Fine mapping of "mini-muscle," a recessive mutation causing reduced hindlimb muscle mass in mice - Wikidata - awgldk - TriplyDB

Fine mapping of "mini-muscle," a recessive mutation causing reduced hindlimb muscle mass in mice

Fine mapping of "mini-muscle," a recessive mutation causing reduced hindlimb muscle mass in mice

http://www.wikidata.org/entity/Q37324197

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The biological control of voluntary exercise, spontaneous physical activity and daily energy expenditure in relation to obesity: human and rodent perspectives Glycogen storage and muscle glucose transporters (GLUT-4) of mice selectively bred for high voluntary wheel running.QTL underlying voluntary exercise in mice: interactions with the "mini muscle" locus and sex.Variation in within-bone stiffness measured by nanoindentation in mice bred for high levels of voluntary wheel running.Differential skeletal muscle proteome of high- and low-active mice Behavioral traits are affected by selective breeding for increased wheel-running behavior in mice.Strain screen and haplotype association mapping of wheel running in inbred mouse strains.Driven to be inactive? The genetics of physical activity A novel intronic single nucleotide polymorphism in the myosin heavy polypeptide 4 gene is responsible for the mini-muscle phenotype characterized by major reduction in hind-limb muscle mass in mice Genetic architecture of voluntary exercise in an advanced intercross line of mice.Current understanding of the genetic basis for physical activity Sex-specific heterosis in line crosses of mice selectively bred for high locomotor activity QTL Analysis of Type I and Type IIA Fibers in Soleus Muscle in a Cross between LG/J and SM/J Mouse Strains.Genetic approaches in comparative and evolutionary physiology.Determinants of intra-specific variation in basal metabolic rate.Gene expression profiling of gastrocnemius of "minimuscle" mice.High-saturated fat-sucrose feeding affects lactation energetics in control mice and mice selectively bred for high wheel-running behavior.Differential gene expression in high- and low-active inbred mice.Fine-mapping of genes determining extrafusal fiber properties in murine soleus muscle.Expression of angiogenic regulators and skeletal muscle capillarity in selectively bred high aerobic capacity mice.Selective breeding as a tool to probe skeletal response to high voluntary locomotor activity in mice.Genetic variations and physical activity as determinants of limb bone morphology: an experimental approach using a mouse model.

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Fine mapping of "mini-muscle," a recessive mutation causing reduced hindlimb muscle mass in mice

http://www.wikidata.org/entity/Q37324197

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 June 2008

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Fine mapping of "mini-muscle," ...... d hindlimb muscle mass in mice

@en

Fine mapping of "mini-muscle," ...... hindlimb muscle mass in mice.

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type

label

Fine mapping of "mini-muscle," ...... d hindlimb muscle mass in mice

@en

Fine mapping of "mini-muscle," ...... hindlimb muscle mass in mice.

@nl

prefLabel

Fine mapping of "mini-muscle," ...... d hindlimb muscle mass in mice

@en

Fine mapping of "mini-muscle," ...... hindlimb muscle mass in mice.

@nl

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Journal of Heredity

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Fine mapping of "mini-muscle," ...... d hindlimb muscle mass in mice

@en

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Daniel Pomp

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Gloria Muñoz

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John Hartmann

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Robert M Hannon

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Scott A Kelly

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Theodore Garland

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P2860

Positional cloning of the mouse obese gene and its human homologue

Sox15 is required for skeletal muscle regeneration

Mutation causing congenital myasthenia reveals acetylcholine receptor beta/delta subunit interaction essential for assembly

Autosomal dominant myopathy: missense mutation (Glu-706 --> Lys) in the myosin heavy chain IIa gene

Regulation of skeletal muscle mass in mice by a new TGF-beta superfamily member

Phenotypic and evolutionary plasticity of organ masses in response to voluntary exercise in house mice

GHRH receptor of little mice contains a missense mutation in the extracellular domain that disrupts receptor function

Lack of Socs2 expression causes the high-growth phenotype in mice

Inheritance and mapping of Compact (Cmpt), a new mutation causing hypermuscularity in mice

Evolution of a small-muscle polymorphism in lines of house mice selected for high activity levels.

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679-687

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scholarly article

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10.1093/JHERED/ESN040

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6

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99

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2008-06-09T00:00:00Z

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5311837

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18544554

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2734099

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