Cardiac muscle protein catabolism in diabetes mellitus: activation of the ubiquitin-proteasome system by insulin deficiency. - Wikidata - awgldk - TriplyDB

Cardiac muscle protein catabolism in diabetes mellitus: activation of the ubiquitin-proteasome system by insulin deficiency.

Cardiac muscle protein catabolism in diabetes mellitus: activation of the ubiquitin-proteasome system by insulin deficiency.

http://www.wikidata.org/entity/Q37325269

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Immunoproteasomes: structure, function, and antigen presentation The ubiquitin-proteasome system in cardiac proteinopathy: a quality control perspective.PTEN inhibition improves muscle regeneration in mice fed a high-fat diet.Evidence for a role of immunoproteasomes in regulating cardiac muscle mass in diabetic mice The regulation of skeletal muscle protein turnover during the progression of cancer cachexia in the Apc(Min/+) mouse.Proteome dynamics and proteome function of cardiac 19S proteasomes Insulin alleviates degradation of skeletal muscle protein by inhibiting the ubiquitin-proteasome system in septic rats.Acupuncture plus Low-Frequency Electrical Stimulation (Acu-LFES) Attenuates Diabetic Myopathy by Enhancing Muscle Regeneration.The role of insulin resistance in the development of muscle wasting during cancer cachexia.Insulin deficiency induces rat renal mesangial cell dysfunction via activation of IGF-1/IGF-1R pathway.Prevention of Streptozotocin-Induced Diabetic Nephropathy by MG132: Possible Roles of Nrf2 and IκB.Combating oxidative stress in diabetic complications with Nrf2 activators: how much is too much?Impaired proteostasis: role in the pathogenesis of diabetes mellitus.Lipopolysaccharide inhibits myogenic differentiation of C2C12 myoblasts through the Toll-like receptor 4-nuclear factor-κB signaling pathway and myoblast-derived tumor necrosis factor-α Diabetic Cardiomyopathy: Current Approach and Potential Diagnostic and Therapeutic Targets.Early-stage chronic kidney disease, insulin resistance, and osteoporosis as risk factors of sarcopenia in aged population: the fourth Korea National Health and Nutrition Examination Survey (KNHANES IV), 2008-2009.Therapeutic effect of MG-132 on diabetic cardiomyopathy is associated with its suppression of proteasomal activities: roles of Nrf2 and NF-κB.Cardiac proteasome functional insufficiency plays a pathogenic role in diabetic cardiomyopathy.Exercise training reduces insulin resistance and upregulates the mTOR/p70S6k pathway in cardiac muscle of diet-induced obesity rats.

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Cardiac muscle protein catabolism in diabetes mellitus: activation of the ubiquitin-proteasome system by insulin deficiency.

http://www.wikidata.org/entity/Q37325269

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 24 July 2008

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Cardiac muscle protein catabol ...... system by insulin deficiency.

@en

Cardiac muscle protein catabol ...... system by insulin deficiency.

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type

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Cardiac muscle protein catabol ...... system by insulin deficiency.

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Cardiac muscle protein catabol ...... system by insulin deficiency.

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prefLabel

Cardiac muscle protein catabol ...... system by insulin deficiency.

@en

Cardiac muscle protein catabol ...... system by insulin deficiency.

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Cardiac muscle protein catabol ...... system by insulin deficiency.

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Janet D Klein

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Jie Du

http://www.w3.org/2001/XMLSchema#string

Junping Hu

http://www.w3.org/2001/XMLSchema#string

Xiaonan H Wang

http://www.w3.org/2001/XMLSchema#string

P2860

Muscle-specific RING finger 1 is a bona fide ubiquitin ligase that degrades cardiac troponin I

Epoxomicin, a potent and selective proteasome inhibitor, exhibits in vivo antiinflammatory activity

The ubiquitin-proteasome proteolytic pathway in heart vs skeletal muscle: effects of acute diabetes.

Muscle protein turnover and glucose uptake in acutely uremic rats. Effects of insulin and the duration of renal insufficiency.

Diabetic cardiomyopathy: recent evidence from mouse models of type 1 and type 2 diabetes.

Insulin signaling coordinately regulates cardiac size, metabolism, and contractile protein isoform expression

The murine cardiac 26S proteasome: an organelle awaiting exploration.

Role of changes in cardiac metabolism in development of diabetic cardiomyopathy.

The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway

Muscle wasting in insulinopenic rats results from activation of the ATP-dependent, ubiquitin-proteasome proteolytic pathway by a mechanism including gene transcription.

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5384-5390

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scholarly article

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10.1210/EN.2008-0132

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11

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2008-07-24T00:00:00Z

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51427125

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18653708

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ubiquitin-proteasome system

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2734490

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