Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
about
Deep congenic analysis identifies many strong, context-dependent QTLs, one of which, Slc35b4, regulates obesity and glucose homeostasisHybrid mouse diversity panel: a panel of inbred mouse strains suitable for analysis of complex genetic traits.Effect of chromosome substitution on intrinsic exercise capacity in mice.Body fat distribution and organ weights of 14 common strains and a 22-strain consomic panel of ratsGenetic control of obesity and gut microbiota composition in response to high-fat, high-sucrose diet in mice.Genetic determinants of atherosclerosis, obesity, and energy balance in consomic mice.Chromosome substitution strain assessment of a Huntington's disease modifier locusThe maternal-age-associated risk of congenital heart disease is modifiable.Chromosomal assignment of quantitative trait loci influencing baseline circulating total cholesterol level in male laboratory mice: report of a consomic strain survey and comparison with published results.Contrasting genetic architectures in different mouse reference populations used for studying complex traits.Transcriptional and Linkage Analyses Identify Loci that Mediate the Differential Macrophage Response to Inflammatory Stimuli and Infection.Chromosome substitution strains: gene discovery, functional analysis, and systems studies.The genetic basis for individual differences in mRNA splicing and APOBEC1 editing activity in murine macrophages.A modified vaccinia Ankara vaccine vector expressing a mosaic H5 hemagglutinin reduces viral shedding in rhesus macaques.An improved mouse model that rapidly develops fibrosis in non-alcoholic steatohepatitis.High-Fat Diet-Induced Complement Activation Mediates Intestinal Inflammation and Neoplasia, Independent of Obesity.Comments on Point:Counterpoint: The dominant contributor to systemic hypertension: Chronic activation of the sympathetic nervous system vs. Activation of the intrarenal renin-angiotensin system. Activated intrarenal renin-angiotensin system is correH55N polymorphism as a likely cause of variation in citrate synthase activity of mouse skeletal muscle.Genetics of metabolic syndrome: potential clues from wild-derived inbred mouse strains.Genetic control of obesity, glucose homeostasis, dyslipidemia and fatty liver in a mouse model of diet-induced metabolic syndrome.
P2860
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P2860
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 03 February 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@en
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@nl
type
label
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@en
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@nl
prefLabel
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@en
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@nl
P2093
P2860
P50
P1433
P1476
Genetic resistance to diet-induced obesity in chromosome substitution strains of mice.
@en
P2093
Andrew Kirby
Annie E Baskin-Hill
Colleen M Croniger
David S Sinasac
E J Kulbokas
Jonathan B Singer
Joseph H Nadeau
Lindsay C Burrage
P2860
P2888
P304
P356
10.1007/S00335-010-9247-9
P577
2010-02-03T00:00:00Z