular cell apoptosis through TGF-β pathway inhibition in UUO rat models.Fibroblast activation protein-α in fibrogenic disorders and cancer: more than a prolyl-specific peptidase?The role of thymosin-β4 in kidney disease.Thymosin β4 Deficiency Exacerbates Renal and Cardiac Injury in Angiotensin-II-Induced Hypertension.Mechanisms in hypertension and target organ damage: Is the role of the thymus key? (Review).Potential role of thymosin beta 4 in the treatment of nonalcoholic fatty liver disease.

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Thymosin β4 and its degradation product, Ac-SDKP, are novel reparative factors in renal fibrosis.

http://www.wikidata.org/entity/Q37315924

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 05 June 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Thymosin β4 and its degradatio ...... ive factors in renal fibrosis.

@en

Thymosin β4 and its degradatio ...... ive factors in renal fibrosis.

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type

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Thymosin β4 and its degradatio ...... ive factors in renal fibrosis.

@en

Thymosin β4 and its degradatio ...... ive factors in renal fibrosis.

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prefLabel

Thymosin β4 and its degradatio ...... ive factors in renal fibrosis.

@en

Thymosin β4 and its degradatio ...... ive factors in renal fibrosis.

@nl

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Kidney International

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Thymosin β4 and its degradatio ...... tive factors in renal fibrosis

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Agnes B Fogo

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Bongkwon Chun

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Diclehan Orhan

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Hai-Chun Yang

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Li-Jun Ma

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Naj Kazi

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Nour-Eddine Rhaleb

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Oscar A Carretero

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Sebastian A Potthoff

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Tyler J Brolin

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P2860

Binding of PAI-1 to endothelial cells stimulated by thymosin beta4 and modulation of their fibrinolytic potential

De novo cardiomyocytes from within the activated adult heart after injury

Thymosin beta4 activates integrin-linked kinase and promotes cardiac cell migration, survival and cardiac repair

Renal and metabolic clearance of N-acetyl-seryl-aspartyl-lysyl-proline (AcSDKP) during angiotensin-converting enzyme inhibition in humans

Ureteral obstruction as a model of renal interstitial fibrosis and obstructive nephropathy.

Antifibrotic effects of N-acetyl-seryl-aspartyl-Lysyl-proline on the heart and kidney in aldosterone-salt hypertensive rats.

Proteomic patterns and prediction of glomerulosclerosis and its mechanisms.

N-acetyl-seryl-aspartyl-lysyl-proline attenuates renal injury and dysfunction in hypertensive rats with reduced renal mass: council for high blood pressure research

Significance of thymosin β4 and implication of PINCH-1-ILK-α-parvin (PIP) complex in human dilated cardiomyopathy

Thymosin beta4: structure, function, and biological properties supporting current and future clinical applications.

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scholarly article

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10.1038/KI.2013.209

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Timo T. Myöhänen

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2013-06-05T00:00:00Z

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