Opposite regulation of KCNQ5 and TRPC6 channels contributes to vasopressin-stimulated calcium spiking responses in A7r5 vascular smooth muscle cells.
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Exercise training decreases store-operated Ca2+entry associated with metabolic syndrome and coronary atherosclerosis.Diclofenac distinguishes among homomeric and heteromeric potassium channels composed of KCNQ4 and KCNQ5 subunitsLarge conductance Ca²⁺-activated K⁺ (BK) channels promote secretagogue-induced transition from spiking to bursting in murine anterior pituitary corticotrophs.Activation of vascular KCNQ (Kv7) potassium channels reverses spasmogen-induced constrictor responses in rat basilar artery.Transient receptor potential channels in the vasculature.Kv7.5 Potassium Channel Subunits Are the Primary Targets for PKA-Dependent Enhancement of Vascular Smooth Muscle Kv7 Currents.Activation of hERG3 channel stimulates autophagy and promotes cellular senescence in melanoma.Differential effects of selective cyclooxygenase-2 inhibitors on vascular smooth muscle ion channels may account for differences in cardiovascular risk profiles.Differential protein kinase C-dependent modulation of Kv7.4 and Kv7.5 subunits of vascular Kv7 channels.Protein kinase C-dependent phosphorylation of transient receptor potential canonical 6 (TRPC6) on serine 448 causes channel inhibition.Involvement of phosphoinositide 3-kinase and PTEN protein in mechanism of activation of TRPC6 protein in vascular smooth muscle cells.The serine 814 of TRPC6 is phosphorylated under unstimulated conditions.Effects of Schisandra chinensis extract on the contractility of corpus cavernosal smooth muscle (CSM) and Ca2+ homeostasis in CSM cells.Perivascular adipose tissue and the dynamic regulation of Kv 7 and Kir channels: Implications for resistant hypertension.Modeling the diversity of spontaneous and agonist-induced electrical activity in anterior pituitary corticotrophs.Kv7 potassium channels as signal transduction intermediates in the control of microvascular tone.
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P2860
Opposite regulation of KCNQ5 and TRPC6 channels contributes to vasopressin-stimulated calcium spiking responses in A7r5 vascular smooth muscle cells.
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article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 25 February 2009
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vedecký článok
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vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
@en
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
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type
label
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
@en
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
@nl
prefLabel
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
@en
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
@nl
P2093
P2860
P1433
P1476
Opposite regulation of KCNQ5 a ...... vascular smooth muscle cells.
@en
P2093
Bharath K Mani
Kenneth L Byron
Leanne L Cribbs
Lioubov I Brueggemann
P2860
P304
P356
10.1016/J.CECA.2009.01.004
P577
2009-02-25T00:00:00Z