Inhibition of LTi cell development by CD25 blockade is associated with decreased intrathecal inflammation in multiple sclerosis. - Wikidata - awgldk - TriplyDB

Inhibition of LTi cell development by CD25 blockade is associated with decreased intrathecal inflammation in multiple sclerosis.

Inhibition of LTi cell development by CD25 blockade is associated with decreased intrathecal inflammation in multiple sclerosis.

http://www.wikidata.org/entity/Q37354974

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The origin and role of innate lymphoid cells in the lung Maintenance of Immune Homeostasis through ILC/T Cell Interactions Intrathecal IgG synthesis: a resistant and valuable target for future multiple sclerosis treatments Regulation of the adaptive immune system by innate lymphoid cells Daclizumab therapy for multiple sclerosis Review of daclizumab and its therapeutic potential in the treatment of relapsing-remitting multiple sclerosis Therapeutic efficacy of monthly subcutaneous injection of daclizumab in relapsing multiple sclerosis Tertiary Lymphoid Organs in Central Nervous System Autoimmunity The effects of interleukin-2 on immune response regulation.The efficacy and safety of daclizumab and its potential role in the treatment of multiple sclerosis.How implementation of systems biology into clinical trials accelerates understanding of diseases.Establishment and function of tissue-resident innate lymphoid cells in the skin.A complex role of herpes viruses in the disease process of multiple sclerosis.Cerebrospinal fluid IL-12p40, CXCL13 and IL-8 as a combinatorial biomarker of active intrathecal inflammation.Vitamin D in Autoimmunity: Molecular Mechanisms and Therapeutic Potential.CD56(bright) natural killer cells and response to daclizumab HYP in relapsing-remitting MS The meninges: new therapeutic targets for multiple sclerosis.Daclizumab reverses intrathecal immune cell abnormalities in multiple sclerosis Innate lymphoid cells. Innate lymphoid cells: a new paradigm in immunology RORγt Expression and Lymphoid Neogenesis in the Brain of Patients with Secondary Progressive Multiple Sclerosis.The single nucleotide variant rs12722489 determines differential estrogen receptor binding and enhancer properties of an IL2RA intronic region Innate lymphoid cell interactions with microbiota: implications for intestinal health and disease.Pioglitazone regulates myelin phagocytosis and multiple sclerosis monocytes.Patients with MS under daclizumab therapy mount normal immune responses to influenza vaccination Cutaneous adverse events in multiple sclerosis patients treated with daclizumab The effect of daclizumab on brain atrophy in relapsing-remitting multiple sclerosis.Expansion of inflammatory innate lymphoid cells in patients with common variable immune deficiency Innate lymphoid cells in the initiation, regulation and resolution of inflammation Impaired NK-mediated regulation of T-cell activity in multiple sclerosis is reconstituted by IL-2 receptor modulation Alemtuzumab treatment alters circulating innate immune cells in multiple sclerosis.Emerging concepts and future challenges in innate lymphoid cell biology Effect of daclizumab high-yield process in patients with highly active relapsing-remitting multiple sclerosis.Daclizumab-induced adverse events in multiple organ systems in multiple sclerosis.Teriflunomide and its mechanism of action in multiple sclerosis.The role of natural killer cells in multiple sclerosis and their therapeutic implications.Modulation of IL-2Rα with daclizumab for treatment of multiple sclerosis.Management of disease-modifying treatments in neurological autoimmune diseases of the central nervous system.The CD25-binding antibody Daclizumab High-Yield Process has a distinct glycosylation pattern and reduced antibody-dependent cell-mediated cytotoxicity in comparison to Zenapax®.CD4+ Group 1 Innate Lymphoid Cells (ILC) Form a Functionally Distinct ILC Subset That Is Increased in Systemic Sclerosis.Daclizumab for the treatment of multiple sclerosis.

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Inhibition of LTi cell development by CD25 blockade is associated with decreased intrathecal inflammation in multiple sclerosis.

http://www.wikidata.org/entity/Q37354974

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article científic

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article scientifique

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articolo scientifico

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bilimsel makale

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scientific article published on August 2012

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vedecký článok

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vetenskaplig artikel

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name

Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

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Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

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Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

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Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

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Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

@en

Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

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SCITRANSLMED.3004140

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Science Translational Medicine

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Inhibition of LTi cell develop ...... mmation in multiple sclerosis.

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Bibiana Bielekova

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Gyorgy Csako

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Justin S A Perry

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Lucy B Kennedy

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Matthew L Herman

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Quangang Xu

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Sungpil Han

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Monocyte-like and mature macrophages produce CXCL13 (B cell-attracting chemokine 1) in inflammatory lesions with lymphoid neogenesis

Overlapping roles of CXCL13, interleukin 7 receptor alpha, and CCR7 ligands in lymph node development

Chemokine CXCL13 is essential for lymph node initiation and is induced by retinoic acid and neuronal stimulation

Humanized anti-interleukin-2 (IL-2) receptor alpha therapy: long-term results in uveitis patients and preliminary safety and activity data for establishing parameters for subcutaneous administration.

Fate and function of lymphoid tissue inducer cells.

IL-7 and IL-15 independently program the differentiation of intestinal CD3-NKp46+ cell subsets from Id2-dependent precursors.

Development of peripheral lymphoid organs and natural killer cells depends on the helix-loop-helix inhibitor Id2.

Expansion of human NK-22 cells with IL-7, IL-2, and IL-1beta reveals intrinsic functional plasticity.

The expanding family of innate lymphoid cells: regulators and effectors of immunity and tissue remodeling.

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10.1126/SCITRANSLMED.3004140

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2012-08-01T00:00:00Z

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22855463

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multiple sclerosis

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3846177

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