Mouse Paneth cell antimicrobial function is independent of Nod2. - Wikidata - awgldk - TriplyDB

Mouse Paneth cell antimicrobial function is independent of Nod2.

Mouse Paneth cell antimicrobial function is independent of Nod2.

http://www.wikidata.org/entity/Q37351171

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Do antimicrobial peptides and complement collaborate in the intestinal mucosa?Ontogeny of intestinal epithelial innate immune responses Nod2: A Critical Regulator of Ileal Microbiota and Crohn's Disease Roles for Intestinal Bacteria, Viruses, and Fungi in Pathogenesis of Inflammatory Bowel Diseases and Therapeutic Approaches.Host defense peptide resistance contributes to colonization and maximal intestinal pathology by Crohn's disease-associated adherent-invasive Escherichia coli.Nucleotide-binding oligomerization domain-containing protein 2 controls host response to Campylobacter jejuni in Il10-/- mice.Cancer-promoting effects of microbial dysbiosis.The microbiota protects against ischemia/reperfusion-induced intestinal injury through nucleotide-binding oligomerization domain-containing protein 2 (NOD2) signaling.Bacterial sensor Nod2 prevents inflammation of the small intestine by restricting the expansion of the commensal Bacteroides vulgatus.The genetic predisposition and the interplay of host genetics and gut microbiome in Crohn disease NOD1 and NOD2: signaling, host defense, and inflammatory disease.Antimicrobial peptides and gut microbiota in homeostasis and pathology.The microbiome and cancer.A small intestinal organoid model of non-invasive enteric pathogen-epithelial cell interactions.Critical design aspects involved in the study of Paneth cells and the intestinal microbiota.Cellular and molecular mechanisms underlying NOD2 risk-associated polymorphisms in Crohn's disease.No vacancy: how beneficial microbes cooperate with immunity to provide colonization resistance to pathogens Pathogenesis of Crohn's disease Respective Roles of Hematopoietic and Nonhematopoietic Nod2 on the Gut Microbiota and Mucosal Homeostasis.Acute Infectious Gastroenteritis Potentiates a Crohn's Disease Pathobiont to Fuel Ongoing Inflammation in the Post-Infectious Period.Nod2: The intestinal gate keeper.Paneth cell defects in Crohn's disease patients promote dysbiosis.Microbiota Composition and Immune Responses During Campylobacter Jejuni Infection in Conventionally Colonized IL-10-/- Mice Lacking Nucleotide Oligomerization Domain 2.Debug Your Bugs - How NLRs Shape Intestinal Host-Microbe Interactions.Intestinal barrier homeostasis in inflammatory bowel disease.NOD-Like Receptors: Guardians of Intestinal Mucosal Barriers.Paneth cells: the hub for sensing and regulating intestinal flora.From sensing to shaping microbiota: insights into the role of NOD2 in intestinal homeostasis and progression of Crohn's disease.The intestinal microbiota: Antibiotics, colonization resistance, and enteric pathogens.Rip2 Is Required for Nod2-Mediated Lysozyme Sorting in Paneth Cells.Administration of Lactobacillus johnsonii FI9785 to chickens affects colonisation by Campylobacter jejuni and the intestinal microbiota.NOD2 and bacterial recognition as therapeutic targets for Crohn's disease.Paneth cells in intestinal physiology and pathophysiology.Environmental factors regulate Paneth cell phenotype and host susceptibility to intestinal inflammation in Irgm1-deficient mice.Peptidoglycan recognition by the innate immune system.NOD2 and inflammation: current insights.TCF-1-mediated Wnt signaling regulates Paneth cell innate immune defense effectors HD-5 and -6: implications for Crohn's disease Nod2 Deficiency in mice is Associated with Microbiota Variation Favouring the Expansion of mucosal CD4+ LAP+ Regulatory Cells

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Mouse Paneth cell antimicrobial function is independent of Nod2.

http://www.wikidata.org/entity/Q37351171

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 19 March 2013

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Mouse Paneth cell antimicrobial function is independent of Nod2.

@en

Mouse Paneth cell antimicrobial function is independent of Nod2.

@nl

type

label

Mouse Paneth cell antimicrobial function is independent of Nod2.

@en

Mouse Paneth cell antimicrobial function is independent of Nod2.

@nl

prefLabel

Mouse Paneth cell antimicrobial function is independent of Nod2.

@en

Mouse Paneth cell antimicrobial function is independent of Nod2.

@nl

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GUTJNL-2012-304190

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Mouse Paneth cell antimicrobial function is independent of Nod2.

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Ajay S Gulati

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Andrew White

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Anthony A Fodor

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Emily Grossniklaus

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Ian M Carroll

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Michael T Shanahan

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R Balfour Sartor

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Richard J von Furstenberg

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Roshonda Barner

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Susan J Henning

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P2860

Host-microbe interactions have shaped the genetic architecture of inflammatory bowel disease

XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease

Defensins. Natural peptide antibiotics of human neutrophils

Paneth cells directly sense gut commensals and maintain homeostasis at the intestinal host-microbial interface

Secretion of microbicidal alpha-defensins by intestinal Paneth cells in response to bacteria

Site and strain-specific variation in gut microbiota profiles and metabolism in experimental mice

Increased diversity of intestinal antimicrobial peptides by covalent dimer formation

Modulation of mouse Paneth cell alpha-defensin secretion by mIKCa1, a Ca2+-activated, intermediate conductance potassium channel

Nod2-dependent regulation of innate and adaptive immunity in the intestinal tract

Strain-specific polymorphisms in Paneth cell α-defensins of C57BL/6 mice and evidence of vestigial myeloid α-defensin pseudogenes

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903-910

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10.1136/GUTJNL-2012-304190

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6

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63

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2013-03-19T00:00:00Z

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23512834

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