Generation and characterization of a conditional deletion allele for Lmna in mice. - Wikidata - awgldk - TriplyDB

Generation and characterization of a conditional deletion allele for Lmna in mice.

Generation and characterization of a conditional deletion allele for Lmna in mice.

http://www.wikidata.org/entity/Q37342999

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Building up the nucleus: nuclear organization in the establishment of totipotency and pluripotency during mammalian development Recent advances in animal and human pluripotent stem cell modeling of cardiac laminopathy Lamins position the nuclear pores and centrosomes by modulating dynein.Nuclear lamina remodelling and its implications for human disease.Lamins at the crossroads of mechanosignaling VRK2A is an A-type lamin-dependent nuclear envelope kinase that phosphorylates BAF.Macrophage Lamin A/C Regulates Inflammation and the Development of Obesity-Induced Insulin Resistance.The Pathogenesis and Therapies of Striated Muscle Laminopathies

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Generation and characterization of a conditional deletion allele for Lmna in mice.

http://www.wikidata.org/entity/Q37342999

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 30 August 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Generation and characterization of a conditional deletion allele for Lmna in mice.

@en

Generation and characterization of a conditional deletion allele for Lmna in mice.

@nl

type

label

Generation and characterization of a conditional deletion allele for Lmna in mice.

@en

Generation and characterization of a conditional deletion allele for Lmna in mice.

@nl

prefLabel

Generation and characterization of a conditional deletion allele for Lmna in mice.

@en

Generation and characterization of a conditional deletion allele for Lmna in mice.

@nl

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J.BBRC.2013.08.082

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Biochemical and Biophysical Research Communications

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Generation and characterization of a conditional deletion allele for Lmna in mice.

@en

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Yixian Zheng

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Youngjo Kim

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P2860

Mutations in the gene encoding lamin A/C cause autosomal dominant Emery-Dreifuss muscular dystrophy

Homologies in both primary and secondary structure between nuclear envelope and intermediate filament proteins

Loss of A-type lamin expression compromises nuclear envelope integrity leading to muscular dystrophy

When lamins go bad: nuclear structure and disease

Human laminopathies: nuclei gone genetically awry

Domain organization of human chromosomes revealed by mapping of nuclear lamina interactions

Nuclear lamin LI of Xenopus laevis: cDNA cloning, amino acid sequence and binding specificity of a member of the lamin B subfamily

Lamin A/C haploinsufficiency causes dilated cardiomyopathy and apoptosis-triggered cardiac conduction system disease.

Nuclear lamins: major factors in the structural organization and function of the nucleus and chromatin

Post-natal myogenic and adipogenic developmental: defects and metabolic impairment upon loss of A-type lamins.

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8-13

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scholarly article

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10.1016/J.BBRC.2013.08.082

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2013-08-30T00:00:00Z

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