Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
about
Group V phospholipase A(2) increases pulmonary endothelial permeability through direct hydrolysis of the cell membranePlatelets in Pulmonary Immune Responses and Inflammatory Lung DiseasesLeukocyte integrins: role in leukocyte recruitment and as therapeutic targets in inflammatory diseaseOn, around, and through: neutrophil-endothelial interactions in innate immunityMechanisms regulating endothelial permeabilityRho GAPs and GEFs: controling switches in endothelial cell adhesionGlycolytic regulation of cell rearrangement in angiogenesis.The F-BAR protein pacsin2 inhibits asymmetric VE-cadherin internalization from tensile adherens junctions.microRNA-200b as a Switch for Inducible Adult AngiogenesisDissociation of VE-PTP from VE-cadherin is required for leukocyte extravasation and for VEGF-induced vascular permeability in vivoN-octanoyl dopamine inhibits the expression of a subset of κB regulated genes: potential role of p65 Ser276 phosphorylationPhosphorylation of VE-cadherin controls endothelial phenotypes via p120-catenin coupling and Rac1 activation.ARP2/3-mediated junction-associated lamellipodia control VE-cadherin-based cell junction dynamics and maintain monolayer integritySoluble plasma VE-cadherin concentrations are elevated in patients with STEC infection and haemolytic uraemic syndrome: a case-control studyVascular endothelial-cadherin stabilizes at cell-cell junctions by anchoring to circumferential actin bundles through alpha- and beta-catenins in cyclic AMP-Epac-Rap1 signal-activated endothelial cells.Mechanisms of vascular stability and the relationship to human diseaseLeukocyte-endothelial interactions in inflammation.p38 MAP kinase is necessary for melanoma-mediated regulation of VE-cadherin disassemblyPlacenta growth factor-1 exerts time-dependent stabilization of adherens junctions following VEGF-induced vascular permeability.Targeting Robo4-dependent Slit signaling to survive the cytokine storm in sepsis and influenza.Angiopoietin 2 regulates the transformation and integrity of lymphatic endothelial cell junctions.PEDF regulates vascular permeability by a γ-secretase-mediated pathway.Anti-angiogenic effect of high-dose resveratrol in a swine model of metabolic syndrome.Fibroblast growth factor signaling potentiates VE-cadherin stability at adherens junctions by regulating SHP2Rap1 and Rap2 antagonistically control endothelial barrier resistance.PARP-1 regulates metastatic melanoma through modulation of vimentin-induced malignant transformation.Segregation of VE-cadherin from the LBRC depends on the ectodomain sequence required for homophilic adhesion.Actinomyosin contraction, phosphorylation of VE-cadherin, and actin remodeling enable melanoma-induced endothelial cell-cell junction disassembly.Angiopoietin-1 protects heart against ischemia/reperfusion injury through VE-cadherin dephosphorylation and myocardiac integrin-β1/ERK/caspase-9 phosphorylation cascadeProtective role of insulin-like growth factor-1 receptor in endothelial cells against unilateral ureteral obstruction-induced renal fibrosis.p18, a novel adaptor protein, regulates pulmonary endothelial barrier function via enhanced endocytic recycling of VE-cadherin.Cellular Responses Modulated by FGF-2 Adsorbed on Albumin/Heparin Layer-by-Layer AssembliesContractile actin cables induced by Bacillus anthracis lethal toxin depend on the histone acetylation machinery.Prostaglandins in cancer cell adhesion, migration, and invasionVinculin associates with endothelial VE-cadherin junctions to control force-dependent remodeling.A VE-cadherin-PAR3-α-catenin complex regulates the Golgi localization and activity of cytosolic phospholipase A(2)α in endothelial cells.Role of reactive oxygen and nitrogen species in the vascular responses to inflammation.p120-Catenin prevents neutrophil transmigration independently of RhoA inhibition by impairing Src dependent VE-cadherin phosphorylation.Anti-apoptosis effects of vascular endothelial cadherin in experimental corneal neovascularization.The actin-binding protein EPS8 binds VE-cadherin and modulates YAP localization and signaling.
P2860
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P2860
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@en
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@nl
type
label
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@en
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@nl
prefLabel
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@en
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@nl
P2093
P1476
Cell adhesion dynamics at endothelial junctions: VE-cadherin as a major player.
@en
P2093
Astrid F Nottebaum
Giuseppe Cagna
Mark Winderlich
P356
10.1016/J.TCB.2008.10.001
P577
2008-11-17T00:00:00Z