Increase in cellular cyclic AMP concentrations reverses the profibrogenic phenotype of cardiac myofibroblasts: a novel therapeutic approach for cardiac fibrosis.
about
A computational model of cardiac fibroblast signaling predicts context-dependent drivers of myofibroblast differentiation.Targeting Adenosine Receptors for the Treatment of Cardiac FibrosisBerberine regulates proliferation, collagen synthesis and cytokine secretion of cardiac fibroblasts via AMPK-mTOR-p70S6K signaling pathwayCyclic nucleotide signalling in kidney fibrosis.Dibutyryl-cAMP attenuates pulmonary fibrosis by blocking myofibroblast differentiation via PKA/CREB/CBP signaling in rats with silicosis.Non-coding RNAs as direct and indirect modulators of epigenetic mechanism regulation of cardiac fibrosis.Noncoding RNA as regulators of cardiac fibrosis: current insight and the road ahead.Cardiac Fibroblast GRK2 Deletion Enhances Contractility and Remodeling Following Ischemia/Reperfusion Injury.PDE2-mediated cAMP hydrolysis accelerates cardiac fibroblast to myofibroblast conversion and is antagonized by exogenous activation of cGMP signaling pathways.GPR68, a proton-sensing GPCR, mediates interaction of cancer associated fibroblasts and cancer cells.Epac is required for exogenous and endogenous stimulation of adenosine A2B receptor for inhibition of angiotensin II-induced collagen synthesis and myofibroblast differentiation.Critical effects of long non-coding RNA on fibrosis diseases.Adenosine A2a Receptor Blockade Diminishes Wnt/β-Catenin Signaling in a Murine Model of Bleomycin-Induced Dermal Fibrosis.
P2860
Q30277087-24A43ED6-5260-4789-9573-80469C3D14BFQ33639481-00B829A9-2920-478D-8CC0-90D2D8129798Q34675044-BA5BDE3C-04E1-4F11-9C35-991A31F35FDFQ35138860-6DEB0C49-F3B4-4C4C-B56F-1AA2BAEC713CQ37658195-8B9F2D20-D039-4366-9472-50F3D1DE9E6DQ38344492-FB412113-BFFF-454A-8EB9-84E88B1296DEQ38703251-80CAEE0F-D394-4A98-A8F3-2BFCF7BDEADBQ39416228-44E54FD2-5A2A-49FA-89F8-4A94B90F8178Q42454314-7599F299-B9BB-4648-9E46-335209A5E907Q46218367-F9B5FE14-8F8F-4161-A316-3FFBF709FB80Q48265051-F340B6F1-1A91-492C-AD39-105CEF709EE8Q49922983-7FB3613F-CBAA-4B2C-90A3-4689242689D8Q50197688-DCBE3534-53F5-42D8-BCDF-5885E1732576
P2860
Increase in cellular cyclic AMP concentrations reverses the profibrogenic phenotype of cardiac myofibroblasts: a novel therapeutic approach for cardiac fibrosis.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on October 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@en
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@nl
type
label
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@en
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@nl
prefLabel
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@en
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@nl
P2093
P2860
P356
P1476
Increase in cellular cyclic AM ...... approach for cardiac fibrosis.
@en
P2093
Hemal H Patel
Nakon Aroonsakool
Paul A Insel
Utako Yokoyama
P2860
P304
P356
10.1124/MOL.113.087742
P577
2013-10-01T00:00:00Z