GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation. - Wikidata - awgldk - TriplyDB

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

http://www.wikidata.org/entity/Q37325791

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Mechanobiology of mesenchymal stem cells: Which interest for cell-based treatment?Neutrophil mechanotransduction: A GEF to sense fluid shear stress.Ligand-specific binding forces of LFA-1 and Mac-1 in neutrophil adhesion and crawling.Rho GTPase signaling complexes in cell migration and invasion.Go with the flow: GEF-H1 mediated shear stress mechanotransduction in neutrophils.Cell metabolism regulates integrin mechanosensing via an SLC3A2-dependent sphingolipid biosynthesis pathway

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GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

http://www.wikidata.org/entity/Q37325791

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article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on October 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

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name

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

@en

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

@nl

type

label

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

@en

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

@nl

prefLabel

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

@en

GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation.

@nl

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Journal of Cell Biology

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GEF-H1 is necessary for neutrophil shear stress-induced migration during inflammation

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Hisham Ibrahim

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Jacob Rullo

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Jose La Rose

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Paul Kubes

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Cloning and characterization of GEF-H1, a microtubule-associated guanine nucleotide exchange factor for Rac and Rho GTPases

Nucleotide exchange factor GEF-H1 mediates cross-talk between microtubules and the actin cytoskeleton

Analysis of the myosin-II-responsive focal adhesion proteome reveals a role for β-Pix in negative regulation of focal adhesion maturation

Traffic signals for lymphocyte recirculation and leukocyte emigration: the multistep paradigm

Cell migration: Rho GTPases lead the way

Rapid leukocyte migration by integrin-independent flowing and squeezing

Coordinated RhoA signaling at the leading edge and uropod is required for T cell transendothelial migration.

The RhoA activator GEF-H1/Lfc is a transforming growth factor-beta target gene and effector that regulates alpha-smooth muscle actin expression and cell migration

GEF-H1 couples nocodazole-induced microtubule disassembly to cell contractility via RhoA.

Guanine nucleotide exchange factor-H1 regulates cell migration via localized activation of RhoA at the leading edge.

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107-119

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scholarly article

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10.1083/JCB.201603109

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1

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215

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Michael Glogauer

Myron I. Cybulsky

Robert Rottapel

Maria-Jose Sandi

Ioannis D Dimitriou

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2016-10-01T00:00:00Z

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27738004

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