In vitro susceptibility to rhinovirus infection is greater for bronchial than for nasal airway epithelial cells in human subjects - Wikidata - awgldk - TriplyDB

In vitro susceptibility to rhinovirus infection is greater for bronchial than for nasal airway epithelial cells in human subjects

In vitro susceptibility to rhinovirus infection is greater for bronchial than for nasal airway epithelial cells in human subjects

http://www.wikidata.org/entity/Q37348004

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CX3CR1 Is Expressed in Differentiated Human Ciliated Airway Cells and Co-Localizes with Respiratory Syncytial Virus on Cilia in a G Protein-Dependent Manner Recent advances in understanding and managing asthma Role of infection in the development and exacerbation of asthma Virus/Allergen Interaction in Asthma Exacerbation Validation of normal human bronchial epithelial cells as a model for influenza A infections in human distal trachea Comparison of viral load in individuals with and without asthma during infections with rhinovirus Nerve growth factor modulates human rhinovirus infection in airway epithelial cells by controlling ICAM-1 expression T lymphocytes promote the antiviral and inflammatory responses of airway epithelial cells.In vitro modeling of respiratory syncytial virus infection of pediatric bronchial epithelium, the primary target of infection in vivo Synthetic response of stimulated respiratory epithelium: modulation by prednisolone and iKK2 inhibition.Epidemiology of virus-induced asthma exacerbations: with special reference to the role of human rhinovirus.Phenotypic and genetic aspects of epithelial barrier function in asthmatic patients.Viruses and asthma Human rhinovirus infection causes different DNA methylation changes in nasal epithelial cells from healthy and asthmatic subjects.Rhinovirus-induced modulation of gene expression in bronchial epithelial cells from subjects with asthma.The ABCs of rhinoviruses, wheezing, and asthma Increased cytokine response of rhinovirus-infected airway epithelial cells in chronic obstructive pulmonary disease.Asthma is associated with multiple alterations in anti-viral innate signalling pathways Viruses exacerbating chronic pulmonary disease: the role of immune modulation.Viral and host factors determine innate immune responses in airway epithelial cells from children with wheeze and atopy.Role of viral respiratory infections in asthma and asthma exacerbations Volatile emanations from in vitro airway cells infected with human rhinovirus.Cigarette smoke modulates expression of human rhinovirus-induced airway epithelial host defense genes Virus/allergen interactions and exacerbations of asthma.Nasal cytokine responses to natural colds in asthmatic children The effect of inhaled IFN-β on worsening of asthma symptoms caused by viral infections. A randomized trial.The airway epithelium: soldier in the fight against respiratory viruses Adaptive immunity to rhinoviruses: sex and age matter.Interferon response and respiratory virus control are preserved in bronchial epithelial cells in asthma.D prostanoid receptor 2 (chemoattractant receptor-homologous molecule expressed on TH2 cells) protein expression in asthmatic patients and its effects on bronchial epithelial cells Rhinoviruses, allergic inflammation, and asthma Rhinovirus and asthma: a storied history of incompatibility.Phenotypic responses of differentiated asthmatic human airway epithelial cultures to rhinovirus.Human parainfluenza virus type 3 (HPIV3) induces production of IFNγ and RANTES in human nasal epithelial cells (HNECs).Host epithelial-viral interactions as cause and cure for asthma.Reduced IRF7 response to rhinovirus unrelated with DNA methylation in peripheral mononuclear cells of adult asthmatics.Interferon at the crossroads of allergy and viral infections.Airway epithelial cells activate TH2 cytokine production in mast cells through IL-1 and thymic stromal lymphopoietin.Asthma as a chronic disease of the innate and adaptive immune systems responding to viruses and allergens Rhinovirus-associated wheeze during infancy and asthma development

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In vitro susceptibility to rhinovirus infection is greater for bronchial than for nasal airway epithelial cells in human subjects

http://www.wikidata.org/entity/Q37348004

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 May 2009

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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name

In vitro susceptibility to rhi ...... helial cells in human subjects

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In vitro susceptibility to rhi ...... elial cells in human subjects.

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In vitro susceptibility to rhi ...... helial cells in human subjects

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In vitro susceptibility to rhi ...... elial cells in human subjects.

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In vitro susceptibility to rhi ...... helial cells in human subjects

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In vitro susceptibility to rhi ...... elial cells in human subjects.

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J.JACI.2009.03.010

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The Journal of Allergy and Clinical Immunology

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In vitro susceptibility to rhi ...... helial cells in human subjects

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David Schnurr

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Gregory M Dolganov

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Hofer Wong

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Homer A Boushey

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Jonathan H Widdicombe

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Nilceia Lopez-Souza

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Pedro C Avila

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Shigeo Yagi

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Silvio Favoreto

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Theresa Ward

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P2860

unr, a cellular cytoplasmic RNA-binding protein with five cold-shock domains, is required for internal initiation of translation of human rhinovirus RNA

Localization of human rhinovirus replication in the upper respiratory tract by in situ hybridization.

Respiratory virus infection of monolayer cultures of human nasal epithelial cells.

Rhinoviruses as pathogens of the lower respiratory tract.

Rhinovirus induces airway epithelial gene expression through double-stranded RNA and IFN-dependent pathways

Effect of respiratory virus infections including rhinovirus on clinical status in cystic fibrosis

Respiratory viruses and exacerbations of asthma in adults.

Host defense function of the airway epithelium in health and disease: clinical background.

Asthmatic bronchial epithelial cells have a deficient innate immune response to infection with rhinovirus.

Community study of role of viral infections in exacerbations of asthma in 9-11 year old children.

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1384-90.e2

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scholarly article

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10.1016/J.JACI.2009.03.010

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6

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2009-05-09T00:00:00Z

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24413767

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19428098

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human subject research

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2744461

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