The brain subfornical organ mediates leptin-induced increases in renal sympathetic activity but not its metabolic effects
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Neuroendorine and Epigentic Mechanisms Subserving Autonomic Imbalance and HPA Dysfunction in the Metabolic SyndromeResistin, an Adipokine with Non-Generalized Actions on Sympathetic Nerve ActivityObesity-induced hepatic steatosis is mediated by endoplasmic reticulum stress in the subfornical organ of the brainObesity-Induced Hypertension: Brain Signaling Pathways.Obesity-associated hypertension: recent progress in deciphering the pathogenesisObesity in kidney disease: A heavyweight opponent.Endogenous leptin contributes to baroreflex suppression within the solitary tract nucleus of aged rats.Control of respiratory and cardiovascular functions by leptinObesity-induced hypertension: interaction of neurohumoral and renal mechanismsThe Spleen: A Hub Connecting Nervous and Immune Systems in Cardiovascular and Metabolic DiseasesRole of the Paraventricular Nucleus of the Hypothalamus in the Sympathoexcitatory Effects of LeptinLeptin Mediates High-Fat Diet Sensitization of Angiotensin II-Elicited Hypertension by Upregulating the Brain Renin-Angiotensin System and Inflammation.Central Renin-Angiotensin System Activation and Inflammation Induced by High-Fat Diet Sensitize Angiotensin II-Elicited Hypertension.Selective leptin resistance revisited.Direct and indirect effects of leptin on adipocyte metabolism.Neural Control of Non-vasomotor Organs in Hypertension.Angiotensin AT1A receptors on leptin receptor-expressing cells control resting metabolism.Control of energy balance by the brain renin-angiotensin system.Should the sympathetic nervous system be a target to improve cardiometabolic risk in obesity?Renal Nerves and Long-Term Control of Arterial Pressure.Central neural control of the cardiovascular system: current perspectives.Leptin as a Mediator of Obesity-Induced Hypertension.Factors Responsible for Obesity-Related Hypertension.Recent Advances in Neurogenic Hypertension: Dietary Salt, Obesity, and Inflammation.Angiotensin type 1a receptors in the forebrain subfornical organ facilitate leptin-induced weight loss through brown adipose tissue thermogenesisA synthetic luciferin improves in vivo bioluminescence imaging of gene expression in cardiovascular brain regionsObesity-related glomerulopathy: pathogenesis, pathologic, clinical characteristics and treatment.
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The brain subfornical organ mediates leptin-induced increases in renal sympathetic activity but not its metabolic effects
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 January 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The brain subfornical organ me ...... but not its metabolic effects
@en
The brain subfornical organ me ...... but not its metabolic effects.
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type
label
The brain subfornical organ me ...... but not its metabolic effects
@en
The brain subfornical organ me ...... but not its metabolic effects.
@nl
prefLabel
The brain subfornical organ me ...... but not its metabolic effects
@en
The brain subfornical organ me ...... but not its metabolic effects.
@nl
P2093
P2860
P1433
P1476
The brain subfornical organ me ...... but not its metabolic effects
@en
P2093
Allyn L Mark
Colin N Young
Donald A Morgan
Robin L Davisson
Scott D Butler
P2860
P304
P356
10.1161/HYPERTENSIONAHA.111.00405
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P577
2013-01-28T00:00:00Z