Inhibiting Amadori-modified albumin formation improves biomarkers of podocyte damage in diabetic rats. - Wikidata - awgldk - TriplyDB

Inhibiting Amadori-modified albumin formation improves biomarkers of podocyte damage in diabetic rats.

Inhibiting Amadori-modified albumin formation improves biomarkers of podocyte damage in diabetic rats.

http://www.wikidata.org/entity/Q37319338

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Hypoxia inducible factor-1α mediates the profibrotic effect of albumin in renal tubular cells.Growth arrest specific 2-like protein 1 expression is upregulated in podocytes through advanced glycation end-products.

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Inhibiting Amadori-modified albumin formation improves biomarkers of podocyte damage in diabetic rats.

http://www.wikidata.org/entity/Q37319338

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 17 September 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

@en

Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

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type

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Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

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Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

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Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

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Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

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Physiological Reports

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Inhibiting Amadori-modified al ...... ocyte damage in diabetic rats.

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Clyde W Shearman

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Margo P Cohen

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P2860

cDNA cloning and sequence analysis of beta ig-h3, a novel gene induced in a human adenocarcinoma cell line after treatment with transforming growth factor-beta

beta ig-h3: a transforming growth factor-beta-responsive gene encoding a secreted protein that inhibits cell attachment in vitro and suppresses the growth of CHO cells in nude mice

Glycated albumin stimulation of PKC-beta activity is linked to increased collagen IV in mesangial cells

Proteinuria and the expression of the podocyte slit diaphragm protein, nephrin, in diabetic nephropathy: effects of angiotensin converting enzyme inhibition

Beta IG-H3, a novel secretory protein inducible by transforming growth factor-beta, is present in normal skin and promotes the adhesion and spreading of dermal fibroblasts in vitro

Inhibition of NADPH oxidase prevents advanced glycation end product-mediated damage in diabetic nephropathy through a protein kinase C-alpha-dependent pathway

Dysregulated nephrin in diabetic nephropathy of type 2 diabetes: a cross sectional study

Dedifferentiation of immortalized human podocytes in response to transforming growth factor-β: a model for diabetic podocytopathy.

Nephrin in experimental glomerular disease.

Glycated albumin stimulates TGF-beta 1 production and protein kinase C activity in glomerular endothelial cells.

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scholarly article

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10.1002/PHY2.83

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2013-09-17T00:00:00Z

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