Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
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The physiological and clinical importance of sodium potassium ATPase in cardiovascular diseasesTarget organ cross talk in cardiorenal syndrome: animal modelsThe Trade-Off between Dietary Salt and Cardiovascular Disease; A Role for Na/K-ATPase Signaling?The Role of Na/K-ATPase Signaling in Oxidative Stress Related to Obesity and Cardiovascular DiseaseNa+/Ca2+ exchange and Na+/K+-ATPase in the heartAngiotensin II overcomes strain-dependent resistance of rapid CKD progression in a new remnant kidney mouse modelpNaKtide Attenuates Steatohepatitis and Atherosclerosis by Blocking Na/K-ATPase/ROS Amplification in C57Bl6 and ApoE Knockout Mice Fed a Western Diet.Reduction of Na/K-ATPase affects cardiac remodeling and increases c-kit cell abundance in partial nephrectomized mice.Knee subchondral bone perfusion and its relationship to marrow fat and trabeculation on multi-parametric MRI and micro-CT in experimental CKD.Effects of Na/K-ATPase and its ligands on bone marrow stromal cell differentiationEndogenous cardiotonic steroids and salt-sensitive hypertension.Renal ischemia regulates marinobufagenin release in humans.Angiotensin-(1-7) infusion is associated with increased blood pressure and adverse cardiac remodelling in rats with subtotal nephrectomy.Mice expressing ouabain-sensitive α1-Na,K-ATPase have increased susceptibility to pressure overload-induced cardiac hypertrophyEndogenous cardiotonic steroids: physiology, pharmacology, and novel therapeutic targets.Excisional wound healing is delayed in a murine model of chronic kidney disease.Soluble Klotho Protects against Uremic Cardiomyopathy Independently of Fibroblast Growth Factor 23 and Phosphate.Reactive Oxygen Species Modulation of Na/K-ATPase Regulates Fibrosis and Renal Proximal Tubular Sodium HandlingReduction of Na/K-ATPase potentiates marinobufagenin-induced cardiac dysfunction and myocyte apoptosis.Expression of rat Na-K-ATPase α2 enables ion pumping but not ouabain-induced signaling in α1-deficient porcine renal epithelial cellsDiminazene Aceturate Improves Cardiac Fibrosis and Diastolic Dysfunction in Rats with Kidney Disease.High salt intake causes adverse fetal programming--vascular effects beyond blood pressureAdverse cardiac effects of exogenous angiotensin 1-7 in rats with subtotal nephrectomy are prevented by ACE inhibition.Elevated Plasma Marinobufagenin, An Endogenous Cardiotonic Steroid, Is Associated With Right Ventricular Dysfunction and Nitrative Stress in Heart Failure.Molecular mechanisms of experimental salt-sensitive hypertension.Myocardial dysfunction occurs prior to changes in ventricular geometry in mice with chronic kidney disease (CKD)Endogenous digitalis: pathophysiologic roles and therapeutic applications.Decreased renal AT1 receptor binding in rats after subtotal nephrectomy: PET study with [(18)F]FPyKYNE-losartanMarinobufagenin induces increases in procollagen expression in a process involving protein kinase C and Fli-1: implications for uremic cardiomyopathy.Role of endogenous cardiotonic steroids in sodium homeostasis.Marinobufagenin enhances cardiac contractility in mice with ouabain-sensitive alpha1 Na+-K+-ATPase.Attenuation of Na/K-ATPase Mediated Oxidant Amplification with pNaKtide Ameliorates Experimental Uremic Cardiomyopathy.Rapamycin Attenuates Cardiac Fibrosis in Experimental Uremic Cardiomyopathy by Reducing Marinobufagenin Levels and Inhibiting Downstream Pro-Fibrotic Signaling.Spironolactone attenuates experimental uremic cardiomyopathy by antagonizing marinobufageninExperimental models of renal disease and the cardiovascular system.Cigarette smoking causes epigenetic changes associated with cardiorenal fibrosis.Uremic cardiac hypertrophy is reversed by rapamycin but not by lowering of blood pressure.Reducing Cardiac Fibrosis: Na/K-ATPase Signaling Complex as a Novel Targetp53 negatively regulates the osteogenic differentiation of vascular smooth muscle cells in mice with chronic kidney diseaseCardiac structure and function in a mouse model of uraemia without hypertension.
P2860
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P2860
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@en
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@nl
type
label
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@en
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@nl
prefLabel
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@en
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@nl
P2093
P2860
P1476
Partial nephrectomy as a model for uremic cardiomyopathy in the mouse.
@en
P2093
Alexei Y Bagrov
Amjad Shidyak
Anna P Shapiro
Christopher J Cooper
David J Kennedy
Deepak Malhotra
Eric Morgan
Jiang Tian
Jihad Elkareh
Joseph I Shapiro
P2860
P304
P356
10.1152/AJPRENAL.00472.2007
P577
2007-11-21T00:00:00Z