Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients. - Wikidata - awgldk - TriplyDB

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

http://www.wikidata.org/entity/Q37335559

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Bipolar disorder and mechanisms of action of mood stabilizers An evo-devo approach to thyroid hormones in cerebral and cerebellar cortical development: etiological implications for autism Brain lipid concentrations in bipolar disorder.Altered expression of apoptotic factors and synaptic markers in postmortem brain from bipolar disorder patients.Allele specific analysis of the ADRBK2 gene in lymphoblastoid cells from bipolar disorder patients.Increased excitotoxicity and neuroinflammatory markers in postmortem frontal cortex from bipolar disorder patients.Disturbed neurotransmitter transporter expression in Alzheimer's disease brain Altered arachidonic acid cascade enzymes in postmortem brain from bipolar disorder patients Suppression of G-protein-coupled receptor kinase 3 expression is a feature of classical GBM that is required for maximal growth.Imaging brain signal transduction and metabolism via arachidonic and docosahexaenoic acid in animals and humans.Overexpression of diacylglycerol kinase η enhances Gαq-coupled G protein-coupled receptor signaling.Genes and gene networks implicated in aggression related behaviour.

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Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

http://www.wikidata.org/entity/Q37335559

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 29 April 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@en

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@nl

type

label

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@en

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@nl

prefLabel

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@en

Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@nl

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S146114570900025X

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The International Journal of Neuropsychopharmacology

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Decreased GRK3 but not GRK2 expression in frontal cortex from bipolar disorder patients.

@en

P2093

Hyung-Wook Kim

http://www.w3.org/2001/XMLSchema#string

Jagadeesh S Rao

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P2860

A genome-wide association study implicates diacylglycerol kinase eta (DGKH) and several other genes in the etiology of bipolar disorder

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Evidence that a single nucleotide polymorphism in the promoter of the G protein receptor kinase 3 gene is associated with bipolar disorder

Histamine H2 receptor desensitization: involvement of a select array of G protein-coupled receptor kinases

Decrease in the AP-2 DNA-binding activity and in the protein expression of AP-2 alpha and AP-2 beta in frontal cortex of rats treated with lithium for 6 weeks

The G-protein-coupled receptor kinases beta ARK1 and beta ARK2 are widely distributed at synapses in rat brain

Chronic treatment with mood stabilizers increases membrane GRK3 in rat frontal cortex

The prevalence and disability of bipolar spectrum disorders in the US population: re-analysis of the ECA database taking into account subthreshold cases.

Neuropathology of bipolar disorder.

Regulation of G protein-coupled receptor kinases.

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851-860

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scholarly article

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10.1017/S146114570900025X

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6

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12

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Stanley I. Rapoport

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2009-04-29T00:00:00Z

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19400979

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bipolar disorder

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2738976

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