MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice. - Wikidata - awgldk - TriplyDB

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

http://www.wikidata.org/entity/Q37328435

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A potential regulatory role for intronic microRNA-338-3p for its host gene encoding apoptosis-associated tyrosine kinase Micro-RNA-195 and -451 regulate the LKB1/AMPK signaling axis by targeting MO25 Inhibition of microRNA function by antimiR oligonucleotides Identification of candidate genes potentially relevant to chamber-specific remodeling in postnatal ventricular myocardium MicroRNAs in cardiomyocyte development Mechanical stretch up-regulates microRNA-26a and induces human airway smooth muscle hypertrophy by suppressing glycogen synthase kinase-3β Genome-wide association analysis identifies multiple loci related to resting heart rate MicroRNA-218 regulates vascular patterning by modulation of Slit-Robo signaling MicroRNAs: new players in cardiac injury and protection Identifying circulating microRNAs as biomarkers of cardiovascular disease: a systematic review Functions of miRNAs during Mammalian Heart Development Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and Apoptosis MicroRNA and Heart Failure Micromanaging cardiac regeneration: Targeted delivery of microRNAs for cardiac repair and regeneration Diagnostic and prognostic value of circulating microRNAs in heart failure with preserved and reduced ejection fraction Emerging Roles for MicroRNAs in Perioperative Medicine Regulation of connexin signaling by the epigenetic machinery Myostatin: expanding horizons The function of miRNA in cardiac hypertrophy Circulating microRNAs: a potential role in diagnosis and prognosis of acute myocardial infarction Gene regulatory networks in cardiac conduction system development Non-Coding RNAs Including miRNAs and lncRNAs in Cardiovascular Biology and Disease Molecular basis of physiological heart growth: fundamental concepts and new players MicroRNAs in heart failure: Small molecules with major impact Targeting myocardial remodelling to develop novel therapies for heart failure: a position paper from the Working Group on Myocardial Function of the European Society of Cardiology MicroRNAs in stress signaling and human disease miR-1/133a clusters cooperatively specify the cardiomyogenic lineage by adjustment of myocardin levels during embryonic heart development Perturbation of microRNAs in rat heart during chronic doxorubicin treatment Differential expression of microRNAs in different disease states.Non-coding RNAs: the "dark matter" of cardiovascular pathophysiology.Epigenetic mechanisms in heart development and disease Noncoding RNA in age-related cardiovascular diseases The miR-19a/b family positively regulates cardiomyocyte hypertrophy by targeting atrogin-1 and MuRF-1 A cardiac microRNA governs systemic energy homeostasis by regulation of MED13 Embryonic caffeine exposure acts via A1 adenosine receptors to alter adult cardiac function and DNA methylation in mice Loss of MicroRNA-155 protects the heart from pathological cardiac hypertrophy Mechanical stretch via transforming growth factor-β1 activates microRNA208a to regulate endoglin expression in cultured rat cardiac myoblasts Repression of miR-142 by p300 and MAPK is required for survival signalling via gp130 during adaptive hypertrophy The primary microRNA-208b interacts with Polycomb-group protein, Ezh2, to regulate gene expression in the heart mir-17-92 cluster is required for and sufficient to induce cardiomyocyte proliferation in postnatal and adult hearts

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MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

http://www.wikidata.org/entity/Q37328435

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 10 August 2009

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

@en

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

@nl

type

label

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

@en

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

@nl

prefLabel

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

@en

MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice.

@nl

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Journal of Clinical Investigation

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MicroRNA-208a is a regulator of cardiac hypertrophy and conduction in mice

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Bronwyn Gunn

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Craig H Selzman

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Da-Zhi Wang

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Hee Young Seok

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Janelle Shumate

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Jian-Fu Chen

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Kumar Pandya

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Mariko Tatsuguchi

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Ru-Hang Tang

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Thomas E Callis

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P2860

Identity between TRAP and SMCC complexes indicates novel pathways for the function of nuclear receptors and diverse mammalian activators

Myostatin mutation associated with gross muscle hypertrophy in a child

An mRNA m7G cap binding-like motif within human Ago2 represses translation

MicroRNAs control translation initiation by inhibiting eukaryotic initiation factor 4E/cap and poly(A) tail function

MicroRNA targeting specificity in mammals: determinants beyond seed pairing

Myostatin regulates cardiomyocyte growth through modulation of Akt signaling

Targeted deletion reveals essential and overlapping functions of the miR-17 through 92 family of miRNA clusters

Targeted deletion of Dicer in the heart leads to dilated cardiomyopathy and heart failure

miRBase: tools for microRNA genomics

The role of microRNA-1 and microRNA-133 in skeletal muscle proliferation and differentiation

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2772-2786

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scholarly article

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10.1172/JCI36154

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9

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119

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2009-08-10T00:00:00Z

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26786190

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19726871

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regulation of membrane depolarization during AV node cell action potential

positive regulation of membrane depolarization during AV node cell action potential

negative regulation of membrane depolarization during AV node cell action potential

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2735902

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