The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer. - Wikidata - awgldk - TriplyDB

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

http://www.wikidata.org/entity/Q37350245

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The PI3K/Akt Pathway in Tumors of Endocrine Tissues Mass spectrometry based cellular phosphoinositides profiling and phospholipid analysis: a brief review Selective repression of RET proto-oncogene in medullary thyroid carcinoma by a natural alkaloid berberine.Signaling pathways as specific pharmacologic targets for neuroendocrine tumor therapy: RET, PI3K, MEK, growth factors, and Notch.Vandetanib for the treatment of metastatic medullary thyroid cancer MK-2206 causes growth suppression and reduces neuroendocrine tumor marker production in medullary thyroid cancer through Akt inhibition.Demonstration of a potent RET transcriptional inhibitor for the treatment of medullary thyroid carcinoma based on an ellipticine derivative.On-line surface plasmon resonance biosensing of vascular endothelial growth factor signaling in intact-human hepatoma cell lines.The Phosphatidylinositol 3-kinase/Akt Signaling Pathway in Neuroendocrine Tumors.Cellular signaling pathway alterations and potential targeted therapies for medullary thyroid carcinoma.

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The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

http://www.wikidata.org/entity/Q37350245

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 September 2008

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

@en

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

@nl

type

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The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

@en

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

@nl

prefLabel

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

@en

The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

@nl

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J.SURG.2008.06.028

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The phosphatidylinositol 3-kinase/akt signaling pathway in medullary thyroid cancer.

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Herbert Chen

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Susan C Pitt

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P2860

The role of human achaete-scute homolog-1 in medullary thyroid cancer cells

Targeting the PI3K-Akt pathway in human cancer: rationale and promise.

Inactivation of glycogen synthase kinase-3beta, a downstream target of the raf-1 pathway, is associated with growth suppression in medullary thyroid cancer cells.

Apoptosis-mediated medullary thyroid cancer growth suppression by the PI3K inhibitor LY294002.

Overexpression of the NOTCH1 intracellular domain inhibits cell proliferation and alters the neuroendocrine phenotype of medullary thyroid cancer cells.

In-vivo activation of Raf-1 inhibits tumor growth and development in a xenograft model of human medullary thyroid cancer.

Role of MEN2A-derived RET in maintenance and proliferation of medullary thyroid carcinoma.

Antiproliferative effects of Src inhibition on medullary thyroid cancer.

Increased in vivo phosphorylation of ret tyrosine 1062 is a potential pathogenetic mechanism of multiple endocrine neoplasia type 2B.

Transforming ability of MEN2A-RET requires activation of the phosphatidylinositol 3-kinase/AKT signaling pathway.

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scholarly article

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