Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription. - Wikidata - awgldk - TriplyDB

Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription.

Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription.

http://www.wikidata.org/entity/Q37340291

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Brd4 and JMJD6-associated anti-pause enhancers in regulation of transcriptional pause release Regulation of MYC gene expression by aberrant Wnt/β-catenin signaling in colorectal cancer Architectural and Functional Commonalities between Enhancers and Promoters Noncoding RNAs, Emerging Regulators of Skeletal Muscle Development and Diseases Nuclear Receptors, RXR, and the Big Bang Involvement of the circadian rhythm and inflammatory cytokines in the pathogenesis of rheumatoid arthritis Exploiting genomics and natural genetic variation to decode macrophage enhancers Preparing the first responders: building the inflammatory transcriptome from the ground up The Mediator complex: a central integrator of transcription Enhancer RNAs and regulated transcriptional programs Towards an understanding of cell-specific functions of signal-dependent transcription factors Emerging epigenetic mechanisms of long non-coding RNAs Mechanisms Underlying the Selection and Function of Macrophage-Specific Enhancers Transcriptional Activation of Inflammatory Genes: Mechanistic Insight into Selectivity and Diversity The circadian clock regulates autophagy directly through the nuclear hormone receptor Nr1d1/Rev-erbα and indirectly via Cebpb/(C/ebpβ) in zebrafish.Long noncoding RNAs: pivotal regulators in acute myeloid leukemia Clock Genes in Glia Cells: A Rhythmic History The selection and function of cell type-specific enhancers Genetic and epigenetic fine mapping of causal autoimmune disease variants Mammalian-wide interspersed repeat (MIR)-derived enhancers and the regulation of human gene expression Super-enhancers in the control of cell identity and disease The M1 and M2 paradigm of macrophage activation: time for reassessment Circadian Influence on Metabolism and Inflammation in Atherosclerosis.Computational Approaches for Mining GRO-Seq Data to Identify and Characterize Active Enhancers.ALTRE: workflow for defining ALTered Regulatory Elements using chromatin accessibility data.Human colorectal cancer-specific CCAT1-L lncRNA regulates long-range chromatin interactions at the MYC locus.Activation of P-TEFb by Androgen Receptor-Regulated Enhancer RNAs in Castration-Resistant Prostate Cancer.Anti-diabetic rosiglitazone remodels the adipocyte transcriptome by redistributing transcription to PPARγ-driven enhancers.Enhancer RNAs participate in androgen receptor-driven looping that selectively enhances gene activation lncRNA-dependent mechanisms of androgen-receptor-regulated gene activation programs.Long non-coding RNAs and enhancer RNAs regulate the lipopolysaccharide-induced inflammatory response in human monocytes.Noncoding RNAs in vascular disease.The active enhancer network operated by liganded RXR supports angiogenic activity in macrophages.An Intrinsic Transcriptional Program Underlying Synaptic Scaling during Activity Suppression Long noncoding RNAs usher in a new era in the biology of enhancers.Transcriptional regulatory functions of nuclear long noncoding RNAs.REV-ERB ALPHA polymorphism is associated with obesity in the Spanish obese male population Enhancers as non-coding RNA transcription units: recent insights and future perspectives.Long noncoding RNA MRUL promotes ABCB1 expression in multidrug-resistant gastric cancer cell sublines Tissue-specific RNA expression marks distant-acting developmental enhancers.

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Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription.

http://www.wikidata.org/entity/Q37340291

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 02 June 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Rev-Erbs repress macrophage ge ...... hancer-directed transcription.

@en

Rev-Erbs repress macrophage ge ...... hancer-directed transcription.

@nl

type

label

Rev-Erbs repress macrophage ge ...... hancer-directed transcription.

@en

Rev-Erbs repress macrophage ge ...... hancer-directed transcription.

@nl

prefLabel

Rev-Erbs repress macrophage ge ...... hancer-directed transcription.

@en

Rev-Erbs repress macrophage ge ...... hancer-directed transcription.

@nl

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Rev-Erbs repress macrophage gene expression by inhibiting enhancer-directed transcription

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Andy Watt

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Aneeza S Kim

http://www.w3.org/2001/XMLSchema#string

Cindy Y Lee

http://www.w3.org/2001/XMLSchema#string

David Gosselin

http://www.w3.org/2001/XMLSchema#string

Han Cho

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Hanna P Lesch

http://www.w3.org/2001/XMLSchema#string

Michael G Rosenfeld

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Michael T Y Lam

http://www.w3.org/2001/XMLSchema#string

Mika Kosaka

http://www.w3.org/2001/XMLSchema#string

Tamar R Grossman

http://www.w3.org/2001/XMLSchema#string

P2860

Redundant function of REV-ERBalpha and beta and non-essential role for Bmal1 cycling in transcriptional regulation of intracellular circadian rhythms

REV-ERBalpha participates in circadian SREBP signaling and bile acid homeostasis

The orphan nuclear receptor Rev-erbalpha recruits the N-CoR/histone deacetylase 3 corepressor to regulate the circadian Bmal1 gene

Isoform-specific amino-terminal domains dictate DNA-binding properties of ROR alpha, a novel family of orphan hormone nuclear receptors

Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities

edgeR: a Bioconductor package for differential expression analysis of digital gene expression data

The orphan nuclear receptor REV-ERBalpha controls circadian transcription within the positive limb of the mammalian circadian oscillator

Histone modifications at human enhancers reflect global cell-type-specific gene expression

Nascent RNA sequencing reveals widespread pausing and divergent initiation at human promoters

Tie2Cre-mediated gene ablation defines the stem-cell leukemia gene (SCL/tal1)-dependent window during hematopoietic stem-cell development

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511-515

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scholarly article

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10.1038/NATURE12209

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7455

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498

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Minna U Kaikkonen

Christopher K Glass

Ronald M. Evans

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2013-06-02T00:00:00Z

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237004429

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1023040655

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23728303

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