Oxidative stress-induced overexpression of miR-25: the mechanism underlying the degeneration of melanocytes in vitiligo.
about
Dysregulated autophagy increased melanocyte sensitivity to H2O2-induced oxidative stress in vitiligo.Novel approaches to vitiligo treatment via modulation of mTOR and NF-κB pathways in human skin melanocytes.Altered expression of four miRNA (miR-1238-3p, miR-202-3p, miR-630 and miR-766-3p) and their potential targets in peripheral blood from vitiligo patients.Nrf2 in keratinocytes modulates UVB-induced DNA damage and apoptosis in melanocytes through MAPK signaling.Extracellular microRNAs and oxidative stress in liver injury: a systematic mini review
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Oxidative stress-induced overexpression of miR-25: the mechanism underlying the degeneration of melanocytes in vitiligo.
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article científic
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article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 28 August 2015
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@en
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@nl
type
label
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@en
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@nl
prefLabel
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@en
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@nl
P2093
P2860
P356
P1476
Oxidative stress-induced overe ...... on of melanocytes in vitiligo.
@en
P2093
P2860
P2888
P304
P356
10.1038/CDD.2015.117
P577
2015-08-28T00:00:00Z