Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice. - Wikidata - awgldk - TriplyDB

Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

http://www.wikidata.org/entity/Q37318166

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Defective i6A37 modification of mitochondrial and cytosolic tRNAs results from pathogenic mutations in TRIT1 and its substrate tRNA Interferons, signal transduction pathways, and the central nervous system.High Glucose Induces Down-Regulated GRIM-19 Expression to Activate STAT3 Signaling and Promote Cell Proliferation in Cell Culture Grim19 Attenuates DSS Induced Colitis in an Animal Model.GRIM19 ameliorates acute graft-versus-host disease (GVHD) by modulating Th17 and Treg cell balance through down-regulation of STAT3 and NF-AT activation.Mitochondrial GRIM-19 as a potential therapeutic target for STAT3-dependent carcinogenesis of gastric cancer.FGFR4 polymorphic alleles modulate mitochondrial respiration: A novel target for somatostatin analog action in pituitary tumors GRIM-19: A master regulator of cytokine induced tumor suppression, metastasis and energy metabolism.Electron leak from NDUFA13 within mitochondrial complex I attenuates ischemia-reperfusion injury via dimerized STAT3.The Oncojanus Paradigm of Respiratory Complex I.

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

http://www.wikidata.org/entity/Q37318166

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 21 October 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

@en

Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

@en

Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

@nl

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PNAS.1303760110

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Proceedings of the National Academy of Sciences of the United States of America

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Monoallelic loss of tumor suppressor GRIM-19 promotes tumorigenesis in mice.

@en

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Daniel J Lindner

http://www.w3.org/2001/XMLSchema#string

Dhananjaya V Kalvakolanu

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Ellen Rorke

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Gary M Fiskum

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Raghava Munivenkatappa

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Sausan Jaber

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Shreeram C Nallar

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Sudhakar Kalakonda

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Susan K Keay

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Hallmarks of Cancer: The Next Generation

Identification of GRIM-19, a novel cell death-regulatory gene induced by the interferon-beta and retinoic acid combination, using a genetic approach

p53 negatively regulates transcription of the pyruvate dehydrogenase kinase Pdk2

The cell death regulator GRIM-19 is an inhibitor of signal transducer and activator of transcription 3

Immunodeficiency and chronic myelogenous leukemia-like syndrome in mice with a targeted mutation of the ICSBP gene

Chronic CSE treatment induces the growth of normal oral keratinocytes via PDK2 upregulation, increased glycolysis and HIF1α stabilization

Viral interferon regulatory factor 1 of Kaposi's sarcoma-associated herpesvirus interacts with a cell death regulator, GRIM19, and inhibits interferon/retinoic acid-induced cell death.

GRIM-19, a death-regulatory gene product, suppresses Stat3 activity via functional interaction

GRIM-19, a cell death regulatory protein, is essential for assembly and function of mitochondrial complex I

Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor

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scholarly article

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10.1073/PNAS.1303760110

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2013-10-21T00:00:00Z

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