Possible role of glial cells in the onset and progression of Lyme neuroborreliosis. - Wikidata - awgldk - TriplyDB

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

http://www.wikidata.org/entity/Q37356125

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Cytokines and chemokines at the crossroads of neuroinflammation, neurodegeneration, and neuropathic pain Lyme disease and pediatric autoimmune neuropsychiatric disorders associated with streptococcal infections (PANDAS): an overview Anti-inflammatory effects of dexamethasone and meloxicam on Borrelia burgdorferi-induced inflammation in neuronal cultures of dorsal root ganglia and myelinating cells of the peripheral nervous system The chemokine CXCL13 is a key regulator of B cell recruitment to the cerebrospinal fluid in acute Lyme neuroborreliosis.Receptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi.Regulated Production of CXCL13 within the Central Nervous System.A Refined Bead-Free Method to Identify Astrocytic Exosomes in Primary Glial Cultures and Blood Plasma Lymphoid chemokines in the CNS.Inflammatory mediator release from primary rhesus microglia in response to Borrelia burgdorferi results from the activation of several receptors and pathways Inflammation in the pathogenesis of lyme neuroborreliosis Interleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammation Novel Therapeutic Targets in Neuroinflammation and Neuropathic Pain Altered hippocampal synaptic transmission in transgenic mice with astrocyte-targeted enhanced CCL2 expression.An inverted blood-brain barrier model that permits interactions between glia and inflammatory stimuli A possible role for inflammation in mediating apoptosis of oligodendrocytes as induced by the Lyme disease spirochete Borrelia burgdorferi.Cerebrospinal fluid proteome of patients with acute Lyme disease Acetate supplementation reduces microglia activation and brain interleukin-1β levels in a rat model of Lyme neuroborreliosis CXCL13 drives spinal astrocyte activation and neuropathic pain via CXCR5.Targeting CXCL13 During Neuroinflammation The Lyme disease spirochete Borrelia burgdorferi induces inflammation and apoptosis in cells from dorsal root ganglia Chemokine CXCL13 mediates orofacial neuropathic pain via CXCR5/ERK pathway in the trigeminal ganglion of mice The multifaceted responses of primary human astrocytes and brain microvascular endothelial cells to the Lyme disease spirochete, Borrelia burgdorferi.CXCL13/CXCR5 enhances sodium channel Nav1.8 current density via p38 MAP kinase in primary sensory neurons following inflammatory pain Effects of dexamethasone and meloxicam on Borrelia burgdorferi-induced inflammation in glial and neuronal cells of the central nervous system.The MEK/ERK pathway is the primary conduit for Borrelia burgdorferi-induced inflammation and P53-mediated apoptosis in oligodendrocytes.Aprepitant limits in vivo neuroinflammatory responses in a rhesus model of Lyme neuroborreliosis.Repetitive hypoxic preconditioning induces an immunosuppressed B cell phenotype during endogenous protection from stroke.Human microglia and astrocytes constitutively express the neurokinin-1 receptor and functionally respond to substance P.Intracellular TLR7 is activated in human oligodendrocytes in response to Borrelia burgdorferi exposure.Primary Human Microglia Are Phagocytically Active and Respond to Borrelia burgdorferi With Upregulation of Chemokines and Cytokines.Role of CXCL13 in the formation of the meningeal tertiary lymphoid organ in multiple sclerosis

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Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

http://www.wikidata.org/entity/Q37356125

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 25 August 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

@en

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

@nl

type

label

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

@en

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

@nl

prefLabel

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

@en

Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

@nl

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Journal of Neuroinflammation

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Possible role of glial cells in the onset and progression of Lyme neuroborreliosis.

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Amy Gill

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Dale S Martin

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Erin P Ribka

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Geeta Ramesh

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Juan T Borda

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Lisa A Morici

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Mario T Philipp

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Mary B Jacobs

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Peter J Didier

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Peter Mottram

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P2860

Inflammatory brain changes in Lyme borreliosis

Nervous System Lyme Disease

Pathogenesis of Lyme neuroborreliosis:Borrelia burgdorferi lipoproteins induce both proliferation and apoptosis in rhesus monkey astrocytes

The pathogenesis of lyme neuroborreliosis: from infection to inflammation

Satellite glial cells in sensory ganglia: from form to function

Monocyte chemoattractant protein 1 acts as a T-lymphocyte chemoattractant

Absence of monocyte chemoattractant protein 1 in mice leads to decreased local macrophage recruitment and antigen-specific T helper cell type 1 immune response in experimental autoimmune encephalomyelitis

Pathophysiology of bacterial meningitis: mechanism(s) of neuronal injury.

Cytokines and chemokines in meningeal inflammation: biology and clinical implications.

Visualizing cytokine-secreting cells in situ in the rhesus macaque model of chronic gut inflammation

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10.1186/1742-2094-6-23

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