Ah receptor represses acute-phase response gene expression without binding to its cognate response element. - Wikidata - awgldk - TriplyDB

Ah receptor represses acute-phase response gene expression without binding to its cognate response element.

Ah receptor represses acute-phase response gene expression without binding to its cognate response element.

http://www.wikidata.org/entity/Q37345054

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The aryl hydrocarbon receptor: a molecular pathway for the environmental control of the immune response Autoimmunity and infection in Sjögren's syndrome PCB126 inhibits adipogenesis of human preadipocytes Exactly the same but different: promiscuity and diversity in the molecular mechanisms of action of the aryl hydrocarbon (dioxin) receptor Differential gene regulation by the human and mouse aryl hydrocarbon receptor.Antagonism of aryl hydrocarbon receptor signaling by 6,2',4'-trimethoxyflavone The uremic toxin 3-indoxyl sulfate is a potent endogenous agonist for the human aryl hydrocarbon receptor.Evidence for ligand-mediated selective modulation of aryl hydrocarbon receptor activity.The aryl hydrocarbon receptor: regulation of hematopoiesis and involvement in the progression of blood diseases.Kynurenic acid is a potent endogenous aryl hydrocarbon receptor ligand that synergistically induces interleukin-6 in the presence of inflammatory signaling.Development of a selective modulator of aryl hydrocarbon (Ah) receptor activity that exhibits anti-inflammatory properties.Protein function analysis: rapid, cell-based siRNA-mediated ablation of endogenous expression with simultaneous ectopic replacement.Regulation of tissue factor gene expression in monocytes and endothelial cells: Thromboxane A2 as a new player The role of caveolae in endothelial cell dysfunction with a focus on nutrition and environmental toxicants An integrated genomic analysis of aryl hydrocarbon receptor-mediated inhibition of B-cell differentiation.Ah receptor antagonism inhibits constitutive and cytokine inducible IL6 production in head and neck tumor cell lines.Xenobiotic metabolism, disposition, and regulation by receptors: from biochemical phenomenon to predictors of major toxicities Suppression of cytokine-mediated complement factor gene expression through selective activation of the Ah receptor with 3',4'-dimethoxy-α-naphthoflavone Aryl hydrocarbon receptor-dependent retention of nuclear HuR suppresses cigarette smoke-induced cyclooxygenase-2 expression independent of DNA-binding.Identification of a high-affinity ligand that exhibits complete aryl hydrocarbon receptor antagonism.Indoxyl sulfate downregulates expression of Mas receptor via OAT3/AhR/Stat3 pathway in proximal tubular cells Genetic and pharmacological analysis identifies a physiological role for the AHR in epidermal differentiation.Potential health-modulating effects of isoflavones and metabolites via activation of PPAR and AhR.Differential regulation of Th17 and T regulatory cell differentiation by aryl hydrocarbon receptor dependent xenobiotic response element dependent and independent pathways Aryl hydrocarbon receptor regulates the cholesterol biosynthetic pathway in a dioxin response element-independent manner Indole and Tryptophan Metabolism: Endogenous and Dietary Routes to Ah Receptor Activation.Selective aryl hydrocarbon receptor modulator-mediated repression of CD55 expression induced by cytokine exposure Role of the Ah receptor in homeostatic control of fatty acid synthesis in the liver Aryl hydrocarbon receptor-dependence of dioxin's effects on constitutive mouse hepatic cytochromes P450 and growth hormone signaling components Comparative analysis of temporal and dose-dependent TCDD-elicited gene expression in human, mouse, and rat primary hepatocytes Inhibition of the aryl hydrocarbon receptor prevents Western diet-induced obesity. Model for AHR activation by kynurenine via oxidized-LDL, TLR2/4, TGFβ, and IDO1 A distal enhancer controls cytokine-dependent human cPLA2α gene expression Aryl hydrocarbon receptor signaling regulates NF-κB RelB activation during dendritic-cell differentiation Lack of ligand-selective binding of the aryl hydrocarbon receptor to putative DNA binding sites regulating expression of Bax and paraoxonase 1 genes.Identification of cinnabarinic acid as a novel endogenous aryl hydrocarbon receptor ligand that drives IL-22 production.Nuclear receptors in the cross-talk of drug metabolism and inflammation.Structural modeling of the AhR:ARNT complex in the bHLH-PASA-PASB region elucidates the key determinants of dimerization.Control of immune-mediated pathology via the aryl hydrocarbon receptor.Mechanistic insights into the events that lead to synergistic induction of interleukin 6 transcription upon activation of the aryl hydrocarbon receptor and inflammatory signaling.Atranorin and lecanoric acid antagonize TCDD-induced xenobiotic response element-driven activity, but not xenobiotic response element-independent activity.

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Ah receptor represses acute-phase response gene expression without binding to its cognate response element.

http://www.wikidata.org/entity/Q37345054

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 30 March 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Ah receptor represses acute-ph ...... its cognate response element.

@en

Ah receptor represses acute-ph ...... its cognate response element.

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type

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Ah receptor represses acute-ph ...... its cognate response element.

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Ah receptor represses acute-ph ...... its cognate response element.

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Ah receptor represses acute-ph ...... its cognate response element.

@en

Ah receptor represses acute-ph ...... its cognate response element.

@nl

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LABINVEST.2009.24

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Laboratory Investigation

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Ah receptor represses acute-ph ...... its cognate response element.

@en

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Ann Kusnadi

http://www.w3.org/2001/XMLSchema#string

Colin A Flaveny

http://www.w3.org/2001/XMLSchema#string

Gary H Perdew

http://www.w3.org/2001/XMLSchema#string

Iain A Murray

http://www.w3.org/2001/XMLSchema#string

Rushang D Patel

http://www.w3.org/2001/XMLSchema#string

P2860

NF-kappa B and C/EBP transcription factor families synergistically function in mouse serum amyloid A gene expression induced by inflammatory cytokines

Aryl hydrocarbon receptor-dependent liver development and hepatotoxicity are mediated by different cell types.

The hsp90 Co-chaperone XAP2 alters importin beta recognition of the bipartite nuclear localization signal of the Ah receptor and represses transcriptional activity

Nuclear receptors versus inflammation: mechanisms of transrepression

Cutting edge: TLR2 is a functional receptor for acute-phase serum amyloid A

Serum amyloid A: an acute-phase protein involved in tumour pathogenesis

Orientation of the heterodimeric aryl hydrocarbon (dioxin) receptor complex on its asymmetric DNA recognition sequence

Nonexpression of the human serum amyloid A three (SAA3) gene

Cytokines and the hepatic acute-phase response.

Expression of hepatitis B virus X protein does not alter the accumulation of spontaneous mutations in transgenic mice

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labinvest.2009.24

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695-707

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scholarly article

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10.1038/LABINVEST.2009.24

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6

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89

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1018990543

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19333233

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2743259

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