Anti-coreceptor therapy drives selective T cell egress by suppressing inflammation-dependent chemotactic cues. - Wikidata - awgldk - TriplyDB

Anti-coreceptor therapy drives selective T cell egress by suppressing inflammation-dependent chemotactic cues.

Anti-coreceptor therapy drives selective T cell egress by suppressing inflammation-dependent chemotactic cues.

http://www.wikidata.org/entity/Q37351576

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Type 1 Diabetes: A Chronic Anti-Self-Inflammatory Response.Therapies to Suppress β Cell Autoimmunity in Type 1 Diabetes

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Anti-coreceptor therapy drives selective T cell egress by suppressing inflammation-dependent chemotactic cues.

http://www.wikidata.org/entity/Q37351576

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 20 October 2016

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

@en

Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

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type

label

Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

@en

Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

@nl

prefLabel

Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

@en

Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

@nl

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Anti-coreceptor therapy drives ...... on-dependent chemotactic cues.

@en

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Aaron J Martin

http://www.w3.org/2001/XMLSchema#string

Bo Wang

http://www.w3.org/2001/XMLSchema#string

Douglas E Kline

http://www.w3.org/2001/XMLSchema#string

Fatima Manzoor

http://www.w3.org/2001/XMLSchema#string

Gregory Gojanovich

http://www.w3.org/2001/XMLSchema#string

Keith Miller

http://www.w3.org/2001/XMLSchema#string

Matthew Clark

http://www.w3.org/2001/XMLSchema#string

Roland Tisch

http://www.w3.org/2001/XMLSchema#string

Y Maurice Morillon

http://www.w3.org/2001/XMLSchema#string

P2860

Gamma-interferon transcriptionally regulates an early-response gene containing homology to platelet proteins

Cross-linking of T-cell surface molecules CD4 and CD8 stimulates phosphorylation of the lck tyrosine protein kinase at the autophosphorylation site

Beta cells are responsible for CXCR3-mediated T-cell infiltration in insulitis

Localization of the gene for the human MIG cytokine on chromosome 4q21 adjacent to INP10 reveals a chemokine "mini-cluster"

IFN-gamma-dependent regulatory circuits in immune inflammation highlighted in diabetes.

Therapeutic antibodies directed at G protein-coupled receptors

Entry of diabetogenic T cells into islets induces changes that lead to amplification of the cellular response.

Cellular and molecular events in the localization of diabetogenic T cells to islets of Langerhans.

Defining the transcriptional and cellular landscape of type 1 diabetes in the NOD mouse

Reprogramming the immune system: co-receptor blockade as a paradigm for harnessing tolerance mechanisms.

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e87636

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scholarly article

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10.1172/JCI.INSIGHT.87636

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17

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1

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2016-10-20T00:00:00Z

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