Defects in Jak-STAT-mediated cytokine signals cause hyper-IgE syndrome: lessons from a primary immunodeficiency.
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The hyperimmunoglobulin E syndrome--clinical manifestation diversity in primary immune deficiencyPrimary immunodeficiencies associated with eosinophiliaThe involvement of the JAK-STAT signaling pathway in chronic inflammatory skin disease atopic dermatitisIL-23/IL-17A Dysfunction Phenotypes Inform Possible Clinical Effects from Anti-IL-17A TherapiesInterleukin-17 regulation: an attractive therapeutic approach for asthmaMechanisms of NDV-3 vaccine efficacy in MRSA skin versus invasive infection.Recombinant ESAT-6-like proteins provoke protective immune responses against invasive Staphylococcus aureus disease in a murine model.New mechanism of oral immunity to mucosal candidiasis in hyper-IgE syndrome.Noncanonical STAT3 activation regulates excess TGF-β1 and collagen I expression in muscle of stricturing Crohn's disease.Safety and immunogenicity of an investigational 4-component Staphylococcus aureus vaccine with or without AS03B adjuvant: Results of a randomized phase I trial.Hyper Ig E syndrome (Job syndrome, HIES) - radiological images of pulmonary complications on the basis of three casesImmunization with Staphylococcus aureus iron regulated surface determinant B (IsdB) confers protection via Th17/IL17 pathway in a murine sepsis modelIdentification of novel HIV-1 dependency factors in primary CCR4(+)CCR6(+)Th17 cells via a genome-wide transcriptional approach.Antibody blockade of IL-17 family cytokines in immunity to acute murine oral mucosal candidiasis.Molecular explanation for the contradiction between systemic Th17 defect and localized bacterial infection in hyper-IgE syndromeChallenges for a universal Staphylococcus aureus vaccine.Host- and microbe determinants that may influence the success of S. aureus colonization.New genetic discoveries and primary immune deficiencies.STAT3 inhibitors: finding a home in lymphoma and leukemia.STAT3 signaling in immunity.Hyper-IgE syndrome with STAT3 mutation: a case report in Mainland ChinaVertebral aspergillosis in a patient with autosomal-dominant hyper-IgE syndrome.Design, Synthesis, and Antitumor Evaluation of 4-Amino-(1H)-pyrazole Derivatives as JAKs InhibitorsClinical aspects and genetic analysis of Taiwanese patients with the phenotype of hyper-immunoglobulin E recurrent infection syndromes (HIES).Eight-color immunophenotyping of T-, B-, and NK-cell subpopulations for characterization of chronic immunodeficiencies.S. aureus colonization induces strain-specific suppression of IL-17.Association of JAK-STAT pathway related genes with lymphoma risk: results of a European case-control study (EpiLymph).MicroRNA regulation of liver cancer stem cells
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Defects in Jak-STAT-mediated cytokine signals cause hyper-IgE syndrome: lessons from a primary immunodeficiency.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 15 December 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@en
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@nl
type
label
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@en
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@nl
prefLabel
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@en
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@nl
P2860
P356
P1476
Defects in Jak-STAT-mediated c ...... om a primary immunodeficiency.
@en
P2093
Hajime Karasuyama
Yoshiyuki Minegishi
P2860
P304
P356
10.1093/INTIMM/DXN134
P577
2008-12-15T00:00:00Z