An efficient and reproducible method for quantifying macrophages in different experimental models of central nervous system pathology.
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Tumor growth increases neuroinflammation, fatigue and depressive-like behavior prior to alterations in muscle function.Characterization of inflammatory gene expression and galectin-3 function after spinal cord injury in mice.Development of NMDAR antagonists with reduced neurotoxic side effects: a study on GK11Evidence for Status Epilepticus and Pro-Inflammatory Changes after Intranasal Kainic Acid Administration in MiceRe-establishment of anxiety in stress-sensitized mice is caused by monocyte trafficking from the spleen to the brain.Age decreases macrophage IL-10 expression: Implications for functional recovery and tissue repair in spinal cord injuryIntranasal insulin treatment of an experimental model of moderate traumatic brain injury.Therapeutic effect of apocynin through antioxidant activity and suppression of apoptosis and inflammation after spinal cord injury.TGFβ produced by IL-10 redirected astrocytes attenuates microglial activationRescuing vasculature with intravenous angiopoietin-1 and alpha v beta 3 integrin peptide is protective after spinal cord injury.CD36 deletion improves recovery from spinal cord injury.Bone marrow mesenchymal stromal cells drive protective M2 microglia polarization after brain traumaSystem x(c)(-) regulates microglia and macrophage glutamate excitotoxicity in vivoSelective expression of Narp in primary nociceptive neurons: role in microglia/macrophage activation following nerve injury.Fractalkine receptor (CX3CR1) deficiency sensitizes mice to the behavioral changes induced by lipopolysaccharideProgranulin expression is upregulated after spinal contusion in mice.Interleukin 1 type 1 receptor restore: a genetic mouse model for studying interleukin 1 receptor-mediated effects in specific cell typesDeficient CX3CR1 signaling promotes recovery after mouse spinal cord injury by limiting the recruitment and activation of Ly6Clo/iNOS+ macrophages.Anti-inflammatory treatments during the chronic phase of spinal cord injury improve locomotor function in adult mice.Delayed inflammatory mRNA and protein expression after spinal cord injury.In vivo magnetization transfer MRI shows dysmyelination in an ischemic mouse model of periventricular leukomalacia.Temporal pattern of expression and colocalization of microglia/macrophage phenotype markers following brain ischemic injury in mice.Toll-Like Receptors and Dectin-1, a C-Type Lectin Receptor, Trigger Divergent Functions in CNS Macrophages.Influenza infection induces neuroinflammation, alters hippocampal neuron morphology, and impairs cognition in adult micePeripheral innate immune challenge exaggerated microglia activation, increased the number of inflammatory CNS macrophages, and prolonged social withdrawal in socially defeated mice.Stress exacerbates neuron loss and microglia proliferation in a rat model of excitotoxic lower motor neuron injuryLong-Term Consequences of Traumatic Brain Injury: Current Status of Potential Mechanisms of Injury and Neurological OutcomesSequential activation of microglia and astrocyte cytokine expression precedes increased Iba-1 or GFAP immunoreactivity following systemic immune challenge.Indoleamine 2,3-dioxygenase inhibition attenuates lipopolysaccharide induced persistent microglial activation and depressive-like complications in fractalkine receptor (CX(3)CR1)-deficient mice.Quantitating the subtleties of microglial morphology with fractal analysis.Cognitive deficits develop 1month after diffuse brain injury and are exaggerated by microglia-associated reactivity to peripheral immune challenge.Neuroinflammatory Dynamics Underlie Memory Impairments after Repeated Social Defeat.Elevated MMP-9 in the lumbar cord early after thoracic spinal cord injury impedes motor relearning in mice.Neonatal E. coli infection causes neuro-behavioral deficits associated with hypomyelination and neuronal sequestration of ironMinocycline Transiently Reduces Microglia/Macrophage Activation but Exacerbates Cognitive Deficits Following Repetitive Traumatic Brain Injury in the Neonatal Rat(18)F-FDG-PET imaging of rat spinal cord demonstrates altered glucose uptake acutely after contusion injury.Molecular and Cellular Responses to Interleukin-4 Treatment in a Rat Model of Transient Ischemia.Predictive screening of M1 and M2 macrophages reveals the immunomodulatory effectiveness of post spinal cord injury azithromycin treatmentKnockdown of interleukin-1 receptor type-1 on endothelial cells attenuated stress-induced neuroinflammation and prevented anxiety-like behavior.Digital image analysis: a review of reproducibility, stability and basic requirements for optimal results.
P2860
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P2860
An efficient and reproducible method for quantifying macrophages in different experimental models of central nervous system pathology.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 23 April 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
An efficient and reproducible ...... tral nervous system pathology.
@en
An efficient and reproducible ...... tral nervous system pathology.
@nl
type
label
An efficient and reproducible ...... tral nervous system pathology.
@en
An efficient and reproducible ...... tral nervous system pathology.
@nl
prefLabel
An efficient and reproducible ...... tral nervous system pathology.
@en
An efficient and reproducible ...... tral nervous system pathology.
@nl
P2093
P2860
P921
P1476
An efficient and reproducible ...... tral nervous system pathology.
@en
P2093
Daniel P Ankeny
Dustin J Donnelly
John C Gensel
Phillip G Popovich
P2860
P356
10.1016/J.JNEUMETH.2009.04.010
P577
2009-04-23T00:00:00Z